Cold Pressure Test Producing Coronary Spasm, Coronary Thrombosis and Myocardial Infarction in a Patient with IgM Antibodies against Coxsackie B Virus

Cardiology ◽  
1999 ◽  
Vol 92 (4) ◽  
pp. 278-281
Author(s):  
Werner Haberbosch ◽  
Norbert Roerich ◽  
Joerg Neuzner
Keyword(s):  
Myocardial Infarction ◽  
Coronary Thrombosis ◽  
Coronary Spasm ◽  
Igm Antibodies ◽  
B Virus ◽  
Pressure Test ◽  
Coxsackie B Virus ◽  
Cold Pressure ◽  
Cold Pressure Test ◽  
Coxsackie B

Coxsackie B virus and myocardial infarction

The Lancet ◽  
2002 ◽  
Vol 359 (9308) ◽  
pp. 804 ◽  
Author(s):  
Harold D Foster
Keyword(s):  
Myocardial Infarction ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B

Coxsackie B Virus Antibodies in Myocardial Infarction

2009 ◽  
Vol 214 (1) ◽  
pp. 29-32 ◽  
Author(s):  
JUKKA NIKOSKELAINEN ◽  
J. LENNART KALLIOMÁKI ◽  
KAISA LAPINLEIMU ◽  
MIRJA STENVIK ◽  
PEKKA E. HALONEN
Keyword(s):  
Myocardial Infarction ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B

Coxsackie B Viruses and the Post Viral Syndrome in Nuclear Submariners

1988 ◽  
Vol 74 (2) ◽  
pp. 95-95
Author(s):  
P. J. Warnock ◽  
E. J. Bell ◽  
M. H. Riding
Keyword(s):  
Immunosorbent Assay ◽  
Enzyme Linked Immunosorbent Assay ◽  
Igm Antibodies ◽  
Fatigue Syndrome ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B Viruses ◽  
Antibody Capture ◽  
Coxsackie B

SummarySera from 168 submariners, the entire crew of a Polaris submarine, were tested for Coxsackie B Virus (CBV) specific IgM antibodies using a µ antibody capture enzyme linked immunosorbent assay (ELISA). 36 (21%) were positive or weak positive with 22 (13%) confirmed by CBV neutralisation tests (NT) suggesting recent or active CBV infection. The sailors were observed over a two year period during which three patients (2%) developed illnesses suggestive of the post viral fatigue syndrome. These cases, all previously seronegative for CBV did not seroconvert once they became unwell and their illnesses are presumed to have been due to other viruses. The prevalence of CBV infections and the post viral fatigue syndrome in this close and confined community are discussed.

COXSACKIE B VIRUS INFECTIONS AND MYOCARDIAL INFARCTION

The Lancet ◽  
1980 ◽  
Vol 315 (8183) ◽  
pp. 1387-1389 ◽  
Author(s):  
P.D. Griffiths ◽  
Gillian Hannington ◽  
J.C. Booth
Keyword(s):  
Myocardial Infarction ◽  
Virus Infections ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B

scholarly journals Coxsackie B virus-specific IgM antibody and myocardial infarction

1986 ◽  
Vol 21 (4) ◽  
pp. 287-291 ◽  
Author(s):  
G. Hannington ◽  
J. C. Booth ◽  
R. J. Bowes ◽  
H. Stern
Keyword(s):  
Myocardial Infarction ◽  
Igm Antibody ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B

Myocarditis in Children

1987 ◽  
Vol 9 (3) ◽  
pp. 83-88
Author(s):  
Arno R. Hohn ◽  
Robert E. Stanton
Keyword(s):  
Acute Myocarditis ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Coxsackie B

Acute myocarditis may go undetected. Nearly half of the known cases of myocarditis are caused by coxsackie B virus. Findings in myocarditis are often nonspecific. Echo cardiograms show reduced function. Some cases of myocarditis progress to cardiomyopathy.

TRANSIENT SYNOVITIS OF THE HIP JOINT IN CHILDREN

PEDIATRICS ◽  
1959 ◽  
Vol 24 (6) ◽  
pp. 1042-1049 ◽  
Author(s):  
Alexander Spock
Keyword(s):  
Hip Joint ◽  
Streptococcal Infection ◽  
Weight Bearing ◽  
Bed Rest ◽  
Transient Synovitis ◽  
Treatment Results ◽  
B Virus ◽  
Coxsackie B Virus ◽  
Roentgenographic Examination ◽  
Coxsackie B

A clinical review of 47 eases of transient synovitis of the hip joint occurring in patients less than 14 years of age is presented. Evidence from clinical, bacteriologic and serologic data is presented to show that in four patients acute streptococcal infection was associated with the development of transient synovitis. In one other patient clinical and serologic data disclosed a similar parallelism with an infection by the Coxsackie B virus. These findings suggest that these agents may be among those etiologically responsible for this syndrome. An obese or stocky physique probably predisposes a child to this disease. Patients with transient synovitis do not have any diagnostic abnormalities which can be detected by roentgenographic examination. Prevention of weight bearing by bed rest until the patient is completely asymptomatic provides the best form of therapy, and failure to follow such treatment results in prolongation of the illness. Antibiotics do not appear to influence the course of the disease. Three patients in this series developed Legg-Perthes disease within an interval of 3 to 17 months after convalescing from transient synovitis of the hip.

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