scholarly journals International migration and adverse birth outcomes: role of ethnicity, region of origin and destination

2009 ◽  
Vol 64 (3) ◽  
pp. 243-251 ◽  
Author(s):  
M. L. Urquia ◽  
R. H. Glazier ◽  
B. Blondel ◽  
J. Zeitlin ◽  
M. Gissler ◽  
...  
Keyword(s):  
International Migration ◽  
Birth Outcomes ◽  
Adverse Birth Outcomes ◽  
Region Of Origin

scholarly journals Role of maternal occupational physical activity and psychosocial stressors on adverse birth outcomes

2016 ◽  
Vol 74 (3) ◽  
pp. 192-199 ◽  
Author(s):  
Laura J Lee ◽  
Elaine Symanski ◽  
Philip J Lupo ◽  
Sarah C Tinker ◽  
Hilda Razzaghi ◽  
...  
Keyword(s):  
Physical Activity ◽  
Birth Outcomes ◽  
Psychosocial Stressors ◽  
Occupational Physical Activity ◽  
Adverse Birth Outcomes

scholarly journals The Role of Congenital Cytomegalovirus Infection in Adverse Birth Outcomes: A Review of the Potential Mechanisms

Viruses ◽  
2020 ◽  
Vol 13 (1) ◽  
pp. 20
Author(s):  
Annete Njue ◽  
Carolyn Coyne ◽  
Andrea V. Margulis ◽  
Dai Wang ◽  
Morgan A. Marks ◽  
...  
Keyword(s):  
Birth Outcomes ◽  
Stem Cell Differentiation ◽  
Extravillous Trophoblast ◽  
Adverse Birth Outcomes ◽  
Placental Development ◽  
Low Passage ◽  
And Function

Human cytomegalovirus (CMV) is a major cause of nonhereditary adverse birth outcomes, including hearing and visual loss, neurologic deficits, and intrauterine growth retardation (IUGR), and may contribute to outcomes such as stillbirth and preterm delivery. However, the mechanisms by which CMV could cause adverse birth outcomes are not fully understood. This study reviewed proposed mechanisms underlying the role of CMV in stillbirth, preterm birth, and IUGR. Targeted literature searches were performed in PubMed and Embase to identify relevant articles. Several potential mechanisms were identified from in vitro studies in which laboratory-adapted and low-passage strains of CMV and various human placental models were used. Potential mechanisms identified included impairment of trophoblast progenitor stem cell differentiation and function, impairment of extravillous trophoblast invasiveness, dysregulation of Wnt signaling pathways in cytotrophoblasts, tumor necrosis factor-α mediated apoptosis of trophoblasts, CMV-induced cytokine changes in the placenta, inhibition of indoleamine 2,3-dioxygenase activity, and downregulation of trophoblast class I major histocompatibility complex molecules. Inherent challenges for the field remain in the identification of suitable in vivo animal models. Nonetheless, we believe that our review provides useful insights into the mechanisms by which CMV impairs placental development and function and how these changes could result in adverse birth outcomes.

scholarly journals Women's Reproductive Rights Policies and Adverse Birth Outcomes: A State-Level Analysis to Assess the Role of Race and Nativity Status

2020 ◽  
Vol 59 (6) ◽  
pp. 787-795
Author(s):  
May Sudhinaraset ◽  
Dovile Vilda ◽  
Jessica D. Gipson ◽  
Marta Bornstein ◽  
Maeve E. Wallace
Keyword(s):  
Birth Outcomes ◽  
Reproductive Rights ◽  
State Level ◽  
Adverse Birth Outcomes ◽  
Nativity Status ◽  
Level Analysis

1365: Admission for Nephrolithiasis in Pregnancy and Risk of Adverse Birth Outcomes

2007 ◽  
Vol 177 (4S) ◽  
pp. 450-450
Author(s):  
Mia A. Swartz ◽  
Mona T. Lydon-Rochelle ◽  
David Simon ◽  
Jonathan L. Wright ◽  
Michael P. Porter
Keyword(s):  
Birth Outcomes ◽  
Adverse Birth Outcomes ◽  
In Pregnancy
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