scholarly journals Observing and understanding arterial and venous circulation differences in a physiology laboratory activity

2015 ◽  
Vol 39 (4) ◽  
pp. 405-410 ◽  
Author(s):  
Caroline Altermann ◽  
Rithiele Gonçalves ◽  
Marcus Vinícius S. Lara ◽  
Ben-Hur S. Neves ◽  
Pâmela B. Mello-Carpes

The purpose of the present article is to describe three simple practical experiments that aim to observe and discuss the anatomic and physiological functions and differences between arteries and veins as well as the alterations observed in skin blood flow in different situations. For this activity, students were divided in small groups. In each group, a volunteer is recruited for each experiment. The experiments only require a sphygmomanometer, rubber bands, and a clock and allow students to develop a hypothesis to explain the different responses to the interruption of arterial and venous blood flow. At the end, students prepare a short report, and the results are discussed. This activity allows students to perceive the presence of physiology in their daily lives and helps them to understand the concepts related to the cardiovascular system and hemodynamics.

2016 ◽  
Author(s):  
Bart Weijts ◽  
Edgar Gutierrez ◽  
Semion K Saikin ◽  
Ararat J Ablooglu ◽  
David Traver ◽  
...  

Arteries and veins are formed independently by different types of endothelial cells (ECs). In vascular remodeling, arteries and veins become connected and some arteries become veins. It is unclear how ECs in transforming vessels change their type and how fates of individual vessels are determined. In embryonic trunk, vascular remodeling transforms arterial intersegmental vessels (ISVs) into a functional network of arteries and veins. We found that, once an ISV is connected to venous circulation, venous blood flow promotes upstream migration of ECs that results in displacement of arterial ECs by venous ECs, completing the transformation of this ISV into a vein without trans-differentiation of ECs. Arterial blood flow initiated in two neighboring ISVs prevents their transformation into veins by activating Notch signaling in ECs. Together, different responses of ECs to arterial and venous blood flow lead to the formation of a balanced network with equal numbers of arteries and veins.


Author(s):  
Vladimir Usachev ◽  
Pierre-Marie Gagey

Background: In the 90s, Inamura et al. have drawn our attention to the role played by the postural system in the return venous circulation, thanks to plethysmographic recordings which gave free rein to the imagination of the authors to suppose the functioning mechanisms of this venous pump. In 2010, two anatomists, Uhl & Gillot, transformed our representation of the venous network and made assumptions about the functioning of the venous pump. Objective/ Methods: The aim of this work is to verify these hypotheses by scanning the venous blood flow at the level of the sinus soleus and the popliteal vein during various posture-kinetic situations. Conclusion: These studies fully confirm the hypotheses. It is therefore likely that the postural system intervenes not only in the phenomena of stabilisation of the body, but also in the back venous circulation.


2021 ◽  
Vol 17 (1) ◽  
pp. 42-49
Author(s):  
O.Ye. Kovalenko ◽  
N.G. Prityko

The article presents an analytical review of the results of domestic and foreign studies on chronic disorders of cerebral venous circulation, which is given less attention against the arterial system due to blurred manifestations, especially early, anatomical variability of the venous system, even in healthy, difficulty in diagnosis. When the balance of arterial and venous inflow is disturbed and this state exa­cerbates, irreversible changes evolve other structures of the cranial ca­vity — primarily in the brain (consistent with the concept of Monroe-Kelly). Chronic disorders of cerebral venous circulation may have different causes and varying degrees of severity. The equivalent of chronic cerebral venous insufficiency (SCVD), terminologically accepted in our country, in some countries is considered as chronic venous cerebrospinal insufficiency, which emphasizes the pathogenetic link of disorders of cerebral venous blood flow and extracranial veins, which is, in our opinion, is logical and reasonable. Recognition of chronic cerebrospinal venous insufficiency has aroused intense interest for a better understanding of the role of extracranial venous pathology and developmental options. Although the diagnosis was originally based on Doppler sonography, there are currently no diagnostic imaging methods, non-invasive or invasive, that can serve as the gold standard for detecting venous abnormalities indicative of chronic cerebrospinal venous insufficiency. The results of some studies are discussed, in particular, the hypothesis that chronic cerebrospinal venous insufficiency plays a role in the pathoge-nesis of multiple sclerosis or in many concomitant clinical manifestations. The affinity of the pathogenesis of idiopathic intracranial hypertension, Ménière’s disease, spondylotic vertebrobasilar insufficiency syndrome is analyzed. Attention is also focused on the other opinion of scientists when the increase in venous blood supply to the brain and other formations in the head cavity under certain conditions is considered as a compensatory reaction. The anatomical and physiological features of the venous system of the cranial cavity are considered, which substantiate the clinical manifestations of venous dysfunction, methods of diagnosis and treatment of pathology. Summarizing the above, chronic venous blood circulation in the cranial cavity, which is largely associated with problems of extracranial venous blood flow, not only in itself is a very characteristic syndrome complex t that adversely affects neurological functions due to cerebral hypoxia and metabolic changes, deteriorates the quality of life, but also has a negative impact on the course of the disease or has a pathogenetic link with other diseases, in particular, may be accompanied by different levels of blood pressure.


JAMA ◽  
1966 ◽  
Vol 198 (7) ◽  
pp. 784-785 ◽  
Author(s):  
A. Neistadt

2009 ◽  
Vol 8 (2,3) ◽  
pp. 125-132 ◽  
Author(s):  
Erik Levin ◽  
Deborah Macintosh ◽  
Tanya Baker ◽  
Mark Weatherall ◽  
Richard Beasley

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