scholarly journals Treatment change and coronary artery abnormality in incomplete Kawasaki disease

2020 ◽  
Vol 62 (7) ◽  
pp. 779-784
Author(s):  
Yuichi Nomura ◽  
Mayumi Yashiro ◽  
Kiminori Masuda ◽  
Yoshikazu Nakamura
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Incomplete Kawasaki Disease ◽  
Treatment Change ◽  
Coronary Artery Abnormality

scholarly journals The Incidence of Coronary Artery Abnormality in Incomplete Kawasaki Disease

2003 ◽  
Vol 53 (1) ◽  
pp. 164-164 ◽  
Author(s):  
Tomoyoshi Sonobe ◽  
Seijiro Aso ◽  
Yoshio Imada ◽  
Keiji Tsuchiya ◽  
Yosikazu Nakamura ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Incomplete Kawasaki Disease ◽  
Coronary Artery Abnormality

Prevalence of coronary artery abnormality in incomplete Kawasaki disease

2007 ◽  
Vol 49 (4) ◽  
pp. 421-426 ◽  
Author(s):  
TOMOYOSHI SONOBE ◽  
NOBUYUKI KIYOSAWA ◽  
KEIJI TSUCHIYA ◽  
SEIJIRO ASO ◽  
YOSHIO IMADA ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Incomplete Kawasaki Disease ◽  
Coronary Artery Abnormality

scholarly journals Predictive value of brachial artery flow-mediated dilation on coronary artery abnormality in acute stage of Kawasaki disease

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Yizhou Wen ◽  
Xianmin Wang ◽  
Yonghong Guo ◽  
Mei Jin ◽  
Jimei Xi ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Brachial Artery ◽  
Characteristic Curve ◽  
Coronary Aneurysm ◽  
Acute Stage ◽  
Flow Mediated Dilation ◽  
Strong Negative Correlation ◽  
Coronary Artery Abnormality ◽  
Artery Flow

AbstractCoronary artery abnormalities (CAAs) are a severe complication of Kawasaki disease (KD) that may lead to cardiovascular events. Given the evidence that brachial artery flow-mediated dilation (FMD) decreases in children after the onset of KD, we hypothesized that it could be an early marker of CAA development in the acute stage and investigated its relationship with variation in the coronary artery diameter. A total of 326 sex- and age-matched children were enrolled, including 120 with KD, 109 febrile children and 97 healthy controls. In this study, FMD was significantly decreased in the KD group compared with the febrile and healthy groups. FMD was lower in the CAA group than in the no coronary artery abnormality group. The comparison of FMD showed an obvious difference among the CAA subgroups. The FMD in the coronary aneurysm (CA) group showed a strong negative correlation with the pretreatment maximum coronary artery Z-score (preZmax). While preZmax was 2.5, the receiver operating characteristic curve indicated an optimal cutoff point of 3.44% for FMD. FMD ≤ 3.44% could be considered as a signal of coronary lesions in acute stage of KD.

scholarly journals Combination Thrombolytic and Anti-Platelet Therapies in an Infant with Incomplete Kawasaki Disease and Coronary Aneurysms

2008 ◽  
Vol 13 (4) ◽  
pp. 242-250
Author(s):  
Peter N. Johnson ◽  
Robert J. Kuhn
Keyword(s):  
Septic Shock ◽  
Coronary Artery ◽  
Kawasaki Disease ◽  
Thrombus Formation ◽  
Coronary Aneurysm ◽  
Intravenous Immunoglobulins ◽  
Minimal Change ◽  
Incomplete Kawasaki Disease ◽  
Persistent Fever ◽  
Coronary Aneurysms

A 3-month-old infant was transferred to our facility with persistent fever and concerns for septic shock. A 2-D echocardiogram revealed multiple coronary aneurysms and axillary and coronary artery thrombi, and a diagnosis of incomplete Kawasaki disease (KD) was established. Aggressive therapies including intravenous immunoglobulins, enoxaparin, abciximab, aspirin, and alteplase were used to decrease the size of the coronary aneurysms and inhibit further thrombus formation. After minimal change in the size of coronary aneurysms and in thrombus formation, clopidogrel was added. Approximately 2 weeks after initiation of these therapies, a decrease in the coronary aneurysm size was noted with no signs of thrombus. This case documents successful use of thrombolytic and combination anti-platelet agents (i.e., clopidogrel, abciximab, and aspirin) in an infant with KD and cardiovascular sequelae.

Kawasaki Disease in a Patient With Williams Syndrome

2018 ◽  
Vol 11 (4) ◽  
pp. NP144-NP147
Author(s):  
Aleisha M. Nabower ◽  
Lois J. Starr ◽  
Jonathan Cramer
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Williams Syndrome ◽  
Coronary Arteries ◽  
Incomplete Kawasaki Disease ◽  
Cardiac Anomalies ◽  
Coronary Artery Dilatation ◽  
Coronary Aneurysms

Kawasaki disease can be difficult to diagnose in infants, putting them at higher risk for developing coronary artery dilatation. It can be even more difficult to diagnose in the setting of preexisting cardiac anomalies such as those found in Williams syndrome. We present a case of a three-month-old male with Williams syndrome with rapidly developing giant coronary aneurysms due to Kawasaki disease. This case demonstrates the importance of repeat echocardiography in diagnosing incomplete Kawasaki disease in infants. We speculate that elastin changes, as present in Williams syndrome, may put affected children at higher risk for development of giant coronary arteries should they acquire Kawasaki disease.

scholarly journals The relationship between catechol-O-methyltransferase gene polymorphism and coronary artery abnormality in Kawasaki disease

2009 ◽  
Vol 52 (1) ◽  
pp. 87 ◽  
Author(s):  
Hyo Jin Lee ◽  
Myung Sook Lee ◽  
Ji Sook Kim ◽  
Eun Ryoung Kim ◽  
Sung Wook Kang ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Gene Polymorphism ◽  
Coronary Artery Abnormality ◽  
Methyltransferase Gene ◽  
The Relationship

scholarly journals Giant coronary artery aneurysms in a 12-week-old infant with incomplete Kawasaki disease

2018 ◽  
pp. bcr-2018-224479 ◽  
Author(s):  
Lucy Guile ◽  
Simon Parke ◽  
Alison Kelly ◽  
Robert Tulloh
Keyword(s):  
Coronary Artery ◽  
Kawasaki Disease ◽  
Incomplete Kawasaki Disease ◽  
Coronary Artery Aneurysms
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