Morphologically Different Biopsy Specimens of the Human Gastric Mucosa

Pharmacology ◽  
1986 ◽  
Vol 33 (3) ◽  
pp. 131-138 ◽  
Author(s):  
H.-J. Ruoff ◽  
T. Leyhe ◽  
G. Pfaff ◽  
G. Rettenmaier ◽  
E. Schmid
Keyword(s):  
Gastric Mucosa ◽  
Human Gastric Mucosa ◽  
Biopsy Specimens

Adenylate cyclase in human gastric mucosa: its activation by histamine in morphologically different biopsy specimens

1979 ◽  
Vol 57 (14) ◽  
pp. 725-730 ◽  
Author(s):  
H. -J. Ruoff ◽  
B. Painz ◽  
M. Becker ◽  
M. Rack ◽  
K. -Fr Sewing ◽  
...  
Keyword(s):  
Gastric Mucosa ◽  
Adenylate Cyclase ◽  
Human Gastric Mucosa ◽  
Biopsy Specimens

Morphologically Different Biopsy Specimens of the Human Gastric Mucosa

Pharmacology ◽  
1986 ◽  
Vol 33 (3) ◽  
pp. 121-130 ◽  
Author(s):  
H.-J. Ruoff ◽  
T. Leyhe ◽  
U.B. Eichhorst ◽  
E.-M. Haile ◽  
H. Makhow
Keyword(s):  
Gastric Mucosa ◽  
Human Gastric Mucosa ◽  
Biopsy Specimens

Enzyme Activities in Biopsy Specimens from Human Gastric Mucosa

1986 ◽  
Vol 21 (9) ◽  
pp. 1051-1057 ◽  
Author(s):  
B. Børkje ◽  
S. Ødegaard ◽  
D. W. Skagen ◽  
K.-J. Andersen ◽  
E. Schrumpf
Keyword(s):  
Gastric Mucosa ◽  
Enzyme Activities ◽  
Human Gastric Mucosa ◽  
Biopsy Specimens

An Electron Microscopic Study of the Human Gastric Epithelia with Special Reference to the Ultrastructural Changes of the Parietal cells Induced by “Histalog“

1972 ◽  
Vol 30 ◽  
pp. 346-347
Author(s):  
Hiroshi Saito ◽  
Goro Asano ◽  
Kaoru Aihara ◽  
Katsunari Fukushi ◽  
Minoru Yoshida ◽  
...  
Keyword(s):  
Electron Microscope ◽  
Gastric Mucosa ◽  
Atrophic Gastritis ◽  
Endoscopic Biopsy ◽  
Parietal Cells ◽  
Ultrastructural Changes ◽  
Human Gastric Mucosa ◽  
Electron Microscopic ◽  
Biopsy Specimens ◽  
Control Study

This is to communicate the ultrastructural changes of the parietal cells of the human gastric mucosa in “Histalog” stimulation.Gastro-endoscopic biopsy specimens from 48 adult patients suffering from various gastritic conditions are studied. The patients are roughly divided into two groups. The first group consists of the control study. In essentially intact gastric mucosa (hyperacidic) cases, the “Histolog” 50mg had been injected intramuscularly to the subjects and chronologically endoscopic biopsy study had been achieved at 5, 10, 15, 20, 30, and 60 minutes respectively. The second group consists of the study with advanced atrophic gastritis (anacidity) and similar manipulation has been attemped. The obtained specimens were quickly fixed in 2.5% glutaraldehyde, postosmificated, and dehydrated in graded alcohols and embedded in Epon 812. Sections were cut and observed with the Hitachi 11U-B electron microscope with 50kv or 75kv accelerating voltages.

scholarly journals Interleukin-17C in Human Helicobacter pylori Gastritis

2017 ◽  
Vol 85 (10) ◽  
Author(s):  
Shingo Tanaka ◽  
Hiroyuki Nagashima ◽  
Modesto Cruz ◽  
Tomohisa Uchida ◽  
Takahiro Uotani ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Gastric Mucosa ◽  
Gastric Biopsy ◽  
Interleukin 17 ◽  
Human Gastric Mucosa ◽  
Mrna Levels ◽  
Content Type ◽  
Biopsy Specimens ◽  
H Pylori

ABSTRACT The interleukin-17 (IL-17) family of cytokines (IL-17A to IL-17F) is involved in many inflammatory diseases. Although IL-17A is recognized as being involved in the pathophysiology of Helicobacter pylori-associated diseases, the role of other IL-17 cytokine family members remains unclear. Microarray analysis of IL-17 family cytokines was performed in H. pylori-infected and uninfected gastric biopsy specimens. IL-17C mRNA was upregulated approximately 4.5-fold in H. pylori-infected gastric biopsy specimens. This was confirmed by quantitative reverse transcriptase PCR in infected and uninfected gastric mucosa obtained from Bhutan and from the Dominican Republic. Immunohistochemical analysis showed that IL-17C expression in H. pylori-infected gastric biopsy specimens was predominantly localized to epithelial and chromogranin A-positive endocrine cells. IL-17C mRNA levels were also significantly greater among cagA-positive than cagA-negative H. pylori infections (P = 0.012). In vitro studies confirmed an increase in IL-17C mRNA and protein levels in cells infected with cagA-positive infections compared to cells infected with either cagA-negative or cag pathogenicity island (PAI) mutant. Chemical inhibition of IκB kinase (IKK), mitogen-activated protein extracellular signal-regulated kinase (MEK), and Jun N-terminal kinase (JNK) inhibited induction of IL-17C proteins in infected cells, whereas p38 inhibition had no effect on IL-17C protein secretion. In conclusion, H. pylori infection was associated with a significant increase in IL-17C expression in human gastric mucosa. The role of IL-17C in the pathogenesis of H. pylori-induced diseases remains to be determined.

scholarly journals Phospholipid composition of human gastric mucosa: a study of endoscopic biopsy specimens.

Gut ◽  
1993 ◽  
Vol 34 (4) ◽  
pp. 456-460 ◽  
Author(s):  
G Nardone ◽  
P Laccetti ◽  
C Civiletti ◽  
G Budillon
Keyword(s):  
Gastric Mucosa ◽  
Phospholipid Composition ◽  
Endoscopic Biopsy ◽  
Human Gastric Mucosa ◽  
Biopsy Specimens

H. pylori down-regulates TRAIL (DR4)-receptors in the human gastric mucosa, a possible mechanism for chronic persistence

2001 ◽  
Vol 120 (5) ◽  
pp. A81-A81
Author(s):  
B NEU ◽  
R RAD ◽  
M NEUHOFER ◽  
C TRAUTWEIN ◽  
M GERHARD ◽  
...  
Keyword(s):  
Gastric Mucosa ◽  
Human Gastric Mucosa ◽  
H Pylori
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