FURTHER STUDIES ON THE PANCREATIC SECRETION IN THE SKATE

1932 ◽  
Vol 7 (1) ◽  
pp. 1-9 ◽  
Author(s):  
B. P. BABKIN
Keyword(s):  
Pancreatic Secretion ◽  
Secretory Activity ◽  
Nervous Systems ◽  
Acetyl Choline ◽  
Sympathetic Nervous Systems ◽  
Sympathetic Nervous ◽  
Pancreatic Gland

The parasympathetic drugs (acetyl choline and choline), and sympathetic drug (adrenaline), as well as histamine fail to increase the spontaneous pancreatic secretion in skates. A conclusion is drawn from these experiments that the parasympathetic and sympathetic nervous systems in skates play no part in the regulation of the secretory activity of the pancreatic gland.

[BP.11.05] URIC ACID AND HYPERTENSION. ROLE OF RENIN ANGIOTENSIN AND SYMPATHETIC NERVOUS SYSTEMS ACTIVITY

2017 ◽  
Vol 35 ◽  
pp. e347-e348
Author(s):  
C. Fania ◽  
F. Saladini ◽  
L. Mos ◽  
A. Bortolazzi ◽  
G. Zanata ◽  
...  
Keyword(s):  
Uric Acid ◽  
Nervous Systems ◽  
Renin Angiotensin ◽  
Sympathetic Nervous Systems ◽  
Sympathetic Nervous

scholarly journals Sympathetic nervous systems in chronic cor pulmonale.

1981 ◽  
Vol 45 (6) ◽  
pp. 646-653 ◽  
Author(s):  
EISUKE WATANABE ◽  
KOUICHI OGAWA ◽  
MASAAKI BAN ◽  
TATSUO SATAKE
Keyword(s):  
Cor Pulmonale ◽  
Nervous Systems ◽  
Sympathetic Nervous Systems ◽  
Sympathetic Nervous ◽  
Chronic Cor Pulmonale

scholarly journals The Relation between Fructose-Induced Metabolic Syndrome and Altered Renal Haemodynamic and Excretory Function in the Rat

2011 ◽  
Vol 2011 ◽  
pp. 1-17 ◽  
Author(s):  
Mohammed H. Abdulla ◽  
Munavvar A. Sattar ◽  
Edward J. Johns
Keyword(s):  
Metabolic Syndrome ◽  
Nervous Systems ◽  
Renin Angiotensin ◽  
Excretory Function ◽  
Sympathetic Nervous Systems ◽  
Neurohumoral Regulation ◽  
Dietary Fructose ◽  
Induced Hypertension ◽  
Vascular Sensitivity ◽  
Sympathetic Nervous

This paper explores the possible relationships between dietary fructose and altered neurohumoral regulation of renal haemodynamic and excretory function in this model of metabolic syndrome. Fructose consumption induces hyperinsulinemia, hypertriglyceridaemia, insulin resistance, and hypertension. The pathogenesis of fructose-induced hypertension is dubious and involves numerous pathways acting both singly and together. In addition, hyperinsulinemia and hypertension contribute significantly to progressive renal disease in fructose-fed rats. Moreover, increased activity of the renin-angiotensin and sympathetic nervous systems leading to downregulation of receptors may be responsible for the blunted vascular sensitivity to angiotensin II and catecholamines, respectively. Various approaches have been suggested to prevent the development of fructose-induced hypertension and/or metabolic alteration. In this paper, we address the role played by the renin-angiotensin and sympathetic nervous systems in the haemodynamic alterations that occur due to prolonged consumption of fructose.

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