Transradial Approach for Cardiovascular Interventions and Its Implications for Hemodialysis Vascular Access

2012 ◽  
Vol 26 (3) ◽  
pp. E20-E29 ◽  
Author(s):  
Shouwen Wang ◽  
Arif Asif
Keyword(s):  
Vascular Access ◽  
Transradial Approach ◽  
Hemodialysis Vascular Access ◽  
Cardiovascular Interventions

Diagnostic ultrasound imaging of hemodialysis vascular access-related complications

Choonpa Igaku ◽  
2019 ◽  
Vol 46 (2) ◽  
pp. 171-180
Author(s):  
Hiroki KOBAYASHI ◽  
Kotaro SUEMITSU ◽  
Masayoshi NANAMI
Keyword(s):  
Ultrasound Imaging ◽  
Vascular Access ◽  
Diagnostic Ultrasound ◽  
Hemodialysis Vascular Access

Differences in Initial Hemodialysis Vascular Access Use Among Glomerulonephritis Subtypes in the United States

2016 ◽  
Vol 67 (4) ◽  
pp. 638-647 ◽  
Author(s):  
Michelle M. O’Shaughnessy ◽  
Maria E. Montez-Rath ◽  
Yuanchao Zheng ◽  
Richard A. Lafayette ◽  
Wolfgang C. Winkelmayer
Keyword(s):  
United States ◽  
Vascular Access ◽  
The United States ◽  
Hemodialysis Vascular Access

Care pathway reduces hospitalizations and cost for hemodialysis vascular access surgery

1997 ◽  
Vol 30 (4) ◽  
pp. 525-531 ◽  
Author(s):  
Bryan N. Becker ◽  
Randee Breiterman-White ◽  
William Nylander ◽  
David Van Buren ◽  
Chris Fotiadis ◽  
...  
Keyword(s):  
Vascular Access ◽  
Care Pathway ◽  
Vascular Access Surgery ◽  
Hemodialysis Vascular Access ◽  
Access Surgery

scholarly journals Medicare Reimbursement Policies and Hemodialysis Vascular Access Outcomes: A Need for Change: Table 1.

2011 ◽  
Vol 22 (3) ◽  
pp. 426-430 ◽  
Author(s):  
Michael Allon ◽  
Lesley Dinwiddie ◽  
Eduardo Lacson ◽  
Derrick L. Latos ◽  
Charmaine E. Lok ◽  
...  
Keyword(s):  
Vascular Access ◽  
Medicare Reimbursement ◽  
Hemodialysis Vascular Access

scholarly journals Hemodialysis vascular access preferences and outcomes in the Dialysis Outcomes and Practice Patterns Study (DOPPS)

2002 ◽  
Vol 61 (6) ◽  
pp. 2266-2271 ◽  
Author(s):  
Eric W. Young ◽  
Dawn M. Dykstra ◽  
David A. Goodkin ◽  
Donna L. Mapes ◽  
Robert A. Wolfe ◽  
...  
Keyword(s):  
Vascular Access ◽  
Practice Patterns ◽  
Hemodialysis Vascular Access ◽  
Dialysis Outcomes

Abstract 466: CX3CR1 Down Regulation Reduces Venous Neointimal Hyperplasia Formation in a Murine Hemodialysis Vascular Access Model

2017 ◽  
Vol 37 (suppl_1) ◽  
Author(s):  
Binxia Yang ◽  
Sreenivaslisu Kilari ◽  
Deborah L McCall ◽  
John Broadwater ◽  
Steven Kerr ◽  
...  
Keyword(s):  
Cell Density ◽  
Vascular Access ◽  
Jugular Vein ◽  
Neointimal Hyperplasia ◽  
Genetically Engineered ◽  
Vehicle Group ◽  
Immunohistochemistry Analysis ◽  
Hemodialysis Vascular Access ◽  
Histology And Immunohistochemistry ◽  
Hemodialysis Arteriovenous Fistula

Introduction: Venous neointimal hyperplasia (VNH) is a major cause of hemodialysis arteriovenous fistula (AVF) vascular access failure. CX3CR1 mediates macrophage infiltration into the vasculature. Mice were used which had been genetically engineered to knock in the human CX3CR1 gene. Hypothesis: The hypothesis to be tested is that increased CX3CR1 gene expression results in VNH formation associated with AVF. Methods: We used 50 CX3CR1 knock in mice which were divided into 2 groups: 1. CX3CR1 antibody (30 mg/kg administered intraperitoneally two times per week) or vehicle (equal amount of volume used for CX3CR1 antibody administered intraperitoneally). AVFs were created by connecting the carotid artery to ipsilateral jugular vein 28 days after nephrectomy was performed to induce chronic kidney disease. Histology and Immunohistochemistry analysis at the outflow vein of AVF were performed to assess the pathological changes. Results: There was a significant decrease in neointima area in the outflow veins of AVF in CX3CR1 antibody group compared to the vehicle group at day 28 (43085 ± 12678 vs. 58963.4 ± 9273 μm 2 , P<0.05). There was a significant decrease in the ratio of neointima/media+adventitia area in the outflow veins of AVF in CX3CR1 antibody group compared to the vehicle group at day 28 (0.34 ± 0.1 vs. 0.61 ± 0.1, P<0.05). Cell density in neointima area in CX3CR1 group outflow veins was significantly lower than the vehicle group at day 14 (9600 ± 1000 vs. 13000 ± 3000 /mm 2 , P<0.01). There was a significant decrease in the average CD68-positive cell density in the CX3CR1 group outflow veins compared to the vehicle group (0.96 ± 0.34 vs.15.9 ±10, P<0.001) at day 14. Conclusion: Decreasing CX3CR1 significantly reduces macrophages infiltration and results in a significant reduction in VNH. This study provides a rationale for using CX3CR1 antibody in reducing VNH formation.

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