Histone H3K4 methyltransferases SDG25 and ATX1 maintain heat‐stress gene expression during recovery in Arabidopsis

2021 ◽  
Author(s):  
Ze‐Ting Song ◽  
Lin‐Lin Zhang ◽  
Jia‐Jia Han ◽  
Ming Zhou ◽  
Jian‐Xiang Liu
Keyword(s):  
Gene Expression ◽  
Heat Stress ◽  
Histone H3k4 ◽  
H3k4 Methyltransferases ◽  
Stress Gene

Heat stress and sudden infant death syndrome—Stress gene expression after exposure to moderate heat stress

2013 ◽  
Vol 232 (1-3) ◽  
pp. 16-24 ◽  
Author(s):  
Marianne Cathrine Rohde ◽  
Thomas Juhl Corydon ◽  
Jakob Hansen ◽  
Christina Bak Pedersen ◽  
Stinne P. Schmidt ◽  
...  
Keyword(s):  
Gene Expression ◽  
Heat Stress ◽  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Sudden Infant ◽  
Stress Gene

scholarly journals Specific Interaction between Tomato HsfA1 and HsfA2 Creates Hetero-oligomeric Superactivator Complexes for Synergistic Activation of Heat Stress Gene Expression

2009 ◽  
Vol 284 (31) ◽  
pp. 20848-20857 ◽  
Author(s):  
Kwan Yu Chan-Schaminet ◽  
Sanjeev K. Baniwal ◽  
Daniela Bublak ◽  
Lutz Nover ◽  
Klaus-Dieter Scharf
Keyword(s):  
Gene Expression ◽  
Heat Stress ◽  
Specific Interaction ◽  
Synergistic Activation ◽  
Stress Gene

scholarly journals HSP GENE EXPRESSION IN WHEAT UNDER HEAT STRESS

2016 ◽  
pp. 55 ◽  
Author(s):  
Ирина Александровна Нилова ◽  
Людмила Владимировна Топчиева ◽  
Александр Федорович Титов ◽  
Irina Nilova ◽  
Lyudmila Topchieva ◽  
...  
Keyword(s):  
Gene Expression ◽  
Heat Stress

scholarly journals Disruption of TFIIH activities generates a stress gene expression response and reveals possible new targets against cancer

2019 ◽  
Author(s):  
Maritere Urioistegui-Arcos ◽  
Rodrigo Aguayo-Ortiz ◽  
María del Pilar Valencia-Morales ◽  
Erika Melchy-Pérez ◽  
Yvonne Rosenstein ◽  
...  
Keyword(s):  
Gene Expression ◽  
Tumour Growth ◽  
Transformed Cells ◽  
Rna Seq ◽  
Gene Expression Response ◽  
New Targets ◽  
Increased Sensitivity ◽  
Promoter Occupancy ◽  
Expression Response ◽  
Stress Gene

AbstractDisruption of the enzymatic activities of the transcription factor TFIIH by Triptolide (TPL) or THZ1 could be used against cancer. Here, we used an oncogenesis model to compare the effect of TFIIH inhibitors between transformed cells and their progenitors. We report that tumour cells exhibited highly increased sensitivity to TPL or THZ1 and that the combination of both had an additive effect. TPL affects the interaction between XPB and P52, causing a reduction in the levels of XPB, P52, and P8, but not other TFIIH subunits. RNA-Seq and RNAPII-ChIP-Seq experiments showed that although the levels of many transcripts were reduced, the levels of a significant number were increased after TPL treatment, with maintained or increased RNAPII promoter occupancy. A significant number of these genes encode for factors that have been related to tumour growth and metastasis. Some of these genes were also overexpressed in response to THZ1, which depletion enhances the toxicity of TPL and are possible new targets against cancer.

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