scholarly journals Corrigendum to “Dietary Magnesium Intake and Leukocyte Telomere Attrition in Adults: The Regulatory Role of Serum Tumor Necrosis Factor α”

2020 ◽  
Vol 2020 ◽  
pp. 1-1
Author(s):  
Jie Yu ◽  
Haibin Liu ◽  
Shuli He ◽  
Pingping Li ◽  
Chunxiao Ma ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Α ◽  
Telomere Attrition ◽  
Magnesium Intake ◽  
Dietary Magnesium ◽  
Factor Α ◽  
Necrosis Factor ◽  
Serum Tumor Necrosis

scholarly journals Fcγ-receptor signaling augments the LPS-stimulated increase in serum tumor necrosis factor-α levels

2001 ◽  
Vol 280 (4) ◽  
pp. R1037-R1044 ◽  
Author(s):  
Marion L. Refici ◽  
Dennis W. Metzger ◽  
Bernard P. Arulanandam ◽  
Michelle R. Lennartz ◽  
Daniel J. Loegering
Keyword(s):  
Tumor Necrosis Factor ◽  
Complement Activation ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Α ◽  
Fcγ Receptor ◽  
Tnf Α ◽  
Factor Α ◽  
Necrosis Factor ◽  
Serum Tumor Necrosis

The phagocytosis of IgG-coated erythrocytes (EIgG) has been shown to augment the bacterial lipopolysaccharide (LPS)-stimulated increase in serum tumor necrosis factor-α (TNF-α) levels. The present study evaluated the role of Fcγ-receptor (FcγR) signaling and complement activation in the effect of EIgG on the TNF-α response to LPS. The role of FcγR was determined using FcR γ-chain knockout mice that lack functional FcγRI and FcγRIII. In wild-type animals, EIgG caused a 16-fold augmentation of the serum TNF-α response to LPS, whereas there was no augmentation in the FcγR-deficient animals. Heat-damaged erythrocytes also augmented the TNF-α response to LPS. This effect was absent in FcγR-deficient animals. An IgG antibody against heated erythrocytes was detected in mouse serum. The complement activation caused by EIgG had little effect on the LPS-stimulated increase in serum TNF-α levels as indicated by activation of complement with cobra venom factor or IgM-coated erythrocytes as well as studies with C5-deficient mice. These results indicate that FcγR signaling primarily mediates the augmented serum TNF-α response to LPS caused by EIgG.

The Role of Tumor Necrosis Factor-α (TNF-α) Polymorphisms in Gastric Cancer: a Meta-Analysis

2021 ◽  
Author(s):  
Maryam Gholamalizadeh ◽  
Samaneh Mirzaei Dahka ◽  
Hadi Sedigh Ebrahim-Saraie ◽  
Mohammad Esmail Akbari ◽  
Azam Pourtaheri ◽  
...  
Keyword(s):  
Gastric Cancer ◽  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Α ◽  
Meta Analysis ◽  
Tnf Α ◽  
Factor Α ◽  
Necrosis Factor

Mo1860 - High Fecal and Serum Tumor Necrosis Factor-α Levels are Associated with Non-Response to Infliximab in Patients with Ulcerative Colitis

2018 ◽  
Vol 154 (6) ◽  
pp. S-829
Author(s):  
Hadar Meringer-Milwidsky ◽  
Nitsan Maharshak ◽  
Yulia Ron ◽  
Henit A. Yanai ◽  
Sarit Cohen-Kedar ◽  
...  
Keyword(s):  
Ulcerative Colitis ◽  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Α ◽  
Factor Α ◽  
Necrosis Factor ◽  
Serum Tumor Necrosis

scholarly journals Bidirectional Role of Tumor Necrosis Factor-α in Coronary Microembolization

2007 ◽  
Vol 100 (1) ◽  
pp. 140-146 ◽  
Author(s):  
Andreas Skyschally ◽  
Petra Gres ◽  
Simone Hoffmann ◽  
Michael Haude ◽  
Raimund Erbel ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Tumor Necrosis Factor Α ◽  
Coronary Microembolization ◽  
Factor Α ◽  
Necrosis Factor
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