Overcoming behavioral obstacles to prevent periodontal disease: Behavioral change techniques and self‐performed periodontal infection control

2020 ◽  
Vol 84 (1) ◽  
pp. 134-144 ◽  
Author(s):  
Birgitta Jönsson ◽  
Kajsa H. Abrahamsson
Keyword(s):  
Periodontal Disease ◽  
Infection Control ◽  
Behavioral Change ◽  
Periodontal Infection ◽  
Behavioral Change Techniques

scholarly journals Applications of Photodynamic Therapy in Management of Periodontal Disease - A Review

2018 ◽  
Vol 08 (04) ◽  
pp. 024-027
Author(s):  
Shiny Inasu ◽  
Biju Thomas
Keyword(s):  
Photodynamic Therapy ◽  
Periodontal Disease ◽  
Infection Control ◽  
Singlet Oxygen ◽  
Current Status ◽  
Target Cells ◽  
Oral Diseases ◽  
Dynamic Therapy ◽  
Reactive Agents ◽  
Photo Dynamic Therapy

AbstractA novel noninvasive photochemical approach for infection control, namely photodynamic therapy, has received much attention in the treatment of oral diseases which requires three nontoxic ingredients namely visible harmless light, a photosensitizer and oxygen are involved in this therapy. It is based on the principle that a photosensitizer binds to the target cells which when activated by light of a suitable wavelength results in the production of singlet oxygen and other very reactive agents that are extremely toxic to certain cells and bacteria. This article highlights the application of photo-dynamic therapy in management of periodontal disease and its current status.

scholarly journals Relation between chronic periodontal disease and plasmatic levels of triglycerides, total cholesterol and fractions

2005 ◽  
Vol 19 (4) ◽  
pp. 284-289 ◽  
Author(s):  
Ana Cristina Posch Machado ◽  
Maria Rozeli de Souza Quirino ◽  
Luiz Fernando Costa Nascimento
Keyword(s):  
Periodontal Disease ◽  
Total Cholesterol ◽  
Blood Lipid ◽  
Oral Disease ◽  
Whole Body ◽  
Lipid Levels ◽  
Periodontal Infection ◽  
Chronic Periodontal Disease ◽  
Systemic Health ◽  
Blood Lipid Levels

Many people in the world are affected by hyperlipidemia, which is a known risk factor for atherosclerotic disease. On the other hand, periodontitis, a prevalent oral disease, has been connected to several systemic health changes, including an altered lipid metabolism. Transient and recurrent bacteremias, which may be caused by periodontal infection, induce an intense local and systemic inflammatory response, leading to changes in the whole body. The aim of the present study was to verify the relationship between severe and moderate periodontal disease and blood lipid levels. Sixty individuals seen at the clinics of the University of Taubaté, São Paulo, over 20 years old, were divided into two groups, with and without periodontitis, and paired according to sex and age. Their levels of total cholesterol, tryglicerides and fractions were determined. Variables related to high cholesterol levels, including age, sex and body mass index, were evaluated. The values recommended by the Brazilian Society of Cardiology were considered to classify lipidemia. The results showed that mean levels of cholesterol (192.1 mg/dl ± 40.9) and triglycerides (153.5 mg/dl ± 105.6) in individuals with periodontitis were higher than, but not statistically different from, those of individuals without periodontitis (186.1 mg/dl ± 35.4 and 117.5 mg/dl ± 68, respectively). Therefore, this study has demonstrated that there is no significant relationship between periodontal disease, regardless of its intensity, and blood lipid levels in the studied population.

scholarly journals Invasion of Oral and Aortic Tissues by Oral Spirochete Treponema denticola in ApoE−/−Mice Causally Links Periodontal Disease and Atherosclerosis

2014 ◽  
Vol 82 (5) ◽  
pp. 1959-1967 ◽  
Author(s):  
Sasanka S. Chukkapalli ◽  
Mercedes F. Rivera ◽  
Irina M. Velsko ◽  
Ju-Youn Lee ◽  
Hao Chen ◽  
...  
Keyword(s):  
Periodontal Disease ◽  
Alveolar Bone ◽  
Tissue Growth ◽  
Aortic Tissue ◽  
Treponema Denticola ◽  
Rapid Progression ◽  
Causative Role ◽  
Igg Antibody ◽  
Content Type ◽  
Periodontal Infection

