GAFChromic film dosimetry with a flatbed color scanner for Leksell Gamma Knife therapy

2004 ◽  
Vol 31 (5) ◽  
pp. 1243-1248 ◽  
Author(s):  
M. Yamauchi ◽  
T. Tominaga ◽  
O. Nakamura ◽  
R. Ueda ◽  
M. Hoshi
2016 ◽  
Vol 07 (S 01) ◽  
pp. S088-S090 ◽  
Author(s):  
Guru Dutta Satyarthee ◽  
M. D. Sudhan ◽  
V. S. Mehta

ABSTRACTBrainstem glioma usually carries a poor prognosis and prolonged survival is very infrequent. In a detailed Pubmed, Medline search for prolonged survival, authors could got a longest survival only up to seventeen years, reported by Umehara et al, who was subjected to gamma knife therapy and got symptomatic, MRI brain reveled large tumor growth during pregnancy necessitating emergency surgery and histopathological diagnosis was pilocytic astrocytoma. Authors report an interesting case of midbrain glioma diagnosed 21 years back, who underwent gross resection in the year 1993, histopathology was pilocytic astrocytoma, WHO grade I, and received gamma knife surgery for residual subsequently and he presented with sudden onset left sided hemiplegia on the current admission. The cranial MRI imaging revealed an infarct involving right hemi midbrain, contrast MRI brain revealed no residual glioma. To the best knowledge of authors such prolonged survival is not reported with a case of brainstem glioma survived twenty- one years with non residual tumor on the last imaging study represents first case of its kind in the western literature and probably developed hemiplegia due to bleed, highlighting bleed as delayed complication following gamma knife therapy for cranial tumors


2019 ◽  
Vol 121 ◽  
pp. 69-76 ◽  
Author(s):  
Somayeh Gholami ◽  
Hassan Ali Nedaie ◽  
Ali S. Meigooni

2018 ◽  
Vol 45 (5) ◽  
pp. 2329-2336 ◽  
Author(s):  
Neda Gholizadeh Sendani ◽  
Alireza Karimian ◽  
Clara Ferreira ◽  
Parham Alaei

Author(s):  
Takahiko Taniguchi ◽  
Terushige Toyooka ◽  
Masataka Miyama ◽  
Satoru Takeuchi ◽  
Naoki Otani ◽  
...  

2011 ◽  
Vol 196 (1) ◽  
pp. 15-22 ◽  
Author(s):  
Jeffrey M. Pollock ◽  
Christopher T. Whitlow ◽  
Justin Simonds ◽  
E. Andrew Stevens ◽  
Robert A. Kraft ◽  
...  

2015 ◽  
Vol 16 (6) ◽  
pp. 325-332 ◽  
Author(s):  
Meral L. Reyhan ◽  
Ting Chen ◽  
Miao Zhang

Neurosurgery ◽  
1990 ◽  
Vol 26 (5) ◽  
pp. 725-735 ◽  
Author(s):  
Christopher S. Ogilvy

Abstract There have been numerous case reports and series of patients treated with partial brain irradiation, linear accelerator-based radiosurgery, gamma knife radiosurgery, and Bragg peak therapy for inoperable arteriovenous malformations (AVMs). These cases are summarized and compared. There is convincing evidence that radiation therapy does have a role in obliterating carefully chosen inoperable lesions. The changes that occur in vessel walls after radiation are reviewed. Data about x-ray and gamma radiation are mostly historical and difficult to evaluate because of the techniques of partial brain irradiation. There is a lack of data about the volume of AVM treated and the minimum dose delivered to the AVM nidus. For gamma knife, heavy particle, and linear accelerator therapy, more complete data are available. The incidence of hemorrhage during the first 2 years after treatment, when radiation-induced vascular changes are proposed to occur, is approximately 2.6% per year for gamma knife therapy, 2% per year for proton beam therapy, 2.3% per year for helium beam therapy, and 2.3% per year for linear accelerator therapy. These rates are similar to the recurrence rate for hemorrhage of 2.2 to 3% per year expected based on the natural history of untreated AVMs. If AVM obliteration after therapy is not achieved, the incidence of recurrent hemorrhage remains between 2% per year after treatment with gamma knife therapy. The incidence of hemorrhage for all patients treated was reported as 0.15% per year in one study and 20% over 8 years in a follow-up study using proton beam therapy. Mortality from hemorrhage after treatment was 0.6% after gamma knife therapy, 2.3% after helium beam therapy, and 2 to 5% after proton beam therapy. These figures for mortality are all lower than the 11% observed for the natural history of untreated AVMs. Permanent neurological deficits experienced as a complication of radiation occurred in 2 to 3% of patients treated with gamma knife therapy, 4% of patients treated with helium beam therapy, 1.7% of patients treated with proton beam therapy, and 3% of patients treated with stereotactic linear accelerator therapy. Proton beam therapy has been used for both small and large lesions. The majority of lesions in patients treated with gamma knife, helium beam, and linear accelerator therapy have been small (usually less than 3.0 cm average diameter) lesions. In these patients with small inoperable lesions treated with accurately directed fields of isocentric radiation, the greatest incidence of AVM obliteration has been observed on follow-up angiograms. Larger lesions may undergo vascular wall thickening with subsequent protection from recurrent hemorrhage, but more data are needed to support this hypothesis.


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