scholarly journals Amplitude modulation reduces loudness adaptation to high-frequency tones

2015 ◽  
Vol 138 (1) ◽  
pp. 279-283 ◽  
Author(s):  
Dwight P. Wynne ◽  
Sahara E. George ◽  
Fan-Gang Zeng
Author(s):  
S. A. Nayfeh ◽  
A. H. Nayfeh

Abstract We study the response of a single-degree-of-freedom system with cubic nonlinearities to an amplitude-modulated excitation whose carrier frequency is much higher than the natural frequency of the system. The only restriction on the amplitude modulation is that it contain frequencies much lower than the carrier frequency of the excitation. We apply the theory to different types of amplitude modulation and find that resonant excitation of the system may occur under some conditions.


1993 ◽  
Vol 93 (4) ◽  
pp. 2368-2368
Author(s):  
Andrzej Miskiewicz ◽  
Rhona P. Hellman ◽  
Carol H. Meiselman ◽  
Bertram Scharf

2010 ◽  
Vol 645-648 ◽  
pp. 953-956 ◽  
Author(s):  
Daniel Brennan ◽  
Bing Miao ◽  
Konstantin Vassilevski ◽  
Nicolas G. Wright ◽  
Alton B. Horsfall

This work presents the amplitude modulation radio transmission system for communications in hostile environments. The commissioning of a high temperature oscillator and AM mixer system for the purpose of Amplitude Shift Keyed modulation is presented. While previous work has demonstrated oscillators in the Ultra High Frequency (UHF) band, these have been targeted at applications such as radar and mobile telephones. In this paper we have concentrated on the shortwave bands to maximize the range between a sensor unit and the receiver within wireless networks. The work demonstrates that simple communication systems are already possible for hostile environments and allow for simple sensor data to be wirelessly transmitted to safer working areas.


2019 ◽  
Vol 28 (11) ◽  
pp. 1950179
Author(s):  
Montree Kumngern ◽  
Thanat Nonthaputha ◽  
Fabian Khateb

This paper presents a new amplitude modulation (AM), frequency modulation (FM), amplitude shift keying (ASK), frequency shift keying (FSK), phase shift keying (PSK) and quadrature amplitude modulation (QAM) waveform generators using current current-controlled conveyor transconductance amplifier (CCCTA) and current conveyor analog switches (CCASs). The CCCTA around with capacitors and resistors are used to generate high-frequency carrier which is worked as a quadrature oscillator. The oscillating frequency of oscillator can be controlled using the bias current of CCCTA, therefore FM and FSK waveforms can be obtained by applying information signal through the bias current of CCCTA. Unlike previous waveform generators, proposed circuit uses second generation current conveyor (CCII) to work as CCAS and AM, ASK, PSK and QAM waveforms can be obtained by putting information signals to control switches that realized using CCIIs. The proposed circuit has been simulated using 0.18[Formula: see text][Formula: see text]m CMOS [Formula: see text]-well process from TSMC. The simulation results are used to confirm workability of the proposed circuit.


1998 ◽  
Vol 79 (1) ◽  
pp. 181-189 ◽  
Author(s):  
Kavita R. Peshori ◽  
William F. Collins ◽  
Lorne M. Mendell

Peshori, Kavita R., William F. Collins III, and Lorne M. Mendell. EPSP amplitude modulation at the rat Ia-alpha motoneuron synapse: effects of GABAB receptor agonists and antagonists. J. Neurophysiol. 79: 181–189, 1998. The object of this study was to examine the relationship between excitatory postsynaptic potential (EPSP) amplitude, posttetanic potentiation, and EPSP amplitude modulation at synapses made by group Ia afferents on motoneurons in the rat. These relationships were evaluated in cells in untreated rats and in cells in rats treated with the γ-aminobutyric acid-B (GABAB) receptor agonist baclofen and antagonist CGP-35348, which were used to manipulate Ca2+ entry into presynaptic terminals and consequently probability of transmitter release from them. There was no evidence for postsynaptic action of these drugs from measurement of their effects on motoneuron properties. During high-frequency stimulation (32 shock bursts at 167 Hz), EPSP amplitude either decreased (negative modulation) or increased (positive modulation) in response to successive stimuli at different connections. In untreated rats this frequency-dependent amplitude modulation behavior was inversely but weakly correlated with EPSP amplitude measured at low frequency. Intravenous (iv) administration of the GABAB agonist, baclofen, produced a marked and progressive decrease in EPSP amplitude measured at low frequency coincident with a change in frequency-dependent EPSP amplitude modulation toward more positive values (synaptic facilitation). In contrast, an increase in EPSP amplitude occurred after iv administration of the GABAB antagonist CGP-35348 that was accompanied by a negative shift in EPSP amplitude modulation during high-frequency stimulation. The negative shift in EPSP amplitude modulation (synaptic depression) after CGP-35348 application was much smaller than the positive shift induced by baclofen when normalized to the change in EPSP amplitude. Posttetanic potentiation decreased after baclofen but did not increase after CGP-35348. The relationship between modulation and EPSP amplitude was much steeper after GABAB receptor manipulation in either direction than that observed in the population of motoneurons in untreated preparations. This suggests that in the rat differences in probability of release play at most a small role in determining EPSP amplitude across the motoneuron pool.


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