scholarly journals Inflammation and Macular Oedema after Pars Plana Vitrectomy

2013 ◽  
Vol 2013 ◽  
pp. 1-8 ◽  
Author(s):  
Vito Romano ◽  
Martina Angi ◽  
Fabrizio Scotti ◽  
Renata del Grosso ◽  
Davide Romano ◽  
...  
Keyword(s):  
Macular Oedema ◽  
Pars Plana Vitrectomy ◽  
Extracellular Fluid ◽  
Cystoid Macular Oedema ◽  
Therapeutic Approaches ◽  
Fluid Accumulation ◽  
Primary Defect ◽  
Blood Retinal Barrier ◽  
Vasogenic Oedema ◽  
Pars Plana

Cystoid macular oedema (CMO) is a major cause of reduced vision following intraocular surgery. Although the aetiology of CMO is not completely clarified, intraocular inflammation is known to play a major role in its development. The macula may develop cytotoxic oedema when the primary lesion and fluid accumulation occur in the parenchymatous cells (intracellular oedema) or vasogenic oedema when the primary defect occurs in the blood-retinal barrier and leads to extracellular fluid accumulation (extracellular oedema). We report on the mechanisms of CMO formation after pars plana vitrectomy and associated surgical procedures and discuss possible therapeutic approaches.

scholarly journals Pars plana vitrectomy with intravitreal triamcinolone: effect on uveitic cystoid macular oedema and treatment limitations

2006 ◽  
Vol 91 (3) ◽  
pp. 345-348 ◽  
Author(s):  
M. Gutfleisch ◽  
G. Spital ◽  
A. Mingels ◽  
D. Pauleikhoff ◽  
A. Lommatzsch ◽  
...  
Keyword(s):  
Macular Oedema ◽  
Pars Plana Vitrectomy ◽  
Cystoid Macular Oedema ◽  
Intravitreal Triamcinolone ◽  
Pars Plana

Pathogenesis of Pseudophakic Cystoid Macular Oedema

2012 ◽  
Vol 06 (05) ◽  
pp. 290
Author(s):  
Conceição Lobo ◽  
Keyword(s):  
Optical Coherence Tomography ◽  
Cataract Surgery ◽  
Inflammatory Mediators ◽  
Diagnostic Tool ◽  
Macular Oedema ◽  
Cystoid Macular Oedema ◽  
Blood Retinal Barrier ◽  
Non Invasive ◽  
Operative Complications ◽  
Inflammatory Drugs

Cystoid macular oedema (CMO) is a primary cause of reduced vision after cataract surgery even after uneventful surgery. The incidence of clinical CMO following modern cataract surgery is 1.0-2.0 % but the high number of surgeries performed worldwide makes this entity an important problem. Pre-existing conditions such as diabetes and intra-operative complications increase the risk of developing CMO post-operatively. CMO is caused by an accumulation of intra-retinal fluid in the outer plexiform and inner nuclear layers of the retina, as a result of the breakdown of the blood-retinal barrier. The mechanisms that lead to this condition are not completely understood. However, the principal hypothesis is that the surgical procedure is responsible for the release of inflammatory mediators, such as prostaglandins. Optical coherence tomography is at present an extremely useful non-invasive diagnostic tool. Guidelines for the management CMO should be focused essentially on prevention and are based on the principal pathogenetic mechanisms, including the use of anti-inflammatory drugs.

Incidence of Retinal Detachment after Macular Surgery

2021 ◽  
Author(s):  
Luise Grajewski ◽  
Olaf Grajewski ◽  
Jens Carstens ◽  
Lothar Krause
Keyword(s):  
Retinal Detachment ◽  
Functional Outcomes ◽  
Pars Plana Vitrectomy ◽  
Cystoid Macular Oedema ◽  
Retinal Break ◽  
Macular Surgery ◽  
Surgical Methods ◽  
Pars Plana ◽  
The Mean ◽  
Mean Time

