scholarly journals Human Cell Tropism and Innate Immune System Interactions of Human Respiratory Coronavirus EMC Compared to Those of Severe Acute Respiratory Syndrome Coronavirus

2013 ◽  
Vol 87 (9) ◽  
pp. 5300-5304 ◽  
Author(s):  
F. Zielecki ◽  
M. Weber ◽  
M. Eickmann ◽  
L. Spiegelberg ◽  
A. M. Zaki ◽  
...  
Keyword(s):  
Immune System ◽  
Severe Acute Respiratory Syndrome ◽  
Human Cell ◽  
Innate Immune System ◽  
Innate Immune ◽  
Cell Tropism ◽  
Respiratory Coronavirus

scholarly journals TRINITY OF SEVERE ACUTE RESPIRATORY SYNDROME CORONAVIRUS-2: ORIGIN, GENOTYPE, PHENOTYPE, AND IMMUNE RESPONSE IN CORONAVIRUS DISEASE-2019

2021 ◽  
pp. 28-36
Author(s):  
MURUGAN NANDAGOPAL ◽  
ARULMOZHI BALAKRISHNAN ◽  
CHIRAYU PADHIAR
Keyword(s):  
Innate Immunity ◽  
Immune System ◽  
Severe Acute Respiratory Syndrome ◽  
Innate Immune System ◽  
Clinical Symptoms ◽  
Innate Immune ◽  
Host Immune System ◽  
Wide Range ◽  
Novel Coronavirus ◽  
Classification Structure

The coronavirus disease-2019 (COVID-19) outbreak by severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) or a novel coronavirus (2019-CoV) has prompted global health concerns. A pandemic resulted from the disease’s transmission through many routes. In this pandemic, the interaction between coronavirus and the host immune system, particularly the innate immune system, is becoming more prominent. Against viruses and pathogens, innate immunity serves as a first line of defense. Our understanding of pathogenesis will benefit from a better grasp of the mechanisms of immune evasion techniques. The origin, classification, structure, and method of transmission of SARS-CoV-2 were summarized in this paper. We have discussed the importance of important communications. In this review, we have discussed the function of important components of the innate immune system in COVID-19 infection, as well as how the virus evades innate immunity through multiple tactics and contributes to a wide range of clinical symptoms and outcomes.

Panic makes you more susceptible to covid-19 (Preprint)

2020 ◽  
Author(s):  
Md Musharraf Hossain ◽  
Colin R Dunstan
Keyword(s):  
Immune System ◽  
Severe Acute Respiratory Syndrome ◽  
Innate Immune System ◽  
Severe Disease ◽  
Adaptive Immune System ◽  
Innate Immune ◽  
Cytokine Storm ◽  
Adaptive Immune ◽  
Storm Response ◽  
Short Period

UNSTRUCTURED COVID-19 is the official name describing the coronavirus disease causing by the virus SARS-CoV-2 (severe acute respiratory syndrome coronavirus 2). Its higher infection rate has led to worldwide spread making it a more dangerous threat than other coronavirus related diseases. As this virus spreads and responses from governments become more constraining of normal activities, we are seeing levels of anxiety among people increase over a relatively short period of time. When we are stressed the immune system’s ability to defend us from infections is reduced. Stress can suppress the innate immune system that provides the initial defence against viral infection making us more susceptible to Covid-19. In contrast, stress can cause overactivity of the adaptive immune system making a cytokine storm response leading to severe disease and death more likely. Management of stress levels has become an important strategy to reduce the risks of COVID-19.

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