ABSTRACTTreponema denticolais a predominantly subgingival oral spirochete closely associated with periodontal disease and has been detected in atherosclerosis. This study was designed to evaluate causative links between periodontal disease induced by chronic oralT. denticolainfection and atherosclerosis in hyperlipidemic ApoE−/−mice. ApoE−/−mice (n= 24) were orally infected withT. denticolaATCC 35404 and were euthanized after 12 and 24 weeks.T. denticolagenomic DNA was detected in oral plaque samples, indicating colonization of the oral cavity. Infection elicited significantly (P= 0.0172) higher IgG antibody levels and enhanced intrabony defects than sham infection.T. denticola-infected mice had higher levels of horizontal alveolar bone resorption than sham-infected mice and an associated significant increase in aortic plaque area (P≤ 0.05). Increased atherosclerotic plaque correlated with reduced serum nitric oxide (NO) levels and increased serum-oxidized low-density lipoprotein (LDL) levels compared to those of sham-infected mice.T. denticolainfection altered the expression of genes known to be involved in atherosclerotic development, including the leukocyte/endothelial cell adhesion gene (Thbs4), the connective tissue growth factor gene (Ctgf), and the selectin-E gene (Sele). Fluorescentin situhybridization (FISH) revealedT. denticolaclusters in both gingival and aortic tissue of infected mice. This is the first study examining the potential causative role of chronicT. denticolaperiodontal infection and vascular atherosclerosisin vivoin hyperlipidemic ApoE−/−mice.T. denticolais closely associated with periodontal disease and the rapid progression of atheroma in ApoE−/−mice. These studies confirm a causal link for active oralT. denticolainfection with both atheroma and periodontal disease.

Periodontal infection control: current clinical concepts

2006 ◽  
Vol 13 (1) ◽  
pp. 3-12 ◽  
Author(s):  
JAN L. WENNSTRÖM ◽  
CRISTIANO TOMASI
Keyword(s):  
Infection Control ◽  
Control Current ◽  
Periodontal Infection

scholarly journals Possible interaction between visfatin, periodontal infection, and other systemic diseases: A brief review of literature

2017 ◽  
Vol 11 (03) ◽  
pp. 407-410 ◽  
Author(s):  
Mojtaba Bayani ◽  
Mohammad Pourali ◽  
Mohammad Keivan
Keyword(s):  
Periodontal Disease ◽  
Bacterial Infections ◽  
Alveolar Bone ◽  
Gingival Crevicular Fluid ◽  
Systemic Diseases ◽  
Hand Search ◽  
Periodontal Infection ◽  
Etiologic Agents ◽  
Visfatin Level ◽  
Crevicular Fluid

ABSTRACTGingivitis and periodontitis are common bacterial infections caused by a variety of microorganisms. Despite the microorganisms' roles as etiologic agents, inflammation-induced substances also have crucial parts in the loss of connective tissue and the supporting alveolar bone. Visfatin is a pleiotropic mediator, which acts as growth factor, cytokine, and pre-B-cell colony-enhancing factor. A positive correlation was detected between the serum/plasma levels of visfatin and inflammatory disorders such as diabetes mellitus and cardiovascular disease. In addition, the visfatin level was higher in saliva and the gingival crevicular fluid (GCF) of subjects with periodontal disease. This review defined current, predictable patterns of possible interaction of visfatin with periodontal infection and other systemic diseases, using PubMed and Medline databases searching for articles written in English. Peer-reviewed articles were targeted using the following keywords: “visfatin,” “periodontal disease,” “inflammatory mediator,” and “biomarker.” Available full-text articles were read, and related articles were also scrutinized, while a hand search was also performed. Search was confined to human studies, and articles written in English and published between 1985 and 2016 were selected. It was concluded that periodontal infection and other systemic diseases could be related to the levels of visfatin in GCF, saliva, and serum as a biomarker of these diseases.

scholarly journals Analysis of behavioral change techniques in community-led total sanitation programs

