scholarly journals Network interactions within the canine intrinsic cardiac nervous system: implications for reflex control of regional cardiac function

2013 ◽  
Vol 591 (18) ◽  
pp. 4515-4533 ◽  
Author(s):  
Eric Beaumont ◽  
Siamak Salavatian ◽  
E. Marie Southerland ◽  
Alain Vinet ◽  
Vincent Jacquemet ◽  
...  
Keyword(s):  
Nervous System ◽  
Cardiac Function ◽  
Regional Cardiac Function ◽  
Reflex Control

scholarly journals Network interactions within the intrinsic cardiac nervous system: Implications for reflex control of regional cardiac function

2013 ◽  
Vol 27 (S1) ◽  
Author(s):  
Eric Beaumont ◽  
John Andrew Armour ◽  
Siamak Salavatian ◽  
Alain Vinet ◽  
Vincent Jacquemet ◽  
...  
Keyword(s):  
Nervous System ◽  
Cardiac Function ◽  
Regional Cardiac Function ◽  
Reflex Control

scholarly journals Improved method for quantification of regional cardiac function in mice using phase‐contrast MRI

2011 ◽  
Vol 67 (2) ◽  
pp. 541-551 ◽  
Author(s):  
Erica Dall'Armellina ◽  
Bernd A. Jung ◽  
Craig A. Lygate ◽  
Stefan Neubauer ◽  
Michael Markl ◽  
...  
Keyword(s):  
Cardiac Function ◽  
Phase Contrast ◽  
Improved Method ◽  
Phase Contrast Mri ◽  
Regional Cardiac Function

Accurate estimation of global and regional cardiac function by retrospectively gated multidetector row computed tomography

2006 ◽  
Vol 16 (7) ◽  
pp. 1424-1433 ◽  
Author(s):  
Bénédicte Belge ◽  
Emmanuel Coche ◽  
Agnès Pasquet ◽  
Jean-Louis J. Vanoverschelde ◽  
Bernhard L. Gerber
Keyword(s):  
Computed Tomography ◽  
Cardiac Function ◽  
Accurate Estimation ◽  
Multidetector Row Computed Tomography ◽  
Multidetector Row ◽  
Regional Cardiac Function

scholarly journals Early Hypertension Is Associated With Reduced Regional Cardiac Function, Insulin Resistance, Epicardial, and Visceral Fat

Hypertension ◽  
2008 ◽  
Vol 51 (2) ◽  
pp. 282-288 ◽  
Author(s):  
Anna Maria Sironi ◽  
Alessandro Pingitore ◽  
Sergio Ghione ◽  
Daniele De Marchi ◽  
Barbara Scattini ◽  
...  
Keyword(s):  
Insulin Resistance ◽  
Cardiac Function ◽  
Visceral Fat ◽  
Regional Cardiac Function

A Bioassay Model for Testing the Incapacitating Effects of Exposure To Combustion Product Atmospheres Using Cynomolgus Monkeys

1984 ◽  
Vol 2 (1) ◽  
pp. 20-36 ◽  
Author(s):  
David A. Purser
Keyword(s):  
Central Nervous System ◽  
Carbon Monoxide ◽  
Nervous System ◽  
Cardiac Function ◽  
Conduction Velocity ◽  
Hydrogen Cyanide ◽  
Nerve Conduction ◽  
Nerve Conduction Velocity ◽  
Central Nervous System Depression ◽  
Physiological Tests

A bioassay model has been developed to test the time course and degree of in capacitation produced by exposures to thermal decomposition products from polymeric materials. A battery of physiological tests was used during separate exposures to atmospheres of carbon monoxide, hydrogen cyanide, hypoxia, hypercapnia and heated air. Each atmosphere was designed to simulate one aspect of the conditions commonly encountered in fires. Measurements were made of the animals' respiration, cardiac function and respiratory blood gases. Neurological function was monitored by measurements of the elec troencephalogram, auditory cortical evoked potentials and peripheral nerve con duction velocity. Hypoxia (10% oxygen) caused muscle weakness, a decrease in nerve conduction velocity, abnormal cardiac function accompanied by a fall in blood pressure and central nervous system depression. At 1000 ppm carbon monoxide, venous carboxyhaemoglobin levels reached 30%. There was a reduc tion of nerve conduction velocity and in some cases severe central nervous system depression. At 60 ppm hydrogen cyanide had a slight depressive effect on the central nervous system, while at 80-150 ppm severe central nervous system depression and incapacitation occurred. The main result of 5% carbon dioxide exposure was a three-fold increase in respiratory minute volume. It is concluded that the model is capable of detecting early physiological signs of in capacitation induced by fire conditions. It is suggested that exposures to a com bination of hydrogen cyanide and carbon monoxide with accompanying changes in cerebral blood flow during attempts to escape from fires may be a cause of col lapse and subsequent death.

Sign in / Sign up

Export Citation Format

Share Document