scholarly journals The role of α- and β-adrenergic receptors in some actions of catecholamines on intestinal smooth muscle

1967 ◽  
Vol 188 (3) ◽  
pp. 387-402 ◽  
Author(s):  
D. H. Jenkinson ◽  
I. K. M. Morton
Keyword(s):  
Smooth Muscle ◽  
Adrenergic Receptors ◽  
Intestinal Smooth Muscle ◽  
Β Adrenergic Receptors

Role of stat6 in hypercontractility of murine small intestinal smooth muscle induced by nematode infection

2001 ◽  
Vol 120 (5) ◽  
pp. A534-A534
Author(s):  
A ZHAO ◽  
D MULLOY ◽  
J URBANJR ◽  
W GAUSE ◽  
T SHEADONOHUE
Keyword(s):  
Smooth Muscle ◽  
Nematode Infection ◽  
Intestinal Smooth Muscle ◽  
Small Intestinal

scholarly journals Role of β-Adrenergic Receptors and Nitric Oxide Signaling in Exercise-Mediated Cardioprotection

Physiology ◽  
2013 ◽  
Vol 28 (4) ◽  
pp. 216-224 ◽  
Author(s):  
John W. Calvert ◽  
David J. Lefer
Keyword(s):  
Nitric Oxide ◽  
Risk Factors ◽  
Adrenergic Receptors ◽  
Ischemia Reperfusion Injury ◽  
Ischemia Reperfusion ◽  
Nitric Oxide Signaling ◽  
Factors Associated ◽  
Cardioprotective Effects ◽  
Β Adrenergic Receptors

Exercise promotes cardioprotection in both humans and animals not only by reducing risk factors associated with cardiovascular disease but by reducing myocardial infarction and improving survival following ischemia. This article will define the role that nitric oxide and β-adrenergic receptors play in mediating the cardioprotective effects of exercise in the setting of ischemia-reperfusion injury.

Mesovarial Leiomyomas in Rats in a Chronic Toxicity Study of Soterenol Hydrochloride

1971 ◽  
Vol 8 (5-6) ◽  
pp. 452-457 ◽  
Author(s):  
L. W. Nelson ◽  
W. A. Kelly
Keyword(s):  
Smooth Muscle ◽  
Adrenergic Receptors ◽  
Dose Group ◽  
Chronic Toxicity ◽  
Low Dose ◽  
Toxicity Study ◽  
Ovarian Cysts ◽  
High Dose ◽  
Oral Toxicity ◽  
Β Adrenergic Receptors

In an 18-month oral toxicity study of soterenol hydrochloride, a stimulant of the β-adrenergic receptors, mesovarial leiomyomas were observed in three of 30 low-dose, six of 30 middle-dose and 10 of 30 high-dose rats. There was also an increase in the prevalence of ovarian cysts and of focal hyperplasia of smooth muscle in the mesovaria in the treated rats, especially in the high-dose group.

scholarly journals Adrenergic Regulation of Macrophage-Mediated Innate/Inflammatory Responses in Obesity and Exercise in this Condition: Role of β2 Adrenergic Receptors

2019 ◽  
Vol 19 (8) ◽  
pp. 1089-1099 ◽  
Author(s):  
Eduardo Ortega ◽  
Isabel Gálvez ◽  
Leticia Martín-Cordero
Keyword(s):  
Stress Responses ◽  
Adrenergic Receptors ◽  
Inflammatory Responses ◽  
Low Grade ◽  
Adrenergic Stimulation ◽  
Adrenergic Regulation ◽  
Induce Insulin Resistance ◽  
Antiinflammatory Effects ◽  
Β Adrenergic Receptors

Background: The effects of exercise on the innate/inflammatory immune responses are crucially mediated by catecholamines and adrenoreceptors; and mediations in both stimulatory and anti-inflammatory responses have been attributed to them. Obesity and metabolic syndrome are included among low-grade chronic inflammatory pathologies; particularly because patients have a dysregulation of the inflammatory and stress responses, which can lead to high levels of inflammatory cytokines that induce insulin resistance, contributing to the onset or exacerbation of type 2 diabetes. Macrophages play a crucial role in this obesity-induced inflammation. Although most of the antiinflammatory effects of catecholamines are mediated by β adrenergic receptors (particularly β2), it is not known whether in altered homeostatic conditions, such as obesity and during exercise, innate/ inflammatory responses of macrophages to β2 adrenergic stimulation are similar to those in cells of healthy organisms at baseline. Objective: This review aims to emphasize that there could be possible different responses to β2 adrenergic stimulation in obesity, and exercise in this condition. Methods: A revision of the literature based on the hypothesis that obesity affects β2 adrenergic regulation of macrophage-mediated innate/inflammatory responses, as well as the effect of exercise in this context. Conclusion: The inflammatory responses mediated by β2 adrenoreceptors are different in obese individuals with altered inflammatory states at baseline compared to healthy individuals, and exercise can also interfere with these responses. Nevertheless, it is clearly necessary to develop more studies that contribute to widening the knowledge of the neuroimmune regulation process in obesity, particularly in this context.

Induction of functional β-adrenergic receptors in rat aortic smooth muscle cells by sodium butyrate

1986 ◽  
Vol 35 (21) ◽  
pp. 3813-3820 ◽  
Author(s):  
Ponnal Nambi ◽  
Matthew H. Whitman ◽  
Dulcie B. Schmidt ◽  
Grace D. Heckman ◽  
Frans L. Stassen ◽  
...  
Keyword(s):  
Smooth Muscle ◽  
Smooth Muscle Cells ◽  
Sodium Butyrate ◽  
Adrenergic Receptors ◽  
Muscle Cells ◽  
Aortic Smooth Muscle Cells ◽  
Aortic Smooth Muscle ◽  
Β Adrenergic Receptors

Role of stat6 in hypercontractility of murine small intestinal smooth muscle induced by nematode infection

2001 ◽  
Vol 120 (5) ◽  
pp. A534 ◽  
Author(s):  
Aiping Zhao ◽  
Daniel P. Mulloy ◽  
Joseph F. Urban ◽  
William C. Gause ◽  
Terez Shea-Donohue
Keyword(s):  
Smooth Muscle ◽  
Nematode Infection ◽  
Intestinal Smooth Muscle ◽  
Small Intestinal
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