2014 ◽  
Vol 30 (1) ◽  
pp. 16-28 ◽  
Author(s):  
Rachel Sigler ◽  
Lyana Mahmoudi ◽  
Jay Paul Graham
Keyword(s):  
Behavioral Change ◽  
Behavioral Change Techniques

scholarly journals Diabetes mellitus and periodontal diseases: A two way relationship

2021 ◽  
Vol 6 (2) ◽  
pp. 43-56
Author(s):  
Gowhar Nazir ◽  
◽  
Josee Amin ◽  
Keyword(s):  
Diabetes Mellitus ◽  
Periodontal Disease ◽  
Inflammatory Diseases ◽  
Periodontal Diseases ◽  
Systemic Effect ◽  
Diabetic Patients ◽  
Periodontal Infection ◽  
Bidirectional Relationship ◽  
Patients With Diabetes ◽  
Global Health Care

Diabetes mellitus and periodontits are both highly prevalent chronic inflammatory diseases. Both diseases share the same risk factors and are a significant global health care burden adversely affecting the quality of life. Evidence from various studies have demonstrated that diabetes is a major risk factor for periodontal disease and is associated with increased incidence, prevalence and severity of periodontal disease. Hyperglycemia associated with diabetes mellitus results in an increased host immunoinflammatory response which adversely affects the periodontal health. Conversely, periodontits is associated with poor metabolic control in patients with diabetes and increased development of diabetic complications suggesting a bidirectional relationship between the two diseases. Periodontal infection via bacteremia exerts a wide systemic effect by contributing to chronic systemic inflammatory burden worsening diabetic state by increasing insulin resistance. Moreover, studies have demonstrated an improvement in glycemic control following periodontal therapy in prediabetic and diabetic patients with periodontitis.

scholarly journals The Bi-Directional Relationship between Periodontal Disease and Hyperlipidemia

2008 ◽  
Vol 02 (02) ◽  
pp. 142-149 ◽  
Author(s):  
Ozlem Fentoglu ◽  
F. Yesim Bozkurt
Keyword(s):  
Risk Factors ◽  
Cardiovascular Disease ◽  
Periodontal Disease ◽  
Serum Lipids ◽  
Plasma Lipids ◽  
Periodontal Infection ◽  
Increased Risk ◽  
Potential Link ◽  
Common Risk Factors ◽  
Accumulation Of Lipids

ABSTRACTIt has been proposed for several decades that infections may be responsible for the accelerated development of atherosclerosis. The initiation of the atherosclerotic plaque is ascribed to focal accumulation of lipids. This explains the importance of plasma lipids in the development of atherosclerosis. Recent reports point towards a possible association between periodontal disease and increased risk for cardiovascular disease. Thus, periodontitis and cardiovascular disease may share common risk factors, and association between periodontitis and coronary heart disease may be due to the elevated levels of plasma lipids. Epidemiological and clinical studies have also suggested that there is a relationship between periodontal disease and impaired lipid metabolism. In this review, we summarized the potential link mechanisms in the association between periodontal infection and serum lipids. (Eur J Dent 2008;2:142-149)

Gum Bugs Hit Heart: State-of-the-Science

2011 ◽  
Vol 5 (3) ◽  
pp. 164-167
Author(s):  
Kaushal Luthra ◽  
Sarita Luthra ◽  
Nidhi Aggarwal ◽  
Shaily Luthra
Keyword(s):  
Periodontal Disease ◽  
Direct Effects ◽  
Periodontal Bacteria ◽  
Gingival Bleeding ◽  
Periodontal Infection ◽  
Chronic Periodontal Disease ◽  
Immunological Mechanisms ◽  
Microbial Infections ◽  
State Of The Science ◽  
Molecular Components

ABSTRACT Gingival bleeding is a hallmark of chronic periodontal disease; it is likely that the bacteria associated with this disease may produce a low level bacteraemia over extended time periods and the periodontal bacteria enter the bloodstream and travel to major organs like the heart. Microbial infections associated with periodontal disease contribute to cardiovascular disease via transient bacteraemia. Inflammatory and immunological mechanisms which may link periodontal infection and athermanous diseases may be numerous and diverse, potentially involving various cellular and molecular components, direct effects of bacteremia and indirect effects of periodontal tissue inflammation.

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