Perceived social stigma, self‐concealment, and suicide risk among North Korean refugee women exposed to traumatic events

Author(s):  
Boyoung Nam ◽  
JoonBeom Kim ◽  
Wonjung Ryu ◽  
Dam I Kim ◽  
Jodi J. Frey ◽  
...  
2020 ◽  
pp. 1-19
Author(s):  
Soim Park ◽  
Pamela J. Surkan ◽  
Peter J. Winch ◽  
Jin-Won Kim ◽  
Joel Gittelsohn

2021 ◽  
pp. 1-11
Author(s):  
Renato Polimanti ◽  
Frank R. Wendt

Abstract Posttraumatic stress disorder (PTSD) is a complex mental disorder afflicting approximately 7% of the population. The diverse number of traumatic events and the wide array of symptom combinations leading to PTSD diagnosis contribute substantial heterogeneity to studies of the disorder. Genomic and complimentary-omic investigations have rapidly increased our understanding of the heritable risk for PTSD. In this review, we emphasize the contributions of genome-wide association, epigenome-wide association, transcriptomic, and neuroimaging studies to our understanding of PTSD etiology. We also discuss the shared risk between PTSD and other complex traits derived from studies of causal inference, co-expression, and brain morphological similarities. The investigations completed so far converge on stark contrasts in PTSD risk between sexes, partially attributed to sex-specific prevalence of traumatic experiences with high conditional risk of PTSD. To further understand PTSD biology, future studies should focus on detecting risk for PTSD while accounting for substantial cohort-level heterogeneity (e.g. civilian v. combat-exposed PTSD cases or PTSD risk among cases exposed to specific traumas), expanding ancestral diversity among study cohorts, and remaining cognizant of how these data influence social stigma associated with certain traumatic events among underrepresented minorities and/or high-risk populations.


2020 ◽  
Vol 33 (3) ◽  
pp. 299-327
Author(s):  
Stephanie Lloyd ◽  
Alexandre Larivée

ArgumentIn this article, we trace shifting narratives of trauma within psychiatric, neuroscience, and environmental epigenetics research. We argue that two contemporary narratives of trauma – each of which concerns questions of time and psychopathology, of the past invading the present – had to be stabilized in order for environmental epigenetics models of suicide risk to be posited. Through an examination of these narratives, we consider how early trauma came to be understood as playing an etiologically significant role in the development of suicide risk. Suicide, in these models, has come to be seen as a behavior that has no significant precipitating event, but rather an exceptional precipitating neurochemical state, whose origins are identified in experiences of early traumatic events. We suggest that this is a part of a broader move within contemporary neurosciences and biopsychiatry to see life as post: seeing life as specific form of post-traumatic subjectivity.


Author(s):  
Jeongwon Baik ◽  
Young Ji Yoon ◽  
Priscilla Gibson ◽  
Nancy Lo ◽  
Hee Eun Nam ◽  
...  

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