Necrotizing External Otitis and Chromobacterium violaceum

2001 ◽  
Vol 11 (6) ◽  
pp. 306-307
Author(s):  
H.S. Lawrence ◽  
A.G. Tucker
Keyword(s):  
Chromobacterium Violaceum ◽  
External Otitis ◽  
Necrotizing External Otitis

Unusual case of VIII nerve dysfunction on necrotizing external otitis

2020 ◽  
Vol 69 (4) ◽  
Author(s):  
Diogo A. Pereira ◽  
Luís Guedes ◽  
Abílio Leonardo ◽  
Delfim Duarte ◽  
Gustavo Lopes
Keyword(s):  
Unusual Case ◽  
External Otitis ◽  
Nerve Dysfunction ◽  
Necrotizing External Otitis

scholarly journals NECROTIZING EXTERNAL OTITIS IN CHILDREN

1987 ◽  
Vol 97 (5) ◽  
pp. 598???601 ◽  
Author(s):  
STEVEN SOBIE ◽  
LINDA BRODSKY ◽  
JOHN F. STANIEVICH
Keyword(s):  
External Otitis ◽  
Necrotizing External Otitis

scholarly journals Necrotizing External Otitis in a Child

1979 ◽  
Vol 72 (1) ◽  
pp. 21-26
Author(s):  
Kiyofumi Gyo ◽  
Yasushi Matsumoto ◽  
Hiroshi Okamura ◽  
Naoaki Yanagihara
Keyword(s):  
External Otitis ◽  
Necrotizing External Otitis

Biocompatible implantable antimicrobial release for necrotizing external otitis

1991 ◽  
Vol 105 (4) ◽  
pp. 252-256
Author(s):  
E. J. Ostfeld ◽  
A. Kupferberg
Keyword(s):  
Cardiac Arrest ◽  
Clinical Symptoms ◽  
Sudden Cardiac Arrest ◽  
The Other ◽  
External Otitis ◽  
Therapeutic Modality ◽  
Antibiotic Administration ◽  
Post Implantation ◽  
Necrotizing External Otitis

AbstractThe efficacy of a biocompatible, surgically implantable, antimicrobial release system (IARS) as the exclusive antimicrobial therapy of necrotizing external otitis (NEO) was evaluated in six NEO patients. Gentamicin incorporated polymethyl-methacrylate beads were implanted, following surgical debridement and were removed two months later. Post-implantation alleviation of clinical symptoms: pain, periauricular tissue swelling, otorrhoea, eradication of pseudomonal infection (100 per cent) and substantially shortened hospitalization (4–15 days) were the salient results of this therapeutic modality. Three patients recovered. Two patients who died, one of sudden cardiac arrest and the other of paralytic ileus, 15 and 60 days postoperatively while the beads were still implanted, were symptomless. Recurrence was seen in one patient with early bead extrusion. Ipsilateral sensorineural hearing loss (one patient) and external meatal stenosis were the main complications. IARS appears to offer an effective alternative to long-term systemic antibiotic administration for the eradication of NEO-pseudomonal infection in patients who are sensitive, develop resistance, or when quinolone medical treatment has failed or is contra-indicated.

Necrotizing External Otitis

2007 ◽  
Vol 28 (6) ◽  
pp. 771-773 ◽  
Author(s):  
Valerie Franco-Vidal ◽  
Harold Blanchet ◽  
C??cile Bebear ◽  
Herv?? Dutronc ◽  
Vincent Darrouzet
Keyword(s):  
External Otitis ◽  
Necrotizing External Otitis

Necrotizing external otitis: a severe form of otitis in diabetic patients

2019 ◽  
Author(s):  
Monia Ghammem ◽  
Jihene Houas ◽  
Mouna Bellakhdher ◽  
Abir Meherzi ◽  
Wassim Kermani ◽  
...  
Keyword(s):  
Severe Form ◽  
External Otitis ◽  
Diabetic Patients ◽  
Necrotizing External Otitis

Temporal Bone Histopathology of Necrotizing External Otitis

1981 ◽  
Vol 90 (2) ◽  
pp. 109-115 ◽  
Author(s):  
Isamu Sando ◽  
Takehiko Harada ◽  
Reisuke Saito ◽  
Yasumasa Okano ◽  
Ralph J. Caparosa
Keyword(s):  
Temporal Bone ◽  
Middle Ear ◽  
Granulation Tissue ◽  
Histopathological Examination ◽  
Internal Auditory Canal ◽  
Anterior Wall ◽  
External Otitis ◽  
Lateral Part ◽  
Necrotizing External Otitis ◽  
Infection Spread

Histopathological examination of the temporal bone of an individual with necrotizing external otitis revealed severe inflammation with necrosis in the subcutaneous granulation tissue in the external auditory canal. Erosion of the bony walls of the canal had created a defect in the anterior wall, through which infection spread to the preauricular region. The lateral part of the middle ear cavity was also filled with granulation tissue and purulent exudate. A dehiscence of the horizontal portion of the facial canal had apparently allowed spread of an inflammatory round cell infiltrate along the facial nerve up to the fundus of the internal auditory canal. The only evidence of inner ear pathology was the presence of eosinophilic fluid material in the perilymphatic spaces of the labyrinth. A review of other reports of cases of necrotizing external otitis seems to show that this is the only instance of this disease in which infection spread from the external canal through the tympanic membrane to the middle ear and thence to the internal auditory canal.

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