Quantifying peristaltic activity of a small intestine based on tracking of digests in an abdominal B-mode movie

Author(s):  
Yuichiro Iida ◽  
Osamu Sakata ◽  
Yutaka Suzuki
2018 ◽  
pp. 70-74
Author(s):  
O. A. Yarmolenko ◽  
E. G. Malayeva ◽  
I. A. Khudyakov ◽  
M. N. Menshakova ◽  
L. A. Kobruseva

Diabetic enteropathy is a rarely diagnosed complication of diabetes mellitus. According to literature sources, most researchers note an association of this complication with the manifestation of diabetic autonomic neuropathy. The main clinical manifestations of diabetic enteropathy are: disrupted peristaltic activity of the small intestine, excessive bacterial growth, diarrhea, and steatorrhea. The article describes a clinical case of diabetic enteropathy in a young patient, which developed 8 years after diabetes mellitus type 1 had been diagnosed.


1957 ◽  
Vol 189 (1) ◽  
pp. 108-112 ◽  
Author(s):  
David H. P. Streeten ◽  
Basil I. Hirschowitz ◽  
Keith S. Henley ◽  
H. M. Pollard

Adrenal cortex extract in low concentrations (1:1400–1:250) increases the peristaltic contractions of small intestine and restores to fatigued intestinal segments normal peristaltic activity and the ability to propel fluid against a pressure gradient, in vitro (modified Trendelenburg technique). Adrenal cortex extract in high concentrations (1:150–1:25) reversibly inhibits or abolishes peristalsis in vitro. In adrenalectomized rats in vivo, adrenal cortex extract increases the rate of propulsion of dyes along the small intestine in moderate doses (1 ml b.i.d.), and decreases propulsion in large doses (5 ml b.i.d.). Cortisone, hydrocortisone and corticosterone in the amounts present in stimulant doses of adrenal cortex extract had no effect on intestinal propulsion, in vivo. Doses of aldosterone (0.1 and 0.5 µg b.i.d.) comparable with the amounts contained in the extract used and large doses of the electrolyte-controlling steroids, desoxycorticosterone (2.5 and 5 mg) and corticosterone (2.5 mg), reproduced the stimulant effects of the extract, in vivo. It is possible that the effects of aldosterone may be of significance in controlling intestinal motility under physiological and some pathological conditions.


2005 ◽  
Vol 17 (2) ◽  
pp. 262-272 ◽  
Author(s):  
G. Bogeski ◽  
A. D. Shafton ◽  
P. D. Kitchener ◽  
D. M. Ferens ◽  
J. B. Furness

Author(s):  
A. J. Tousimis

The elemental composition of amino acids is similar to that of the major structural components of the epithelial cells of the small intestine and other tissues. Therefore, their subcellular localization and concentration measurements are not possible by x-ray microanalysis. Radioactive isotope labeling: I131-tyrosine, Se75-methionine and S35-methionine have been successfully employed in numerous absorption and transport studies. The latter two have been utilized both in vitro and vivo, with similar results in the hamster and human small intestine. Non-radioactive Selenomethionine, since its absorption/transport behavior is assumed to be the same as that of Se75- methionine and S75-methionine could serve as a compound tracer for this amino acid.


Author(s):  
D.S. Friend ◽  
N. Ghildyal ◽  
M.F. Gurish ◽  
K.F. Austen ◽  
R.L. Stevens

Trichinella spiralis induces a profound mastocytosis and eosinophilia in the small intestine of the infected mouse. Mouse mast cells (MC) store in their granules various combinations of at least five chymotryptic chymases [designated mouse MC protease (mMCP) 1 to 5], two tryptic proteases designated mMCP-6 and mMCP-7 and an exopeptidase, carboxypeptidase A (mMC-CPA). Using antipeptide, protease -specific antibodies to these MC granule proteases, immunohistochemistry was done to determine the distribution, number and protease phenotype of the MCs in the small intestine and spleen 10 to >60 days after Trichinella infection of BALB/c and C3H mice. TEM was performed to evaluate the granule morphology of the MCs between intestinal epithelial cells and in the lamina propria (mucosal MCs) and in the submucosa, muscle and serosa of the intestine (submucosal MCs).As noted in the table below, the number of submucosal MCs remained constant throughout the study. In contrast, on day 14, the number of MCs in the mucosa increased ~25 fold. Increased numbers of MCs were observed between epithelial cells in the mucosal crypts, in the lamina propria and to a lesser extent, between epithelial cells of the intestinal villi.


2001 ◽  
Vol 120 (5) ◽  
pp. A683-A683
Author(s):  
J GUZMAN ◽  
S SHARP ◽  
J YU ◽  
F MCMORRIS ◽  
A WIEMELT ◽  
...  

2001 ◽  
Vol 120 (5) ◽  
pp. A183-A183
Author(s):  
H KOBAYASHI ◽  
H NAGATA ◽  
S MIURA ◽  
T AZUMA ◽  
H SUZUKI ◽  
...  

2001 ◽  
Vol 120 (5) ◽  
pp. A195-A195
Author(s):  
J PAULA ◽  
E SPINEDI ◽  
A DUBIN ◽  
D BUSTOS ◽  
J DAVOLOS

2001 ◽  
Vol 120 (5) ◽  
pp. A114-A114
Author(s):  
C GAO ◽  
H HU ◽  
S LIU ◽  
N GAO ◽  
Y XIA ◽  
...  

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