scholarly journals Effects of electrical stimulation of the aortic nerve on blood pressure and respiration in cats and rabbits under chloralose and nembutal anaesthesia

1949 ◽  
Vol 109 (3-4) ◽  
pp. 392-401 ◽  
Author(s):  
E. Neil ◽  
C. R. M. Redwood ◽  
A. Schweitzer
Keyword(s):  
Blood Pressure ◽  
Electrical Stimulation ◽  
Aortic Nerve ◽  
Stimulation Of

Pressoceptive and chemoceptive aortic reflexes in decorticate and in decerebrate cats

1965 ◽  
Vol 208 (4) ◽  
pp. 708-714 ◽  
Author(s):  
G. Baccelli ◽  
M. Guazzi ◽  
A. Libretti ◽  
A. Zanchetti
Keyword(s):  
Blood Pressure ◽  
Electrical Stimulation ◽  
Cardiovascular Responses ◽  
Decrease Blood Pressure ◽  
Aortic Nerve ◽  
Decerebrate Cats ◽  
Sham Rage ◽  
Stimulation Of

Graded electrical stimulation of three different components of the aortic nerve has been performed in decorticate cats, and later repeated after intercollicular decerebration. Threshold stimuli, involving only the largest fibers probably originating from pressoceptors, slightly decrease blood pressure and respiration both in quiet decorticate and in decerebrate cats. However, in decorticate animals during "spontaneous" fits of sham rage, inhibition of all somatic and visceral components of rage behavior is observed. When aortic nerve stimuli also involve intermediate-threshold fibers presumably of chemoceptive origin, excitation of rage behavior accompanied by strong hypertension and hyperventilation is seen in decorticate preparations, while after decerebration reflex hyperpnea is accompanied by hypotension or by no consistent pressor change. Stronger stimuli activating smaller fibers of probable pressoceptive origin still have conspicuous excitatory effects in decorticate animals, as shown by appearance of rage activity and pressor reactions, whereas after decerebration prominent hypotension with hyperpnea is observed. The predominance of excitatory cardiovascular responses to aortic nerve stimuli in decorticate animals, and of inhibitory reactions after decerebration, is discussed.

scholarly journals Blood Pressure and Autonomic Responses to Electrical Stimulation of the Renal Arterial Nerves Before and After Ablation of the Renal Artery

Hypertension ◽  
2013 ◽  
Vol 61 (2) ◽  
pp. 450-456 ◽  
Author(s):  
Masaomi Chinushi ◽  
Daisuke Izumi ◽  
Kenichi Iijima ◽  
Katsuya Suzuki ◽  
Hiroshi Furushima ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Electrical Stimulation ◽  
Renal Artery ◽  
Autonomic Responses ◽  
Before And After ◽  
Stimulation Of

The Effect of Electrical Stimulation of the Pudendal Nerve on Sciatic Nerve Blood Flow in Animals

2008 ◽  
Vol 26 (3) ◽  
pp. 145-148 ◽  
Author(s):  
Motohiro Inoue ◽  
Tatsuya Hojo ◽  
Miwa Nakajima ◽  
Hiroshi Kitakoji ◽  
Megumi Itoi ◽  
...  
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Electrical Stimulation ◽  
Sciatic Nerve ◽  
Pudendal Nerve ◽  
Systemic Blood Pressure ◽  
Nerve Blood Flow ◽  
Doppler Flowmetry ◽  
Canal Stenosis ◽  
Stimulation Of

Objective To investigate the mechanism of the clinical effect of electroacupuncture of the pudendal nerve on the lumbar and lower limb symptoms caused by lumbar spinal canal stenosis, we studied changes in sciatic nerve blood flow during electrical stimulation of the pudendal nerve in the rat. Methods Using rats (n=5), efferent electrical stimulation to the pudendal nerve was performed and sciatic nerve blood flow was measured with laser Doppler flowmetry. Simultaneously, changes in the blood pressure and cardiac rate were measured. Furthermore, the effect of atropine on these responses to the stimulation was also studied. Results Electrical stimulation of the pudendal nerve significantly increased blood flow in the sciatic nerve transiently without increasing heart rate and systemic blood pressure. The significant increase in the sciatic nerve blood flow disappeared after administration of atropine. Conclusion Electrical stimulation of the pudendal nerve causes a transient and significant increase in sciatic nerve blood flow. This response is eliminated or attenuated by administration of atropine, indicating that it occurs mainly via cholinergic nerves.

Axons of passage may be responsible for fastigial nucleus pressor response

1989 ◽  
Vol 257 (6) ◽  
pp. R1436-R1440
Author(s):  
R. T. Henry ◽  
J. D. Connor
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Electrical Stimulation ◽  
Kainic Acid ◽  
Pulse Width ◽  
Pressor Response ◽  
Fastigial Nucleus ◽  
Postural Asymmetry ◽  
Bilateral Destruction ◽  
Stimulation Of

Bilateral destruction of perikarya in the fastigial nucleus (FN) of the rat with the cytotoxic agent kainic acid (0.5 mg) did not alter the blood pressure (BP) increases observed during monopolar electrical stimulation (100 microA, 50 Hz, 0.5-ms pulse width) of this region. BP increases in control animals were 30 +/- 8 mm Hg, whereas BP increased 30 +/- 7 mmHg in kainic acid-lesioned rats. Furthermore, picrotoxin (100 ng) and muscimol (25 ng) microinjected unilaterally into the FN of conscious, unrestrained rats produced postural asymmetry but no change in BP or heart rate. These data suggest that the FN pressor response may be due, at least in part, to stimulation of axons of passage.

EFFECT UPON BLOOD PRESSURE OF ELECTRICAL STIMULATION OF TIPS OF TEMPORAL LOBES IN MAN

1950 ◽  
Vol 13 (1) ◽  
pp. 65-71 ◽  
Author(s):  
William P. Chapman ◽  
Kenneth E. Livingston ◽  
James L. Poppen
Keyword(s):  
Blood Pressure ◽  
Electrical Stimulation ◽  
Temporal Lobes ◽  
Stimulation Of

Oxytocin release in response to stimulation of cingulate gyrus

1961 ◽  
Vol 200 (3) ◽  
pp. 625-627 ◽  
Author(s):  
Carlos Beyer F. ◽  
Guillermo Anguiano L. ◽  
Flavio Mena J.
Keyword(s):  
Blood Pressure ◽  
Spinal Cord ◽  
Electrical Stimulation ◽  
Blood Stream ◽  
Spinal Cord Transection ◽  
Cingulate Gyrus ◽  
Uterine Contractions ◽  
Oxytocin Release ◽  
Synthetic Oxytocin ◽  
Stimulation Of

Electrical stimulation of the anterior limbic region in Nembutal-anesthetized cats evokes uterine contractions similar to those elicited by the injection of 50–100 mu of synthetic oxytocin. The contractions of the uterus caused by cortical stimulation must be related to release of an oxytocic agent carried through the blood stream, since transection of the spinal cord together with the severance of the vagi did not prevent the contractions. A moderate rise in blood pressure that appeared in the animals with spinal cord transection also suggests release of vasopressin.

Sign in / Sign up

Export Citation Format

Share Document