Comparison of ray and wave approaches to acoustic impulse propagation prior to a shadow boundary

1990 ◽  
Vol 87 (3) ◽  
pp. 1017-1025 ◽  
Author(s):  
C. G. Don ◽  
A. J. Cramond
Keyword(s):  
Shadow Boundary ◽  
Impulse Propagation

scholarly journals Detection of a Local Slow Potential Preceding the Surface QRS Complex During Non-Preexcited Impulse Propagation

1998 ◽  
Vol 62 (10) ◽  
pp. 760-764 ◽  
Author(s):  
Yoshinori Kobayashi ◽  
Yasushi Miyauchi ◽  
Naomi Kawaguchi ◽  
Kazuko Ohmura ◽  
Hirokazu Saitoh ◽  
...  
Keyword(s):  
Qrs Complex ◽  
Slow Potential ◽  
Impulse Propagation

Deficiency of electroneutral K+ -Cl− cotransporter 3 causes a disruption in impulse propagation along peripheral nerves

Glia ◽  
2010 ◽  
Vol 58 (13) ◽  
pp. 1544-1552 ◽  
Author(s):  
Yuan-Ting Sun ◽  
Thy-Sheng Lin ◽  
Shun-Fen Tzeng ◽  
Eric Delpire ◽  
Meng-Ru Shen
Keyword(s):  
Peripheral Nerves ◽  
Impulse Propagation

scholarly journals Progressive axonopathy when oligodendrocytes lack the myelin protein CMTM5

2021 ◽  
Author(s):  
Tobias J Buscham ◽  
Maria A. Eichel-Vogel ◽  
Anna M Steyer ◽  
Olaf Jahn ◽  
Nicola Strenzke ◽  
...  
Keyword(s):  
Early Onset ◽  
Wallerian Degeneration ◽  
Transmembrane Domain ◽  
Transmembrane Protein ◽  
Myelin Proteins ◽  
Myelin Protein ◽  
Impulse Propagation ◽  
Functional Relevance

Oligodendrocytes facilitate rapid impulse propagation along the axons they myelinate and support their long-term integrity. However, the functional relevance of many myelin proteins has remained unknown. Here we find that expression of the tetraspan-transmembrane protein CMTM5 (Chemokine-like factor-like MARVEL-transmembrane domain containing protein 5) is highly enriched in oligodendrocytes and CNS myelin. Genetic disruption of the Cmtm5-gene in oligodendrocytes of mice does not impair the development or ultrastructure of CNS myelin. However, oligodendroglial Cmtm5-deficiency causes an early-onset progressive axonopathy, which we also observe in global and in tamoxifen-induced oligodendroglial Cmtm5-mutants. Presence of the Wlds mutation ameliorates the axonopathy, implying a Wallerian degeneration-like pathomechanism. These results indicate that CMTM5 is involved in the function of oligodendrocytes to maintain axonal integrity rather than myelin biogenesis.

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