Bow Hunter's Syndrome

2021 ◽  
Author(s):  
Jasmine L. Taylor, MS ◽  
Ahmed Rashad, MD
Keyword(s):  
1998 ◽  
Vol 15 (5) ◽  
pp. 370-373 ◽  
Author(s):  
DM Thappa ◽  
A Singh ◽  
TJ Jaisankar ◽  
R Rao ◽  
C Ratnakar
Keyword(s):  

PEDIATRICS ◽  
1974 ◽  
Vol 54 (4) ◽  
pp. 523-523
Author(s):  
Carol W. Booth ◽  
Henry L. Nadler

We have read with interest the comments of Dr. Kelly regarding the theoretical usefulness of the techniques recently described for the demonstration of the heterozygous state in Hunter's syndrome. Dr. Kelly has suggested that this technique, which might permit certain females to be identified as carriers of Hunter's syndrome, would not significantly alter current genetic counseling concepts nor their application to amniocentesis for the prenatal detection of this disorder. Theoretical questions raised in his letten are certainly sound.


2021 ◽  
Author(s):  
Nickalus R Khan ◽  
Turki Elarjani ◽  
Stephanie H Chen ◽  
Laszlo Miskolczi ◽  
Sheryl Strasser ◽  
...  

Abstract Rotational vertebral artery (VA) occlusion syndrome, also known as bow hunter's syndrome, is an uncommon variant of vertebrobasilar insufficiency typically occurring with head rotation.1-3 The most common presenting symptom is dizziness (76.8%), followed by visual abnormalities and syncope (50.4% and 40.4%, respectively).2 Osteophytic compression due to spinal spondylosis has been shown to be the most common etiology (46.2%), with other factors, such as a fibrous band, muscular compression, or spinal instability, being documented.1,2 Treatment is dependent on the level and site of VA compression with anterior, anterolateral, or posterior approaches being described.1,4 We present the case of a 72-yr-old male with osteophytic compression of the V3 segment of the vertebral artery at the occipital-cervical junction. The patient underwent a C1 hemilaminectomy and removal of osteophytic compression from the occipital-cervical joint. The patient had complete resolution of compression of his vertebral artery on postoperative imaging and remained neurologically intact following the procedure. We review the literature on this topic, the technical nuances of the procedure performed, and review the different treatment modalities available for this rare condition.1-11  The patient consented to the procedure and to publication of their image.


2021 ◽  
Vol 12 ◽  
Author(s):  
Noriya Enomoto ◽  
Kenji Yagi ◽  
Shunji Matsubara ◽  
Masaaki Uno

Bow hunter's syndrome (BHS) is most commonly caused by compression of the vertebral artery (VA). It has not been known to occur due to an extracranially originated posterior inferior cerebellar artery (PICA), the first case of which we present herein. A 71-year-old man presented with reproducible dizziness on leftward head rotation, indicative of BHS. On radiographic examination, the bilateral VAs merged into the basilar artery, and the left VA was predominant. The right PICA originated extracranially from the right VA at the atlas–axis level and ran vertically into the spinal canal. During the head rotation that induced dizziness, the right PICA was occluded, and a VA stenosis was revealed. Occlusion of the PICA was considered to be the primary cause of the dizziness. The patient underwent surgery to decompress the right PICA and VA via a posterior cervical approach. Following surgery, the patient's dizziness disappeared, and the stenotic change at the right VA and PICA improved. The PICA could be a causative artery for BHS when it originates extracranially at the atlas–axis level, and posterior decompression is an effective way to treat it.


2009 ◽  
Vol 52 (04) ◽  
pp. 193-195 ◽  
Author(s):  
K. Sugiu ◽  
T. Agari ◽  
K. Tokunaga ◽  
A. Nishida ◽  
I. Date

2017 ◽  
Vol 26 (2) ◽  
pp. 199-202 ◽  
Author(s):  
Vivek P. Buch ◽  
Peter J. Madsen ◽  
Kerry A. Vaughan ◽  
Paul F. Koch ◽  
David K. Kung ◽  
...  

Rotational vertebrobasilar insufficiency, or bow hunter's syndrome, is a rare cause of posterior circulation ischemia, which, following rotation of the head, results in episodic vertigo, dizziness, nystagmus, or syncope. While typically caused by dynamic occlusion of the vertebral artery in its V2 and V3 segments, the authors here describe a patient with dynamic occlusion of the vertebral artery secondary to a persistent first intersegmental artery, a rare variant course of the vertebral artery. In this case the vertebral artery coursed under rather than over the posterior arch of the C-1. This patient was also found to have incomplete development of the posterior arch of C-1, as is often seen with this variant. The patient underwent dynamic digital subtraction angiography, which demonstrated occlusion at the variant vertebral artery with head turning. He was then taken for decompression of the vertebral artery through removal of the incomplete arch of C-1 that was causing the dynamic compression. After surgery the patient had a complete resolution of symptoms. In this report, the authors present the details of this case, describe the anatomical variants involved, and provide a discussion regarding the need for atlantoaxial fusion in these patients.


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