Nonclonal Lymphocytic Proliferation in Cutaneous Lymphoid Hyperplasia: A Flow-Cytometric and Morphological Analysis

Dermatology ◽  
1992 ◽  
Vol 185 (2) ◽  
pp. 113-119 ◽  
Author(s):  
K. Fan ◽  
R. Kelly ◽  
V. Kendrick
Author(s):  
Kenneth M. Jacobs ◽  
Junhua Ding ◽  
Li V. Yang ◽  
Carissa L. Reynolds ◽  
Andrew E. Ekpenyong ◽  
...  

2010 ◽  
Vol 42 (1) ◽  
pp. 11-19 ◽  
Author(s):  
Bertrand Evrard ◽  
Annie Dosgilbert ◽  
Nathalie Jacquemot ◽  
François Demeocq ◽  
Thibault Gilles ◽  
...  

Phytotaxa ◽  
2018 ◽  
Vol 349 (3) ◽  
pp. 225
Author(s):  
LUCIE KOBRLOVÁ ◽  
TEREZIE MANDÁKOVÁ ◽  
MICHAL HRONEŠ

Symphytum leonhardtianum, a member of the S. tuberosum complex, is investigated. This taxon was described by Pugsley in 1931, from the vicinity of Vienna, Austria. Nevertheless, it is generally not accepted in European floras. In this study, we conducted an evaluation of this taxon using flow cytometry, karyology and morphological analysis. Flow cytometric and karyological investigations of plants from the type locality of S. leonhardtianum revealed only dodecaploids (2n = 12x = 96), a ploidy level corresponding to the S. tuberosum subsp. tuberosum. The chromosome number of the S. tuberosum from Austria is here recorded for the first time. Morphological comparison of Central European populations of S. tuberosum complex showed that S. leonhardtianum did not differ significantly from S. tuberosum subsp. tuberosum. Based on our findings, we propose treating the name S. leonhardtianum as a heterotypic synonym of S. tuberosum subsp. tuberosum. The lectotype of S. leonhardtianum is designated.


Author(s):  
K.S. Kosik ◽  
L.K. Duffy ◽  
S. Bakalis ◽  
C. Abraham ◽  
D.J. Selkoe

The major structural lesions of the human brain during aging and in Alzheimer disease (AD) are the neurofibrillary tangles (NFT) and the senile (neuritic) plaque. Although these fibrous alterations have been recognized by light microscopists for almost a century, detailed biochemical and morphological analysis of the lesions has been undertaken only recently. Because the intraneuronal deposits in the NFT and the plaque neurites and the extraneuronal amyloid cores of the plaques have a filamentous ultrastructure, the neuronal cytoskeleton has played a prominent role in most pathogenetic hypotheses.The approach of our laboratory toward elucidating the origin of plaques and tangles in AD has been two-fold: the use of analytical protein chemistry to purify and then characterize the pathological fibers comprising the tangles and plaques, and the use of certain monoclonal antibodies to neuronal cytoskeletal proteins that, despite high specificity, cross-react with NFT and thus implicate epitopes of these proteins as constituents of the tangles.


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