scholarly journals Contralateral Body Half Hypalgesia in a Patient with Lateral Medullary Infarction: Atypical Wallenberg Syndrome

2008 ◽  
Vol 59 (3-4) ◽  
pp. 211-215 ◽  
Author(s):  
She-Qing Zhang ◽  
Ming-Yuan Liu ◽  
Bo Wan ◽  
Hui-Min Zheng
Keyword(s):  
Lateral Medullary Infarction ◽  
Wallenberg Syndrome ◽  
Medullary Infarction

scholarly journals Wallenberg Syndrome with Associated Motor Weakness in a Two-Week-Postpartum Female

2015 ◽  
Vol 7 (3) ◽  
pp. 186-190 ◽  
Author(s):  
David W. Louis ◽  
Nimit Dholakia ◽  
Michael J. Raymond
Keyword(s):  
Inpatient Rehabilitation ◽  
Acute Onset ◽  
Middle Cerebellar Peduncle ◽  
Lateral Medullary Infarction ◽  
Gradual Improvement ◽  
Motor Weakness ◽  
Wallenberg Syndrome ◽  
Inpatient Rehabilitation Facility ◽  
Cerebellar Peduncle ◽  
Medullary Infarction

A 30-year-old, right-handed female presented 2 weeks postpartum with acute-onset severe headache, vertigo, and vomiting. Initial neurologic examination illustrated lingual dysarthria, horizontal nystagmus, right dysmetria on finger-to-nose testing, and weakness of the extremities. Magnetic resonance imaging showed a large, left lateral medullary infarction (Wallenberg syndrome) with cephalad extension into the ipsilateral pons as well as involvement of the left middle cerebellar peduncle. The patient was discharged 3 weeks later to an inpatient rehabilitation facility with gradual improvement of her symptoms.

scholarly journals Central Pain Mimicking Trigeminal Neuralgia as a Result of Lateral Medullary Ischemic Stroke

2019 ◽  
Vol 2019 ◽  
pp. 1-3
Author(s):  
Abinayaa Ravichandran ◽  
Kareem S. Elsayed ◽  
Hussam A. Yacoub
Keyword(s):  
Ischemic Stroke ◽  
Case Report ◽  
Trigeminal Neuralgia ◽  
Trigeminal Nerve ◽  
Facial Pain ◽  
Central Pain ◽  
Lateral Medullary Infarction ◽  
Wallenberg Syndrome ◽  
Work Up ◽  
Medullary Infarction

Background. Central pain mimicking trigeminal neuralgia (TN) as a result of lateral medullary infarction or Wallenberg syndrome has been rarely reported. Case Report. We discuss a patient who presented with a lateral medullary infarct and shortly after developed facial pain mimicking TN. We also elaborate on the anatomical pathway of the trigeminal nerve explaining facial pain as a result of a lateral medullary lesion. Discussion. Clinicians should be aware of this typical complication of lateral medullary infarct in order to attain proper management and work-up.

Effect of Transcutaneous Vagus Nerve Stimulation in Dysphagia After Lateral Medullary Infarction: A Case Report

2019 ◽  
Vol 28 (4) ◽  
pp. 1381-1387
Author(s):  
Ying Yuan ◽  
Jie Wang ◽  
Dongyu Wu ◽  
Dahua Zhang ◽  
Weiqun Song
Keyword(s):  
Vagus Nerve ◽  
Nerve Stimulation ◽  
Vagus Nerve Stimulation ◽  
Rating Scale ◽  
Tube Feeding ◽  
Nasogastric Feeding ◽  
Lateral Medullary Infarction ◽  
Severe Dysphagia ◽  
Transcutaneous Vagus Nerve Stimulation ◽  
Medullary Infarction

