Intracellular Activity of L-Phenylalanine during Intestinal Transport

Pharmacology ◽  
1967 ◽  
Vol 17 (5) ◽  
pp. 483-488
Author(s):  
A. Faelli ◽  
G. Esposito ◽  
V. Capraro
Keyword(s):  
Intestinal Transport ◽  
Intracellular Activity

Nanoemulsion Formulation Enhances Intracellular Activity of Capuramycin Analogues against Mycobacterium Tuberculosis

2011 ◽  
Vol 1 (2) ◽  
pp. 150-158
Author(s):  
Venkata M. Reddy ◽  
Elena Bogatcheva ◽  
Leo Einck ◽  
Carol A. Nacy
Keyword(s):  
Mycobacterium Tuberculosis ◽  
Intracellular Activity

Intestinal Transport and Processing of Immunoglobulin G in the Neonatal and Adult Rat

Neonatology ◽  
1995 ◽  
Vol 67 (4) ◽  
pp. 254-263 ◽  
Author(s):  
N. Benlounes ◽  
R. Chedid ◽  
F. Thuillier ◽  
J.F. Desjeux ◽  
F. Rousselet ◽  
...  
Keyword(s):  
Immunoglobulin G ◽  
Intestinal Transport ◽  
Adult Rat

Improved Intestinal Transport of PD 158473, an N-methyl-d-Aspartate (NMDA) Antagonist, by Involvement of Multiple Transporters

2002 ◽  
Vol 91 (12) ◽  
pp. 2579-2587 ◽  
Author(s):  
Doo-Man Oh ◽  
Hyo-Kyung Han ◽  
Rufus M. Williamson ◽  
Christopher F. Bigge ◽  
Gordon L. Amidon ◽  
...  
Keyword(s):  
Nmda Antagonist ◽  
Intestinal Transport

The Na+-dependence of thiamin intestinal transport

Life Sciences ◽  
1971 ◽  
Vol 10 (2) ◽  
pp. 67-75 ◽  
Author(s):  
G. Ferrari ◽  
U. Ventura ◽  
G. Rindi
Keyword(s):  
Intestinal Transport ◽  
Thiamin Intestinal Transport

Intestinal transport of HDND-7, a novel hesperetin derivative, in in vitro MDCK cell and in situ single-pass intestinal perfusion models

Xenobiotica ◽  
2016 ◽  
Vol 47 (8) ◽  
pp. 719-730 ◽  
Author(s):  
Ruonan Chen ◽  
Lan Li ◽  
Chenlin Shen ◽  
Cheng Huang ◽  
Taotao Ma ◽  
...  
Keyword(s):  
Mdck Cell ◽  
Intestinal Transport ◽  
Intestinal Perfusion ◽  
Single Pass

Intestinal transport of amino acids studied in vitro with l-[131I]monoiodotyrosine

1960 ◽  
Vol 41 (2) ◽  
pp. 271-282 ◽  
Author(s):  
Daniel Nathans ◽  
Donald F. Tapley ◽  
Joan E. Ross
Keyword(s):  
Amino Acids ◽  
Intestinal Transport

Regulation of intestinal glucose transport

1992 ◽  
Vol 70 (9) ◽  
pp. 1201-1207 ◽  
Author(s):  
D. J. Philpott ◽  
J. D. Butzner ◽  
J. B. Meddings
Keyword(s):  
Small Intestine ◽  
Glucose Transport ◽  
Intestinal Adaptation ◽  
Basolateral Membrane ◽  
Intestinal Transport ◽  
Glucose Absorption ◽  
Dietary Carbohydrate ◽  
Adaptive Responses ◽  
Carbohydrate Levels ◽  
Specific Regulation

The small intestine is capable of adapting nutrient transport in response to numerous stimuli. This review examines several possible mechanisms involved in intestinal adaptation. In some cases, the enhancement of transport is nonspecific, that is, the absorption of many nutrients is affected. Usually, increased transport capacity in these instances can be attributed to an increase in intestinal surface area. Alternatively, some conditions induce specific regulation at the level of the enterocyte that affects the transport of a particular nutrient. Since the absorption of glucose from the intestine is so well characterized, it serves as a useful model for this type of intestinal adaptation. Four potential sites for the specific regulation of glucose transport have been described, and each is implicated in different situations. First, mechanisms at the brush-border membrane of the enterocyte are believed to be involved in the upregulation of glucose transport that occurs in streptozotocin-induced diabetes mellitus and alterations in dietary carbohydrate levels. Also, factors that increase the sodium gradient across the enterocyte may increase the rate of glucose transport. It has been suggested that an increase in activity of the basolaterally located Na+–K+ ATPase could be responsible for this phenomena. The rapid increase in glucose uptake seen in hyperglycemia seems to be mediated by an increase in both the number and activity of glucose carriers located at the basolateral membrane. More recently, it was demonstrated that mechanisms at the basolateral membrane also play a role in the chronic increase in glucose transport observed when dietary carbohydrate levels are increased. Finally, alterations in tight-junction permeability enhance glucose absorption from the small intestine. The possible signals that prompt these adaptive responses in the small intestine include glucose itself and humoral as well as enteric nervous interactions.Key words: intestinal transport, glucose transport, intestinal adaptation.

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