scholarly journals Growth hormone secretion in subjects with ovarian dysgenesis and Turner's syndrome.

1968 ◽  
Vol 43 (231) ◽  
pp. 595-597 ◽  
Author(s):  
S. R. Meadow ◽  
B. J. Boucher ◽  
K. Mashiter ◽  
M. N. King ◽  
L. Stimmler
Author(s):  
S. Bemasconi ◽  
L. Ghizzoni ◽  
C. Volta ◽  
M. Morano ◽  
G. Giovannelli

1989 ◽  
Vol 64 (4) ◽  
pp. 587-592 ◽  
Author(s):  
A A Massarano ◽  
C G Brook ◽  
P C Hindmarsh ◽  
P J Pringle ◽  
J D Teale ◽  
...  

1990 ◽  
Vol 71 (3) ◽  
pp. 770-772 ◽  
Author(s):  
ROBERTO LANES ◽  
SARA BRITO ◽  
MARIA SUNIAGA ◽  
GUSTAVO MONCADA ◽  
MARIETTA BORGES

2007 ◽  
Vol 88 (6) ◽  
pp. 610-613 ◽  
Author(s):  
P Pirazzoli ◽  
L Mazzanti ◽  
R Bergamaschi ◽  
A Perri ◽  
E Scarano ◽  
...  

1976 ◽  
Vol 81 (1) ◽  
pp. 9-18 ◽  
Author(s):  
Paul Saenger ◽  
Ernest Schwartz ◽  
Eckehart Wiedemann ◽  
Lenore S. Levine ◽  
Mary Tsai ◽  
...  

ABSTRACT Baseline somatomedin activity in seven of eight patients with Turner's syndrome was found to be within normal limits. Somatomedin activity readily suppressed with oestrogen administration. The overall mean serum somatomedin activity during oestrogen therapy (0.87 U/ml, sd 0.15) was significantly lower (P < 0.005) than the mean of the control serum somatomedin activities (1.09 U/ml, sd 0.24). During oestrogen therapy, suggestive elevations of fasting growth hormone levels were noted in five of eight patients. The data indicate that oestrogenic suppression of serum somatomedin was not due to decreased growth hormone secretion and suggest the existence of a negative feedback link between somatomedin and growth hormone.


Metabolism ◽  
1995 ◽  
Vol 44 (8) ◽  
pp. 1033-1037 ◽  
Author(s):  
Fabrizio Vaccaro ◽  
Stefano Cianfarani ◽  
Anna Maria Pasquino ◽  
Brunetto Boscherini

1989 ◽  
Vol 120 (3_Suppl) ◽  
pp. S77 ◽  
Author(s):  
A. GRÜTERS ◽  
K. SCHNABEL ◽  
D. L' ALLEMAND ◽  
B. WEBER ◽  
H. HELGE

Author(s):  
A. González-Angulo ◽  
S. Armendares-Sagrera ◽  
I. Ruíz de Chávez ◽  
H. Marquez-Monter ◽  
R. Aznar

It is a well documented fact that endometrial hyperplasia and adenocarcinoma may develop in women with Turner's syndrome who had received unopposed estrogen treatment (1), as well as in normal women under contraceptive medication with the sequential regime (2). The purpose of the present study was to characterize the possible changes in surface and glandular epithelium in these women who were treated with a sequential regime for a period of between three and eight years. The aim was to find organelle modifications which may lead to the understanding of the biology of an endometrium under exogenous hormone stimulation. Light microscopy examination of endometrial biopsies of nine patients disclosed a proliferative pattern; in two of these, there was focal hyperplasia. With the scanning electron microscope the surface epithelium in all biopsies showed secretory cells with microvilli alternating with non secretory ciliated cells. Regardless of the day of the cycle all biopsies disclosed a large number of secretory cells rich in microvilli (fig.l) with long and slender projections some of which were branching (fig. 2).


Sign in / Sign up

Export Citation Format

Share Document