scholarly journals Dynamic right and left ventricular interactions in the pig

2020 ◽  
Vol 105 (8) ◽  
pp. 1293-1315
Author(s):  
Michael R. Pinsky
Keyword(s):  
Left Ventricular ◽  
Ventricular Interactions

Dynamic right and left ventricular interactions in the rabbit: Simultaneous measurement of ventricular pressure-volume loops

1996 ◽  
Vol 11 (2) ◽  
pp. 65-76 ◽  
Author(s):  
Michael R. Pinsky ◽  
Stefano Perlini ◽  
Pier Luigi Solda ◽  
Paolo Pantaleo ◽  
Alessandro Calciati ◽  
...  
Keyword(s):  
Simultaneous Measurement ◽  
Left Ventricular ◽  
Ventricular Pressure ◽  
Ventricular Interactions

Independence of Intrapericardial Right and Left Ventricular Performance in Septic Pulmonary Hypertension

1990 ◽  
Vol 29 (06) ◽  
pp. 246-251
Author(s):  
J. C. Böck ◽  
Aileen Lim ◽  
H. Eichstädt ◽  
M. Pollycove ◽  
R. F. Lewis ◽  
...  
Keyword(s):  
Pulmonary Hypertension ◽  
Left Ventricular ◽  
Sepsis Group ◽  
Trauma Patients ◽  
Ventricular Performance ◽  
Radionuclide Angiocardiography ◽  
Ventricular Interactions ◽  
Pulmonary Arterial ◽  
The Right ◽  
Equilibrium Radionuclide Angiocardiography

To study the effect of septic pulmonary hypertension on right/left ventricular intrapericardial interactions thirteen trauma patients, seven septic and six non-septic controls, were compared. Ventricular volumes were derived from firstpass or gated equilibrium radionuclide angiocardiography, and related to body surface area. Systemic and pulmonary pressures were measured invasively. Pulmonary arterial pressure was significantly increased in the sepsis group. Although right ventricular end-diastolic volumes were higher in sepsis, left ventricular end-diastolic volumes were not decreased. In terms of intrapericardial right/left ventricular interactions these results indicate that the right and left ventricles operate independently in septic pulmonary hypertension.

scholarly journals Effects of acute right ventricular ischemia on ventricular interactions during prosthetic left ventricular support

1991 ◽  
Vol 102 (4) ◽  
pp. 588-595 ◽  
Author(s):  
David J. Farrar ◽  
Edna Chow ◽  
Peter G. Compton ◽  
Linda Foppiano ◽  
John Woodard ◽  
...  
Keyword(s):  
Right Ventricular ◽  
Left Ventricular ◽  
Ventricular Support ◽  
Ventricular Interactions

scholarly journals Is mitral annular ascent useful in studying left ventricular function through left atrio-ventricular interactions?

2018 ◽  
Vol 70 (3) ◽  
pp. 368-372 ◽  
Author(s):  
Paula M. Hernández Burgos ◽  
Francisco Lopez Menedez ◽  
Maria D. Candales ◽  
Angel López-Candales
Keyword(s):  
Left Ventricular Function ◽  
Ventricular Function ◽  
Left Ventricular ◽  
Ventricular Interactions

Diastolic ventricular interactions in endurance-trained athletes during orthostatic stress

2007 ◽  
Vol 293 (1) ◽  
pp. H409-H415 ◽  
Author(s):  
Ben T. A. Esch ◽  
Jessica M. Scott ◽  
Mark J. Haykowsky ◽  
Don C. McKenzie ◽  
Darren E. R. Warburton
Keyword(s):  
Left Ventricle ◽  
Stroke Volume ◽  
Lower Body Negative Pressure ◽  
Left Ventricular ◽  
Lower Body ◽  
End Diastolic Volume ◽  
Cardiac Responses ◽  
Ventricular Compliance ◽  
Body Positive ◽  
Ventricular Interactions

Enhanced left-ventricular (LV) compliance is a common adaptation to endurance training. This adaptation may have differential effects under conditions of altered venous return. The purpose of this investigation was to assess the effect of cardiac (un)loading on right ventricular (RV) cavity dimensions and LV volumes in endurance-trained athletes and normally active males. Eight endurance-trained (Vo2max, 65.4 ± 5.7 ml·kg−1·min−1) and eight normally active (Vo2max, 45.1 ± 6.0 ml·kg−1·min−1) males underwent assessments of the following: 1) Vo2max, 2) orthostatic tolerance, and 3) cardiac responses to lower-body positive (0–60 mmHg) and negative (0 to −80 mmHg) pressures with echocardiography. In response to negative pressures, echocardiographic analysis revealed a similar decrease in RV end-diastolic cavity area in both groups (e.g., at −80 mmHg: normals, 21.4%; athletes, 20.8%) but a greater decrease in LV end-diastolic volume in endurance-trained athletes (e.g., at −80 mmHg: normals, 32.3%; athletes, 44.4%; P < 0.05). Endurance-trained athletes also had significantly greater decreases in LV stroke volume during lower-body negative pressure. During positive pressures, endurance-trained athletes showed larger increases in LV end-diastolic volume (e.g., at +60 mmHg; normals, 14.1%; athletes, 26.8%) and LV stroke volume, despite similar responses in RV end-diastolic cavity area (e.g., at +60 mmHg: normals, 18.2%; athletes, 24.2%; P < 0.05). This investigation revealed that in response to cardiac (un)loading similar changes in RV cavity area occur in endurance-trained and normally active individuals despite a differential response in the left ventricle. These differences may be the result of alterations in RV influence on the left ventricle and/or intrinsic ventricular compliance.

Mitochondria in Cardiomyopathy: Alterations in Size, Number and Internal Structures

1968 ◽  
Vol 26 ◽  
pp. 126-127
Author(s):  
George Hug ◽  
William K. Schubert
Keyword(s):  
Heart Failure ◽  
Biochemical Analysis ◽  
Left Ventricular ◽  
Size Number ◽  
Internal Structures ◽  
Left Ventricular Outflow ◽  
Chest X Ray ◽  
Myocardial Fibers ◽  
Muscle Biopsies ◽  
Needle Liver Biopsy

A white boy six months of age was hospitalized with respiratory distress and congestive heart failure. Control of the heart failure was achieved but marked cardiomegaly, moderate hepatomegaly, and minimal muscular weakness persisted.At birth a chest x-ray had been taken because of rapid breathing and jaundice and showed the heart to be of normal size. Clinical studies included: EKG which showed biventricular hypertrophy, needle liver biopsy which showed toxic hepatitis, and cardiac catheterization which showed no obstruction to left ventricular outflow. Liver and muscle biopsies revealed no biochemical or histological evidence of type II glycogexiosis (Pompe's disease). At thoracotomy, 14 milligrams of left ventricular muscle were removed. Total phosphorylase activity in the biopsy specimen was normal by biochemical analysis as was the degree of phosphorylase activation. By light microscopy, vacuoles and fine granules were seen in practically all myocardial fibers. The fibers were not hypertrophic. The endocardium was not thickened excluding endocardial fibroelastosis. Based on these findings, the diagnosis of idiopathic non-obstructive cardiomyopathy was made.

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