Mesenteric Venous Thrombosis and Small-Bowel Infarction Following Infusion of Vasopressin into the Superior Mesenteric Artery

Radiology ◽  
1972 ◽  
Vol 102 (2) ◽  
pp. 299-302 ◽  
Author(s):  
William A. Renert ◽  
Kenneth F. Button ◽  
Stuart L. Fuld ◽  
William J. Casarella
Keyword(s):  
Small Bowel ◽  
Venous Thrombosis ◽  
Superior Mesenteric Artery ◽  
Mesenteric Artery ◽  
Mesenteric Venous Thrombosis ◽  
Bowel Infarction ◽  
Small Bowel Infarction

CONGENITAL HEPATIC ARTERY ANEURYSM WITH SUPERIOR MESENTERIC ARTERY INSUFFICIENCY: A STEAL SYNDROME

PEDIATRICS ◽  
1967 ◽  
Vol 39 (3) ◽  
pp. 344-347
Author(s):  
Joyce D. Gryboski ◽  
Arthur Clemett
Keyword(s):  
Small Bowel ◽  
Superior Mesenteric Artery ◽  
Mesenteric Artery ◽  
Artery Aneurysm ◽  
Hepatic Artery Aneurysm ◽  
Protein Losing Enteropathy ◽  
Vascular Abnormality ◽  
Bowel Infarction ◽  
Small Bowel Infarction ◽  
Steal Syndrome

An unusual congenital vascular abnormality is described which caused chronic superior mesenteric artery insufficiency and eventual fatal small bowel infarction in an 18-week-old infant. During life the patient had post-prandial distress, intractible diarrhea, persistent melena, malabsorption, and a probable protein-losing enteropathy.

scholarly journals Small bowel infarction due to mesenteric venous thrombosis

2012 ◽  
Vol 95 (3) ◽  
pp. 130
Author(s):  
R M Blom ◽  
P Bracke ◽  
H Brusselaers ◽  
H Degryse
Keyword(s):  
Small Bowel ◽  
Venous Thrombosis ◽  
Mesenteric Venous Thrombosis ◽  
Bowel Infarction ◽  
Small Bowel Infarction

Primary Mural and Venous Thrombosis in the Heart*

1965 ◽  
Vol 14 (03/04) ◽  
pp. 600-604
Author(s):  
F. D Mann ◽  
D. K Buffmire
Keyword(s):  
Small Bowel ◽  
Venous Thrombosis ◽  
Myocardial Necrosis ◽  
Neoplastic Disease ◽  
Bowel Infarction ◽  
Vein Thrombosis ◽  
Small Bowel Infarction

SummaryTen cases are presented of ventricular mural thrombosis and thebesian vein thrombosis with an adequate coronary system and without myocardial necrosis or myocarditis. In six cases there was reason to suspect a thrombosing tendency : namely, extensive neoplastic disease in four cases, thrombophlebitis in one, and small bowel infarction in one.

Infusion of Recombinant Human Tissue Plasminogen Activator Through the Superior Mesenteric Artery in the Treatment of Acute Mesenteric Venous Thrombosis

2011 ◽  
Vol 25 (6) ◽  
pp. 840.e1-840.e4 ◽  
Author(s):  
Joaquim Mauricio da Motta Leal Filho ◽  
Aline Cristine Barbosa Santos ◽  
Francisco Cesar Carnevale ◽  
Wilson de Oliveira Sousa ◽  
Luiz Sérgio Pereira Grillo ◽  
...  
Keyword(s):  
Venous Thrombosis ◽  
Superior Mesenteric Artery ◽  
Plasminogen Activator ◽  
Tissue Plasminogen Activator ◽  
Human Tissue ◽  
Mesenteric Artery ◽  
Mesenteric Venous Thrombosis ◽  
Tissue Plasminogen

scholarly journals Superior Mesenteric venous thrombosis in a patient with protein C deficiency