AbstractMacular surgery has become an increasingly atraumatic procedure for the eye with the surgical methods that have been further developed in recent years. The most common complications include cystoid macular oedema and retinal detachment, more rarely endophthalmitis. The aim of this retrospective study is to record the number of retinal detachments following elective macular surgery. In this study we included all patients who underwent pars plana vitrectomy (ppV, 20 or 25 gauge) in the years 2009 – 2016. We then identified the patients who were hospitalised again because of retinal detachment. For the affected patients, the rate of retinal detachment, functional outcomes and possible risk factors were recorded. A total of 904 eyes were identified, of which 667 had surgery for epiretinal membrane, 188 for macular hole, and 49 for vitreomacular traction with a 20 or 25 gauge ppV. Of these 904, retinal detachment occurred in 17 (1.88%) cases. The mean time between first ppV and second ppV with retinal detachment was 248 days (3 – 1837 days). Two of the 17 patients had at least one retinal break before or during surgery. The retinal break was located inferior in six cases, superior in four; in four cases PVR retinal detachment and in three cases the foramina were distributed. Mean visual acuity was 0.27 (decimal) before macular surgery and 0.28 at the time of last presentation. Modern vitrectomy techniques reduce the complications in elective macular surgery, but do not replace the surgeonʼs experience.

scholarly journals Pars plana vitrectomy for diabetic macular oedema: OCT and functional correlations

Eye ◽  
2005 ◽  
Vol 20 (6) ◽  
pp. 674-680 ◽  
Author(s):  
J I Patel ◽  
P G Hykin ◽  
M Schadt ◽  
V Luong ◽  
F Fitzke ◽  
...  
Keyword(s):  
Macular Oedema ◽  
Pars Plana Vitrectomy ◽  
Diabetic Macular Oedema ◽  
Pars Plana

Pathogenesis of Pseudophakic Cystoid Macular Oedema

2012 ◽  
Vol 06 (03) ◽  
pp. 178
Author(s):  
Conceição Lobo ◽  
Keyword(s):  
Optical Coherence Tomography ◽  
Cataract Surgery ◽  
Inflammatory Mediators ◽  
Diagnostic Tool ◽  
Macular Oedema ◽  
Cystoid Macular Oedema ◽  
Blood Retinal Barrier ◽  
Non Invasive ◽  
Operative Complications ◽  
Inflammatory Drugs

Cystoid macular oedema (CMO) is a primary cause of reduced vision after cataract surgery even after uneventful surgery. The incidence of clinical CMO following modern cataract surgery is 1.0–2.0 % but the high number of surgeries performed worldwide makes this entity an important problem. Pre-existing conditions such as diabetes and intra-operative complications increase the risk of developing CMO post-operatively. CMO is caused by an accumulation of intra-retinal fluid in the outer plexiform and inner nuclear layers of the retina, as a result of the breakdown of the blood–retinal barrier. The mechanisms that lead to this condition are not completely understood. However, the principal hypothesis is that the surgical procedure is responsible for the release of inflammatory mediators, such as prostaglandins. Optical coherence tomography is at present an extremely useful non-invasive diagnostic tool. Guidelines for the management CMO should be focused essentially on prevention and are based on the principal pathogenetic mechanisms, including the use of anti-inflammatory drugs.

scholarly journals Diabetic macular oedema: pilot randomised trial of pars plana vitrectomy vs macular argon photocoagulation

Eye ◽  
2005 ◽  
Vol 20 (8) ◽  
pp. 873-881 ◽  
Author(s):  
J I Patel ◽  
P G Hykin ◽  
M Schadt ◽  
V Luong ◽  
C Bunce ◽  
...  
Keyword(s):  
Macular Oedema ◽  
Pars Plana Vitrectomy ◽  
Randomised Trial ◽  
Diabetic Macular Oedema ◽  
Pars Plana

Pars plana vitrectomy and removal of the internal limiting membrane in the treatment of chronic macular oedema

2004 ◽  
Vol 242 (10) ◽  
pp. 845-852 ◽  
Author(s):  
Remzi Avci ◽  
Berkant Kaderli ◽  
Berrin Avci ◽  
Saban Simsek ◽  
Mehmet Baykara ◽  
...  
Keyword(s):  
Macular Oedema ◽  
Pars Plana Vitrectomy ◽  
Internal Limiting Membrane ◽  
Pars Plana
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