Effect of Severe Muscular Work on Total and Coronary Circulation in Man in Relation to Findings in the Coronary Arteriogram

2015 ◽  
pp. 268-279
Author(s):  
S. Holmberg
Keyword(s):  
Coronary Circulation ◽  
Muscular Work

Exercise-induced rise in glucagon and ketogenesis during prolonged muscular work

Diabetes ◽  
1989 ◽  
Vol 38 (6) ◽  
pp. 799-807 ◽  
Author(s):  
D. H. Wasserman ◽  
J. A. Spalding ◽  
D. Bracy ◽  
D. B. Lacy ◽  
A. D. Cherrington
Keyword(s):  
Muscular Work ◽  
Exercise Induced

Myocardial Lipids After Intense Muscular Work

Cardiology ◽  
1964 ◽  
Vol 45 (5) ◽  
pp. 269-275 ◽  
Author(s):  
M. Rinetti ◽  
O. Visioli ◽  
L. Colombi ◽  
F. Barbaresi
Keyword(s):  
Muscular Work

scholarly journals The coronary circulation

1928 ◽  
Vol 65 (4) ◽  
pp. 357-373 ◽  
Author(s):  
G. V. Anrep ◽  
H. Häusler
Keyword(s):  
Coronary Circulation

Myocardial CO2 buffering: role of transmembrane transport of H+ or HCO3-ions

1976 ◽  
Vol 230 (4) ◽  
pp. 1037-1041 ◽  
Author(s):  
DR Strome ◽  
RL Clancy ◽  
NC Gonzalez
Keyword(s):  
Small Volume ◽  
Coronary Circulation ◽  
Myocardial Cell ◽  
Ringer Solution ◽  
Red Cells ◽  
Transmembrane Transport ◽  
High Degree

Isolated rabbit hearts were perfused with rabbit red cells suspended in Ringer solution. A small volume of perfusate was recirculated for 10 min at Pco2 of 33.4 +/- 0.9 or 150.8 +/- 7.5 mmHg. Hypercapnia resulted in an increase in perfusate HCO3- concentration that was smaller than that observed when isolated perfusate was equilibrated in vitro with the same CO2 tensions (delta HCO-3e = 1.6 mM, P less than 0.01). This difference is consistent with a net movement of HCO3- into or H+ out of the mycardial cell, and cannot be accounted for by dilution of HCO3- in the myocardial interstitium. Recirculation of perfusate through the coronary circulation at normal Pco2 for two consecutive 10-min periods was not followed by changes in perfusate HCO3- concentration. A high degree of correlation (r = 0.81) was observed between intracellular HCO-3e concentration and the corresponding delta HCO-3e in individual experiments. The results suggest that transmembrane exchange of H+ or HCO3- is a buffer mechanism for CO2 in the myocardial cell.

scholarly journals Prostacyclin, but not nitric oxide, mediates the vasodilator response to endothelin-1 in the coronary circulation

1996 ◽  
Vol 27 (2) ◽  
pp. 322
Author(s):  
Jay H. Traverse ◽  
Dianne L. Judd ◽  
Todd J. Pavek ◽  
Melanie J. Crampton ◽  
Robert J. Bache
Keyword(s):  
Nitric Oxide ◽  
Coronary Circulation ◽  
Endothelin 1 ◽  
Vasodilator Response
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