Central Venous Oxygen Saturation: A Potential New Marker for Circulatory Stress in Haemodialysis Patients?

2014 ◽  
Vol 128 (1-2) ◽  
pp. 57-60 ◽  
Author(s):  
Laura E.A. Harrison ◽  
Nicholas M. Selby ◽  
Christopher W. McIntyre
Keyword(s):  
Oxygen Saturation ◽  
Central Venous Oxygen Saturation ◽  
Central Venous ◽  
Venous Oxygen Saturation ◽  
Circulatory Stress

Central Venous Oxygen Saturation

1991 ◽  
Vol XXXV (6) ◽  
pp. 327
Author(s):  
T. M. SCALEA ◽  
R. W. HARTNETT ◽  
A. O. DUNCAN ◽  
N. A. ATWEH ◽  
T. F. PHILLIPS ◽  
...  
Keyword(s):  
Oxygen Saturation ◽  
Central Venous Oxygen Saturation ◽  
Central Venous ◽  
Venous Oxygen Saturation

scholarly journals High central venous oxygen saturation in the latter stages of septic shock is associated with increased mortality

Critical Care ◽  
2011 ◽  
Vol 15 (4) ◽  
pp. R176 ◽  
Author(s):  
Julien Textoris ◽  
Louis Fouché ◽  
Sandrine Wiramus ◽  
François Antonini ◽  
Sowita Tho ◽  
...  
Keyword(s):  
Septic Shock ◽  
Oxygen Saturation ◽  
Central Venous Oxygen Saturation ◽  
Central Venous ◽  
Venous Oxygen Saturation

scholarly journals Accuracy of central venous oxygen saturation with a fiberoptic catheter

Critical Care ◽  
2009 ◽  
Vol 13 (Suppl 1) ◽  
pp. P234
Author(s):  
D Chiumello ◽  
V Berto ◽  
C Mietto ◽  
M Botticelli ◽  
M Chierichetti ◽  
...  
Keyword(s):  
Oxygen Saturation ◽  
Central Venous Oxygen Saturation ◽  
Central Venous ◽  
Venous Oxygen Saturation ◽  
Fiberoptic Catheter

Tissue Oxygenation Monitoring as a Guide for Trauma Resuscitation

2016 ◽  
Vol 36 (3) ◽  
pp. 12-70 ◽  
Author(s):  
Cathy Mitchell
Keyword(s):  
Oxygen Saturation ◽  
Tissue Oxygenation ◽  
Central Venous Oxygen Saturation ◽  
Multiple Organ ◽  
Tissue Oxygen Saturation ◽  
Multiple Organ Dysfunction ◽  
Tissue Oxygen ◽  
Trauma Resuscitation ◽  
Central Venous ◽  
Venous Oxygen Saturation

Hypoperfusion is the most common event preceding the onset of multiple organ dysfunction syndrome during trauma resuscitation. Detecting subtle changes in perfusion is crucial to ensure adequate tissue oxygenation and perfusion. Traditional methods of detecting physiological changes include measurements of blood pressure, heart rate, urine output, serum levels of lactate, mixed venous oxygen saturation, and central venous oxygen saturation. Continuous noninvasive monitoring of tissue oxygen saturation in muscle has the potential to indicate severity of shock, detect occult hypoperfusion, guide resuscitation, and be predictive of the need for interventions to prevent multiple organ dysfunction syndrome. Tissue oxygen saturation is being used in emergency departments, trauma rooms, operating rooms, and emergency medical services. Tissue oxygen saturation technology is just as effective as mixed venous oxygen saturation, central venous oxygen saturation, serum lactate, and Stewart approach with strong ion gap, yet tissue oxygen saturation assessment is also a direct, noninvasive microcirculatory measurement of oxygen saturation.

scholarly journals Does Central Venous Oxygen Saturation-directed Fluid Therapy Affect Postoperative Morbidity after Colorectal Surgery?

2010 ◽  
Vol 113 (5) ◽  
pp. 1072-1080 ◽  
Author(s):  
Ib Jammer ◽  
Atle Ulvik ◽  
Christian Erichsen ◽  
Olav Lødemel ◽  
Gro Østgaard
Keyword(s):  
Oxygen Saturation ◽  
Fluid Therapy ◽  
Treatment Algorithm ◽  
Clinical Signs ◽  
Central Venous Oxygen Saturation ◽  
Intravenous Fluid ◽  
Control Group ◽  
Central Venous ◽  
Intensive Care Patients ◽  
Venous Oxygen Saturation

Background The optimal amount and method for monitoring intravenous fluid in surgical patients is unresolved. Central venous oxygen saturation (Scvo2) has been used to guide therapy and predict outcome in high-risk and intensive-care patients. The aim of this prospective, randomized trial was to compare the rate of postoperative complications in patients receiving fluid therapy guided by Scvo2 and those treated with a traditional effluent fluid scheme. Methods Patients undergoing open colorectal and lower intestinal surgery (n = 241) were randomized to the Scvo2 group or the control group. The Scvo2 group received perioperatively crystalloid infusion 100 ml/h. When Scvo2 was less than 75%, a bolus of 3 ml/kg hydroxyethyl starch was given. The bolus was repeated if Scvo2 increased by 1 percentage point or more. The control group was maintained with crystalloid 800 ml/h and given extra fluid if there were clinical signs of hypovolemia. The participating surgeon, unaware of the group allocation, registered complications within day 30. Results Until 8:00 am on the first postoperative day, the Scvo2 group had received 3,869 ± 992 ml (mean ± SD) intravenous fluid compared with 6,491 ± 1,649 ml in the control group. Increase in weight was 0.8 ± 1.8 kg and 2.5 ± 1.6 kg in the two groups, respectively. The postoperative complication rate was 42% in both groups. Conclusion Clinical outcomes among patients receiving Scvo2-guided perioperative fluid therapy were similar to those for patients treated with a traditional fluid regimen. Limitations in study design prevent full interpretation of these findings, and further large trials of this treatment algorithm are still required.

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