2018 ◽  
Vol 6 (1) ◽  
pp. 50-53
Author(s):  
Tamzeed Hossain ◽  
Nazmun Nahar Munny ◽  
Chowdhury Rifat Niger ◽  
Arman Hossain ◽  
Rawshan Arra Khanam ◽  
...  
Keyword(s):  
Venous Thrombosis ◽  
Protein C ◽  
Antithrombin Iii ◽  
Protein S ◽  
Mesenteric Venous Thrombosis ◽  
Genetic Abnormality ◽  
Protein C Deficiency ◽  
Diffuse Abdominal Pain ◽  
Bowel Infarction ◽  
Small Bowel Infarction

Mesenteric venous thrombosis causing small-bowel infarction is an extremely rare cause of acute abdomen and often difficult to diagnose. Both congenital and acquired causes are responsible. Protein C deficiency is a rare genetic abnormality that predisposes the patient to thrombophilia and leads to thrombosis, often at unusual sites. It mimics clinically with many differentials.1 This paper presents a case of superior mesenteric venous thrombosis caused by protein C deficiency, which is a rare disease. A 68-year-old foreigner female presented with complaints of constant, diffuse abdominal pain of 7 days associated with nausea, vomiting, and anorexia. Even with all sorts of conservative management, pain was not subsiding. Contrasted computed tomography of the abdomen revealed SMV thrombosis. Immediate anticoagulant was started & hypercoagulability workup revealed protein C deficiency. It is concluded that the mesenteric venous thrombosis might be caused by underlying protein C deficiency, while protein S and antithrombin III levels were normal.Bangladesh Crit Care J March 2018; 6(1): 50-53

Mesenteric venous thrombosis treated with urokinase via the superior mesenteric artery

1996 ◽  
Vol 110 (5) ◽  
pp. 1633-1635 ◽  
Author(s):  
MR Poplausky ◽  
JA Kaufman ◽  
SC Geller ◽  
AC Waltman
Keyword(s):  
Venous Thrombosis ◽  
Superior Mesenteric Artery ◽  
Mesenteric Artery ◽  
Mesenteric Venous Thrombosis

scholarly journals Management of Spontaneous Isolated Mesenteric Artery Dissection: A Systematic Review

2021 ◽  
pp. 145749692110005
Author(s):  
S. Acosta ◽  
F. B. Gonçalves
Keyword(s):  
Risk Factors ◽  
Systematic Review ◽  
Superior Mesenteric Artery ◽  
Mesenteric Artery ◽  
Conservative Therapy ◽  
Case Series ◽  
Celiac Artery ◽  
Artery Dissection ◽  
Covered Stents ◽  
Bowel Infarction

Background and Aims: There are increasing reports on case series on spontaneous isolated mesenteric artery dissection, that is, dissections of the superior mesenteric artery and celiac artery, mainly due to improved diagnostic capacity of high-resolution computed tomography angiography performed around the clock. A few case–control studies are now available, while randomized controlled trials are awaited. Material and Methods: The present systematic review based on 97 original studies offers a comprehensive overview on risk factors, management, conservative therapy, morphological modeling of dissection, and prognosis. Results and Conclusions: Male gender, hypertension, and smoking are risk factors for isolated mesenteric artery dissection, while the frequency of diabetes mellitus is reported to be low. Large aortomesenteric angle has also been considered to be a factor for superior mesenteric artery dissection. The overwhelming majority of patients can be conservatively treated without the need of endovascular or open operations. Conservative therapy consists of blood pressure lowering therapy, analgesics, and initial bowel rest, whereas there is no support for antithrombotic agents. Complete remodeling of the dissection after conservative therapy was found in 43% at mid-term follow-up. One absolute indication for surgery and endovascular stenting of the superior mesenteric artery is development of peritonitis due to bowel infarction, which occurs in 2.1% of superior mesenteric artery dissections and none in celiac artery dissections. The most documented end-organ infarction in celiac artery dissections is splenic infarctions, which occurs in 11.2%, and is a condition that should be treated conservatively. The frequency of ruptured pseudoaneurysm in the superior mesenteric artery and celiac artery dissection is very rare, 0.4%, and none of these patients were in shock at presentation. Endovascular therapy with covered stents should be considered in these patients.

Sign in / Sign up

Export Citation Format

Share Document