Endogenous Hypertriglyceridemia Intensifies the Course of Cerulein-Induced Pancreatitis in Rat: Relation with Changes in the VLDL Composition

2006 ◽  
Vol 50 (1) ◽  
pp. 37-44 ◽  
Author(s):  
O. Ponzo ◽  
L. Schreier ◽  
R. Resnik ◽  
G. Negri ◽  
P. Scacchi ◽  
...  
Keyword(s):  
Endogenous Hypertriglyceridemia

Growth Hormone Secretion in Primary Endogenous Hypertriglyceridemia

1973 ◽  
Vol 5 (03) ◽  
pp. 224-225 ◽  
Author(s):  
M. Muggeo ◽  
D. Fedele ◽  
A. Tiengo ◽  
G. Crepaldi
Keyword(s):  
Growth Hormone ◽  
Hormone Secretion ◽  
Growth Hormone Secretion ◽  
Endogenous Hypertriglyceridemia

Plasminogen activator inhibitor activity in various types of endogenous hypertriglyceridemia

1993 ◽  
Vol 7 (3) ◽  
pp. 171-176 ◽  
Author(s):  
D. Raccah ◽  
M.C. Alessi ◽  
V. Scelles ◽  
C. Menard ◽  
I. Juhan-Vague ◽  
...  
Keyword(s):  
Plasminogen Activator ◽  
Plasminogen Activator Inhibitor ◽  
Inhibitor Activity ◽  
Endogenous Hypertriglyceridemia ◽  
Activator Inhibitor

Hepatic steatosis and the elevated plasma insulin level in patients with endogenous hypertriglyceridemia

Metabolism ◽  
1975 ◽  
Vol 24 (5) ◽  
pp. 653-664 ◽  
Author(s):  
Yoshisuke Maruhama ◽  
Akira Ohneda ◽  
Hiroshi Tadaki ◽  
Masao Ohtsuki ◽  
Akira Yanbe ◽  
...  
Keyword(s):  
Hepatic Steatosis ◽  
Plasma Insulin ◽  
Insulin Level ◽  
Plasma Insulin Level ◽  
Elevated Plasma ◽  
Endogenous Hypertriglyceridemia

Clinical Studies Concerning Factor XIII; with Special Reference to Hyperlipemia

1973 ◽  
Vol 30 (03) ◽  
pp. 480-493 ◽  
Author(s):  
Mircea P. Cucuianu ◽  
Vasile V. Vasile ◽  
Titus A. Popescu ◽  
Augustin Opincaru ◽  
Ion Crîsnic ◽  
...  
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Special Reference ◽  
Clinical Studies ◽  
Factor Xiii ◽  
Normal Weight ◽  
Cirrhosis Of The Liver ◽  
Control Subjects ◽  
Low Levels ◽  
Endogenous Hypertriglyceridemia

SummaryFactor XIII, estimated according to the method of Heene, was found to be diminished in patients with cirrhosis of the liver, in acute renal failure and, to a lesser extent, postoperatively, owing probably to both a decreased synthesis and an accelerated utilization. When compared to values obtained in healthy, normal weight, normolipemic control subjects, the level of factor XIII was higher in hyperlipemic and especially in hypertriglyceridemic patients, whether clinical atherosclerosis was present or not. Low levels of factor XIII were usually accompanied by a decrease in serum pseudocholinesterase, while high values of this enzyme produced by the liver were noted in hypertriglyceridemic patients. The hypothesis of an enhanced synthesis of factor XIII by the liver, in subjects with endogenous hypertriglyceridemia is considered. It is also suggested that accelerated stabilization of fibrin might contribute to the fibrinolytic insufficiency of hyperlipemic patients and enhance certain atherogenic mechanisms.

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