An observational thoracic radiographic study of aortic remodeling in dogs with confirmed systemic hypertension

2022 ◽  
Author(s):  
Merrilee Holland ◽  
Judith Hudson ◽  
Erik Hofmeister
Keyword(s):  
Systemic Hypertension ◽  
Radiographic Study ◽  
Aortic Remodeling

Coarctation of the abdominal aorta: Demonstration of a rare anomaly using contrast-enhanced MR-angiography

VASA ◽  
2009 ◽  
Vol 38 (1) ◽  
pp. 66-71 ◽  
Author(s):  
Schubert
Keyword(s):  
Abdominal Aorta ◽  
Mr Angiography ◽  
Parallel Imaging ◽  
Left Renal Vein ◽  
Systemic Hypertension ◽  
Contrast Enhanced ◽  
Younger Age ◽  
Maximum Intensity Projections ◽  
Collateral Vessels ◽  
First Time

We describe a case of aortic coarctation at the level of the infrarenal abdominal aorta which is encountered in less than six individuals in one million. In contrast to aortic narrowing above or including the renal arteries, this seems to be a relatively benign anomaly without systemic hypertension or impaired renal function. For the first time in this type of anomaly, contrast-enhanced MR angiography (ce-MRA) on a multi-receiver channel MR system, with an 8-channel phased array coil and parallel imaging was used. Ce-MRA displayed a tortuous, narrowed aortic segment that was found to be associated with mesenteric artery stenosis and compression of the orthotopic left renal vein, also known as the nutcracker phenomenon. All major aortic branches could be depicted using 3D surface-shaded displays and subvolume maximum intensity projections (MIPs). Collateral vessels of the abdominal wall were identified using whole-volume MIPs. Since the majority of aortic malformations are diagnosed at a younger age, and many suffer from renal insufficiency, we conclude that ce-MRA will eventually place conventional DSA as the modality of choice in malformations of the abdominal aorta.

Dual lumen intervention for aortic dissection: long-term impact on aortic remodeling

2020 ◽  
Vol 27 (1) ◽  
Author(s):  
Xun Yuan ◽  
Andreas Mitsis ◽  
Thomas Semple ◽  
Michael Rubens ◽  
Christoph A. Nienaber
Keyword(s):  
Aortic Dissection ◽  
Long Term Impact ◽  
Aortic Remodeling

Spontaneous intracerebral hemorrhage in a child with systemic hypertension and adrenal adenoma

2007 ◽  
Vol 107 (4) ◽  
pp. 328-329
Author(s):  
Oszkar Szentirmai ◽  
Ken R. Winston ◽  
Paul Jedlicka ◽  
Gary M. Lum
Keyword(s):  
Intracerebral Hemorrhage ◽  
Adrenal Adenoma ◽  
Systemic Hypertension ◽  
Spontaneous Intracerebral Hemorrhage

Epicardial Coronary Artery Remodeling in Systemic Hypertension

2005 ◽  
Vol 12 (3) ◽  
pp. 164
Author(s):  
M. Koz??kov?? ◽  
M. Paterni ◽  
F. Bartolomucci ◽  
C. Morizzo ◽  
C. Palombo
Keyword(s):  
Coronary Artery ◽  
Systemic Hypertension ◽  
Epicardial Coronary Artery ◽  
Artery Remodeling

Cardiovascular Changes in Menopause

2020 ◽  
Vol 16 ◽  
Author(s):  
Anjana R Nair ◽  
Aiswarya J Pillai ◽  
Nandini Nair
Keyword(s):  
Cardiovascular Risk ◽  
Structural Changes ◽  
Systemic Hypertension ◽  
Metabolic Dysfunction ◽  
Review Of Literature ◽  
Coronary Anatomy ◽  
Complex Interactions ◽  
Fat Redistribution ◽  
Current Therapies ◽  
Cardiovascular Aging

: Menopause is associated with changes consistent with cardiovascular aging. The effects on cardiac disease is multifaceted affecting endothelial function, coronary artery physiology and metabolic dysfunction leading to structural changes in the coronary anatomy. A systematic review of literature from 1986 to 2019 was conducted using PubMed and Google Scholar. The search was directed to retrieve papers that addressed the changes in cardiovascular physiology in menopause and the current therapies available to treat cardiovascular manifestations of menopause. The metabolic and clinical factors secondary to menopause such as dyslipidemia, insulin resistance, fat redistribution and systemic hypertension contribute to the accelerated risk for cardiovascular aging and disease. Atherosclerosis appears to be the end result of the interaction between cardiovascular risk factors and their accentuation during the perimenopausal period. Additionally, complex interactions between oxidative stress and levels of L-arginine and ADMA may also influence endothelial dysfunction in menopause. The increased cardiovascular risk in menopause stems from the exaggerated effects of changing physiology on the cardiovascular system affecting peripheral, cardiac and cerebrovascular beds. The differential effects of menopause on cardiovascular disease at the subclinical, biochemical and molecular levels form the highlights of this review.

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