Purpose Severe dysphagia with weak pharyngeal peristalsis after dorsal lateral medullary infarction (LMI) requires long-term tube feeding. However, no study is currently available on therapeutic effectiveness in severe dysphagia caused by nuclear damage of vagus nerve after dorsal LMI. The purpose of the present investigation was to explore the potential of transcutaneous vagus nerve stimulation (tVNS) to improve severe dysphagia with weak pharyngeal peristalsis after dorsal LMI. Method We assessed the efficacy of 6-week tVNS in a 28-year-old woman presented with persisting severe dysphagia after dorsal LMI who had been on nasogastric feeding for 6 months. tVNS was applied for 20 min twice a day, 5 days a week, for 6 weeks. The outcome measures included saliva spitted, Swallow Function Scoring System, Functional Oral Intake Scale, Clinical Assessment of Dysphagia With Wallenberg Syndrome, Yale Pharyngeal Residue Severity Rating Scale, and upper esophagus X-ray examination. Results After tVNS, the patient was advanced to a full oral diet without head rotation or spitting. No saliva residue was found in the valleculae and pyriform sinuses. Contrast medium freely passed through the upper esophageal sphincter. Conclusion Our findings suggest that tVNS might provide a useful means for recovery of severe dysphagia with weak pharyngeal peristalsis after dorsal LMI. Supplemental Material https://doi.org/10.23641/asha.9755438

Spontaneous, headshaking, and positional nystagmus in post-lateral medullary infarction dizziness

2016 ◽  
Vol 368 ◽  
pp. 249-253 ◽  
Author(s):  
Kazumitsu Amari ◽  
Yosuke Kudo ◽  
Kosuke Watanabe ◽  
Masahiro Yamamoto ◽  
Koji Takahashi ◽  
...  
Keyword(s):  
Positional Nystagmus ◽  
Lateral Medullary Infarction ◽  
Medullary Infarction

Lateral Medullary Infarction with Contralateral Segmental Dysesthesia and Ipsilateral Headache: A Case Report

2021 ◽  
Vol 17 ◽  
Author(s):  
Renjie Wang ◽  
Yankun Shao ◽  
Lei Xu
Keyword(s):  
Medulla Oblongata ◽  
Clinical Manifestations ◽  
Lateral Medullary Infarction ◽  
Case Presentation ◽  
Specific Sign ◽  
Temperature Sense ◽  
Magnetic Resonance Imaging Mri ◽  
The Right ◽  
The Brain ◽  
Medullary Infarction

Introduction: The medulla oblongata is the lowest segment of the brain stem, located adjacent to the spinal cord, with a complex anatomical structure. Thus, a small injury to the medulla oblongata can show complex clinical manifestations. Case Presentation: A patient experienced dysesthesia, which manifested as numbness in her right lower limb and decreased temperature sense, and dizziness 20 days before admission. The numbness worsened 1 week before admission, reaching the right thoracic (T) 12 dermatomes. Her thermoception below the T12 dermatomes decreased, and the degree of dizziness increased, accompanied by nausea and vomiting. Magnetic resonance imaging (MRI) of the neck, chest, and abdomen performed at a local hospital showed no abnormalities. MRI of the brain was performed after admission. One week after admission, she experienced a severe headache in the upper left periorbital area. The numbness extended to T4, and thermoception decreased below T4. Diagnosis: Lateral medullary infarction. Interventions: Anti-platelet aggregation and mitochondrial nutritional therapies were performed along with treatments for improving circulation and establishing collateral circulation. Outcomes: The intensity of limb numbness decreased, and the symptoms of headache and dizziness resolved. Conclusion: Lesions leading to segmental sensory disorders can occur in the medulla oblongata. Ipsilateral headaches with contralateral segmental paresthesia can be a specific sign of lateral medullary infarction.

Characteristics and Prognostic Factors of Swallowing Dysfunction in Patients with Lateral Medullary Infarction

2021 ◽  
Vol 30 (12) ◽  
pp. 106122
Author(s):  
Naoyuki Hara ◽  
Masahiro Nakamori ◽  
Tomoko Ayukawa ◽  
Hayato Matsushima ◽  
Yuki Hayashi ◽  
...  
Keyword(s):  
Prognostic Factors ◽  
Lateral Medullary Infarction ◽  
Swallowing Dysfunction ◽  
Medullary Infarction
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