Improved laser-assisted vascular tissue fusion using solder-doped polymer membranes on a canine model

2000 ◽  
Author(s):  
Karen M. McNally-Heintzelman ◽  
Brian S. Sorg ◽  
Daniel X. Hammer ◽  
Douglas L. Heintzelman ◽  
Diane E. Hodges ◽  
...  
Keyword(s):  
Vascular Tissue ◽  
Polymer Membranes ◽  
Canine Model ◽  
Tissue Fusion ◽  
Doped Polymer

Improved vascular tissue fusion using new light-activated surgical adhesive on a canine model

2001 ◽  
Vol 6 (1) ◽  
pp. 68 ◽  
Author(s):  
Karen M. McNally ◽  
Brian S. Sorg ◽  
Daniel X. Hammer ◽  
Douglas L. Heintzelman ◽  
Diane E. Hodges ◽  
...  
Keyword(s):  
Vascular Tissue ◽  
Canine Model ◽  
Tissue Fusion ◽  
Surgical Adhesive

Bond Strength of Thermally Fused Vascular Tissue Varies With Apposition Force

2015 ◽  
Vol 137 (12) ◽  
Author(s):  
Nicholas S. Anderson ◽  
Eric A. Kramer ◽  
James D. Cezo ◽  
Virginia L. Ferguson ◽  
Mark E. Rentschler
Keyword(s):  
Bond Strength ◽  
Blood Vessels ◽  
Thermal Energy ◽  
Molecular Mechanisms ◽  
Vascular Tissue ◽  
Tissue Temperature ◽  
Tissue Fusion ◽  
Tunica Media ◽  
Adhesive Mechanics ◽  
Bond Strengths

Surgical tissue fusion devices ligate blood vessels using thermal energy and coaptation pressure, while the molecular mechanisms underlying tissue fusion remain unclear. This study characterizes the influence of apposition force during fusion on bond strength, tissue temperature, and seal morphology. Porcine splenic arteries were thermally fused at varying apposition forces (10–500 N). Maximum bond strengths were attained at 40 N of apposition force. Bonds formed between 10 and 50 N contained laminated medial layers; those formed above 50 N contained only adventitia. These findings suggest that commercial fusion devices operate at greater than optimal apposition forces, and that constituents of the tunica media may alter the adhesive mechanics of the fusion mechanism.

Biochemical Mechanisms of Laser Vascular Tissue Fusion

1991 ◽  
Vol 4 (1) ◽  
pp. 3-12 ◽  
Author(s):  
Carol R. Guthrie ◽  
Louann W. Murray ◽  
George E. Kopchok ◽  
David Rosenbaum ◽  
Rodney A. White
Keyword(s):  
Vascular Tissue ◽  
Tissue Fusion ◽  
Biochemical Mechanisms

Acellular scaffolding for vascular tissue-engineering in a canine model

2003 ◽  
Vol 114 (2) ◽  
pp. 297
Author(s):  
P.J. Schaner ◽  
F.E. Rosato ◽  
N.A. Tarola ◽  
J. Eisenberg ◽  
N. Martin ◽  
...  
Keyword(s):  
Tissue Engineering ◽  
Vascular Tissue ◽  
Canine Model ◽  
Vascular Tissue Engineering

Photophysics of laser dye-doped polymer membranes for laser-induced fluorescence photogrammetry

2004 ◽  
Vol 43 (36) ◽  
pp. 6629 ◽  
Author(s):  
Adrian A. Dorrington ◽  
Thomas W. Jones ◽  
Paul M. Danehy
Keyword(s):  
Polymer Membranes ◽  
Laser Induced Fluorescence ◽  
Laser Dye ◽  
Doped Polymer

Thermal Studies of In-Vivo Vascular Tissue Fusion by Argon Laser

1988 ◽  
Vol 1 (1) ◽  
pp. 5-12 ◽  
Author(s):  
George Kopchok ◽  
Rodney A. White ◽  
Warren S. Grundfest ◽  
Roy M. Fujitani ◽  
Frank Litvack ◽  
...  
Keyword(s):  
Vascular Tissue ◽  
Thermal Studies ◽  
Argon Laser ◽  
Tissue Fusion

scholarly journals Manipulation of cellular spheroid composition and the effects on vascular tissue fusion

2015 ◽  
Vol 13 ◽  
pp. 188-198 ◽  
Author(s):  
T.R. Olsen ◽  
B. Mattix ◽  
M. Casco ◽  
A. Herbst ◽  
C. Williams ◽  
...  
Keyword(s):  
Vascular Tissue ◽  
Tissue Fusion ◽  
Cellular Spheroid

Elemental Analysis of Phloem Tissue in Lemna Minor Root Tips

1980 ◽  
Vol 38 ◽  
pp. 798-799
Author(s):  
Patrick Echlin ◽  
Thomas Hayes ◽  
Clifford Lai ◽  
Greg Hook
Keyword(s):  
Vascular Tissue ◽  
Lemna Minor ◽  
Atomic Weight ◽  
Companion Cell ◽  
Root Tips ◽  
Phloem Tissue ◽  
Cell Stage ◽  
Phloem Parenchyma ◽  
Parenchyma Cells ◽  
Xylem Element

Studies (1—4) have shown that it is possible to distinguish different stages of phloem tissue differentiation in the developing roots of Lemna minor by examination in the transmission, scanning, and optical microscopes. A disorganized meristem, immediately behind the root-cap, gives rise to the vascular tissue, which consists of single central xylem element surrounded by a ring of phloem parenchyma cells. This ring of cells is first seen at the 4-5 cell stage, but increases to as many as 11 cells by repeated radial anticlinal divisions. At some point, usually at or shortly after the 8 cell stage, two phloem parenchyma cells located opposite each other on the ring of cells, undergo an unsynchronized, periclinal division to give rise to the sieve element and companion cell. Because of the limited number of cells involved, this developmental sequence offers a relatively simple system in which some of the factors underlying cell division and differentiation may be investigated, including the distribution of diffusible low atomic weight elements within individual cells of the phloem tissue.

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

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