Middle ear pressure: Effects on the auditory periphery

1976 ◽  
Vol 59 (1) ◽  
pp. 135-142 ◽  
Author(s):  
D. L. McPherson ◽  
J. M. Miller ◽  
A. Axelsson
Author(s):  
Venkatesh Aithal ◽  
Sreedevi Aithal ◽  
Joseph Kei ◽  
Shane Anderson ◽  
David Wright

Background: Wideband absorbance (WBA) measured at ambient pressure (WBAA) does not directly accountfor middle ear pressure effects. On the other hand, WBA measured at tympanometric peak pressure(TPP) (WBATPP) may compensate for the middle ear pressure effects. To date, there are no studies thathave compared WBAA and WBATPP in ears with surgically confirmed otitis media with effusion (OME).<br />Purpose: The purpose of this study was to compare the predictive accuracy of WBAA and WBATPP inears with OME.<br />Research Design: Prospective cross-sectional study.<br />Study Sample: A total of 60 ears from 38 healthy children (mean age = 6.5 years, SD = 1.84 years) and60 ears from 38 children (mean age = 5.5 years, SD = 3.3 years) with confirmed OME during myringotomywere included in this study.<br />Data Collection and Analysis: Results were analyzed using descriptive statistics and analysis of variance.The predictive accuracy of WBAA and WBATPP was determined using receiver operating characteristics(ROC) analyses.<br />Results: Both WBAA and WBATPP were reduced in ears with OME compared with that in healthy ears.The area under the ROC (AROC) curve was 0.92 for WBAA at 1.5 kHz, whereas that for WBATPP at 1.25kHz was 0.91. In comparison, the AROC for 226-Hz tympanometry based on the static acoustic admittance(Ytm) measure was 0.93.<br />Conclusions: Both WBAA and WBATPP showed high and similar test performance, but neither test performedsignificantly better than 226-Hz tympanometry for detection of surgically confirmed OME.<br />


2019 ◽  
pp. 55-61
Author(s):  
Owen J. O’Neill ◽  
Elizabeth Smykowski ◽  
Jo Ann Marker ◽  
Lubiha Perez ◽  
drah Gurash ◽  
...  

Introduction: Eustachian tube dysfunction (ETD) and middle ear barotrauma (MEB) are the most common adverse effects of hyperbaric oxygen (HBO2) treatments. Patients practice equalization maneuvers to prevent ETD and MEB prior to hyperbaric exposure. Some patients are still unable to equalize middle ear pressure. This ETD results in undesirable consequences, including barotrauma, treatment with medications or surgical myringotomy with tube placement and interruption of HBO2. When additional medications and myringotomy are employed, they are associated with additional complications. Methods: A device known as the Ear Popper® has been reported to reduce complications from serous otitis media and reduce the need for surgical interventions (myringotomy). Patients unable to equalize middle ear pressure during initial compression in the hyperbaric chamber were allowed to use the device for rescue. All hyperbaric treatments were compressed using a United States Navy TT9, or a 45-fsw hyperbaric treatment schedule. Patients with persistent ETD and the inability to equalize middle ear pressure were given the Ear Popper upon consideration of terminating their treatment. Results: The Ear Popper allowed all patients to successfully equalize middle ear pressure and complete their treatments. Conclusion: This study substantiates the use of this device to assist in allowing pressurization of the middle ear space in patients otherwise unable to achieve equalization of middle ear pressure during HBO2 treatment in a multiplace chamber.


2013 ◽  
Vol 80 (6) ◽  
pp. 726-727 ◽  
Author(s):  
V. Rinaldi ◽  
M. Cappadona ◽  
M. Gaffuri ◽  
S. Torretta ◽  
L. Pignataro

1979 ◽  
Vol 88 (3) ◽  
pp. 368-376 ◽  
Author(s):  
A. Axelsson ◽  
J. Miller ◽  
M. Silverman

Acute middle ear (ME) and inner ear changes following brief unilateral phasic ME pressure changes (up to ± 6000/mm H2O) were studied in the guinea pig. Middle ear findings included perforation of the tympanic membrane, serous and serosanguinous exudate and hemorrhage of tympanic membrane and periosteal vessels. Changes were related to magnitude of applied pressure. Perforation and hemorrhage were more commonly seen with negative rather than positive pressure. Air bubbles behind the round window were seen with positive pressures. Occasional distortion, but never perforation of the round window, was noted. Hemorrhage of the scala tympani was observed with both positive and negative pressures; scala vestibuli hemorrhage was found with negative ME pressure. In some instances pressure direction and magnitude related changes were seen in the contralateral ear.


1997 ◽  
Vol 106 (6) ◽  
pp. 478-482 ◽  
Author(s):  
Wolfgang Maier ◽  
Milo Fradis ◽  
Uwe Ross ◽  
Bernhard Richter

Relationships between middle ear pressure and non-infection-related cochleovestibular dysfunction have been suggested by several authors. According to some data, vertiginous attacks can be prevented by the insertion of a ventilation tube in patients suffering from Meniere's syndrome. The aim of our study was to investigate if the incidence of eustachian tube malfunction and pathologic middle ear pressure is frequent, and if routine implantation of ventilation tubes is reasonable in ears with dysfunctions of the labyrinth, including clinical Meniere's syndrome. So, we determined in our pressure chamber all active and passive parameters of eustachian tube function in 40 patients suffering from Meniere's syndrome, sudden sensory hearing impairment (SSHI), or vestibular neuronitis. Our results disclosed no nonrandom incidence of impaired tubal function among our patients compared to healthy control subjects. Pressure equalization was sufficient in most patients suffering from clinical Meniere's syndrome, and only one patient with vestibular neuronitis presented with a patulous tube. Our results show that impairment of vestibular or cochlear function is not regularly accompanied by eustachian tube dysfunction. Furthermore, no patient reported symptoms while pressure variation was performed. We conclude that variation of middle ear pressure does not usually play a role in the genesis of Meniere's syndrome, vestibular neuronitis, or SSHI. Thus, from our data, we cannot recommend routine implantation of tympanic ventilation tubes in patients suffering from Meniere's syndrome, vestibular neuronitis, or sudden hearing loss.


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