scholarly journals Nephrotic Syndrome and Acute Renal Failure Apparently Induced by Sunitinib

2009 ◽  
Vol 2 (3) ◽  
pp. 172-176 ◽  
Author(s):  
Ying-Shou Chen ◽  
Chien-Liang Chen ◽  
Jyh-Seng Wang
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome

A Case with Acute Renal Failure and Subsequent Nephrotic Syndrome

Renal Failure ◽  
2009 ◽  
Vol 31 (2) ◽  
pp. 162-166 ◽  
Author(s):  
Hiroshi Nonoguchi ◽  
Yukimasa Kohda ◽  
Rika Fukutomi ◽  
Yushi Nakayama ◽  
Masahiro Naruse ◽  
...  
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome

scholarly journals Reversible Acute Renal Failure in Idiopathic Nephrotic Syndrome.

1993 ◽  
Vol 32 (1) ◽  
pp. 31-35 ◽  
Author(s):  
Ryuichi FURUYA ◽  
Hiromichi KUMAGAI ◽  
Naoki IKEGAYA ◽  
Shuzo KOBAYASHI ◽  
Masato KIMURA ◽  
...  
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome ◽  
Idiopathic Nephrotic Syndrome

Acute Renal Failure and Tubular Dysfunction Associated with Minimal Change Nephrotic Syndrome

1989 ◽  
Vol 4 (10) ◽  
pp. 914-916 ◽  
Author(s):  
M. Praga ◽  
M. A. Martinez ◽  
A. Andres ◽  
M. Alvarez de Buergo ◽  
I. Bello ◽  
...  
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome ◽  
Minimal Change Nephrotic Syndrome ◽  
Minimal Change ◽  
Tubular Dysfunction

Acute Renal Failure and Nephrotic Syndrome after Angiocardiography with Meglumine Diatrizoate

1971 ◽  
Vol 284 (11) ◽  
pp. 592-593 ◽  
Author(s):  
Sonia Borra ◽  
David Hawkins ◽  
William Duguid ◽  
Michael Kaye
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome ◽  
Meglumine Diatrizoate

Irreversible acute renal failure in idiopathic nephrotic syndrome

1976 ◽  
Vol 61 (2) ◽  
pp. 207-214 ◽  
Author(s):  
Leopoldo Raij ◽  
William F. Keane ◽  
Arthur Leonard ◽  
Fred L. Shapiro
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome ◽  
Idiopathic Nephrotic Syndrome

Glomerulopathy does not increase renal susceptibility to acute ischemic injury

1984 ◽  
Vol 246 (3) ◽  
pp. F272-F281 ◽  
Author(s):  
R. A. Zager ◽  
L. A. Baltes ◽  
H. M. Sharma ◽  
W. G. Couser
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Nephrotic Syndrome ◽  
Renal Injury ◽  
Artery Occlusion ◽  
Minimal Change ◽  
Glomerular Injury ◽  
Bilateral Renal Artery ◽  
Foot Process ◽  
Ischemic Renal Injury

To determine whether preexistent glomerular injury and the nephrotic syndrome increase renal susceptibility to ischemic renal injury, normal rats and rats with either experimental minimal-change disease (Adriamycin nephropathy) (AN) or membranous nephropathy (passive Heymann nephritis) (PHN) underwent renal functional and histologic studies under either basal conditions or 18 h after bilateral renal artery occlusion (over 30 min). Prior to renal ischemia AN and PHN rats had minimally depressed glomerular filtration rate (GFR), normal (AN) or increased (PHN) renal blood flow (RBF), heavy proteinuria, hypoalbuminemia, decreased urine sodium excretion, extensive glomerular foot process fusion, and intratubular hyalin cast formation. Losses of GFR in response to ischemia were comparable among the three groups of rats (controls, 0.29; AN, 0.28; PHN, 0.25 ml X min-1 X 100 g body wt-1) despite prevailing differences in postischemic hemodynamics. Neither light nor transmission electron microscopy showed any differences in the degree of ischemic renal injury. These results suggest that 1) glomerulopathy and the nephrotic syndrome do not significantly increase renal susceptibility to ischemic renal injury; 2) the syndrome of acute renal failure that occurs in patients with minimal-change glomerulopathy is not due to a marked susceptibility of these kidneys to clinically occult ischemic events; and 3) foot process fusion is probably not a pathophysiologically significant lesion in ischemic acute renal failure, as previously suggested.

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