Increasing net immunosuppression after BK polyoma virus infection

2020 ◽  
Author(s):  
Laura Cotiguala ◽  
Ahmad Masood ◽  
Jeong M. Park ◽  
Milagros D. Samaniego‐Picota ◽  
Daniel R. Kaul ◽  
...  
Keyword(s):  
Virus Infection ◽  
Polyoma Virus

604: Polyoma Virus Infection as an Early Marker for Bladder Transitional Cell Carcinoma

2005 ◽  
Vol 173 (4S) ◽  
pp. 165-165
Author(s):  
Paolo Gontero ◽  
Elisabetta Omodeo-Zorini ◽  
Paola Cacciotti ◽  
Filippo Sogni ◽  
Ervin Kocjancic ◽  
...  
Keyword(s):  
Virus Infection ◽  
Transitional Cell Carcinoma ◽  
Cell Carcinoma ◽  
Transitional Cell ◽  
Polyoma Virus ◽  
Bladder Transitional Cell Carcinoma ◽  
Early Marker

Diagnosis of Polyoma Virus Infection In Renal Transplant Recipients

1999 ◽  
Vol 5 (S2) ◽  
pp. 1166-1167
Author(s):  
J. A. C. King ◽  
D. N. Howell ◽  
J. A. Tucker ◽  
R. P. Lowry
Keyword(s):  
Renal Transplant ◽  
Virus Infection ◽  
Renal Transplant Patient ◽  
Bk Virus ◽  
Polyoma Virus ◽  
Immunosuppressed Patient ◽  
Renal Transplant Recipients ◽  
Transplant Recipients ◽  
Immunosuppressed Patients ◽  
Cadaver Kidney

BK polyoma virus is a 40-45 nm DNA virus that was first identified in the urine of an immunosuppressed patient in 1971. BK virus infection often occurs in childhood and is subclinical. The majority of adults have antibodies to the virus. Asymptomatic viruria, ureteral ulceration, and ureteral stenosis have been described with infection in immunosuppressed patients. Renal graft dysfunction and interstitial nephritis have been associated with BK virus. Differentiation between rejection and infection is important. We report a case of a renal transplant patient with BK polyoma virus infection. This case illustrates the variety of methods that can be used for identification of BK polyoma virus.The patient, a 52 year old female, presented with an asymptomatic rise in serum creatinine (1.4 to 2.1 mg/dl). Her past medical history was significant for a cadaver kidney transplant two years prior for polycystic kidney disease.

Role of interferon in the pathogenesis of virus diseases in mice as demonstrated by the use of anti-interferon serum. VI. Polyoma virus infection

1979 ◽  
Vol 24 (2) ◽  
pp. 178-183 ◽  
Author(s):  
Ion Gresser ◽  
Chantal Maury ◽  
Chantal Kress ◽  
Daniel Blangy ◽  
Marie-Thérèse Maunoury
Keyword(s):  
Virus Infection ◽  
Polyoma Virus ◽  
Virus Diseases

scholarly journals Urine cytology screening for polyoma virus infection following renal transplantation: the Oxford experience

2006 ◽  
Vol 60 (8) ◽  
pp. 927-930 ◽  
Author(s):  
T. P Thamboo ◽  
K. J M Jeffery ◽  
P. J Friend ◽  
G. D H Turner ◽  
I. S D Roberts
Keyword(s):  
Renal Transplantation ◽  
Virus Infection ◽  
Polyoma Virus ◽  
Urine Cytology ◽  
Cytology Screening

scholarly journals STUDIES OF MOUSE POLYOMA VIRUS INFECTION

1959 ◽  
Vol 109 (5) ◽  
pp. 449-462 ◽  
Author(s):  
Wallace P. Rowe ◽  
Janet W. Hartley ◽  
Lloyd W. Law ◽  
Robert J. Huebner ◽  
Keyword(s):  
Virus Infection ◽  
Polyoma Virus ◽  
Mouse Strains ◽  
Adult Mice

Eight mouse colonies were surveyed for prevalence of antibody to mouse polyoma virus. Frequency of HI antibody varied from 0 to 84 per cent in adult mice in different colonies. Antibody was infrequent in mice less than 3 months of age, and increased in frequency with age. There was no evidence that infection was specific for particular mouse strains. The highest frequency of infection was found in colonies in which breeding mice are housed in proximity to mice inoculated with polyoma virus or passage tumors, and within an infected colony, the incidence of infection was greatest in rooms housing mice inoculated with polyoma virus. Mice from a colony free of antibody became infected when held in room or cage contact with virus-inoculated mice, but at very low rates except in mothers of inoculated litters. These results were interpreted as indicating that artificial contamination of the environment is an important factor in determining the prevalence of infection in the colonies observed. There was no correlation between polyoma infection and spontaneous leukemia in AK. mice.

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