Annular lesions in a newborn

2018 ◽  
Vol 35 (6) ◽  
pp. 849-850
Author(s):  
Xia Yu ◽  
Chuang Gao ◽  
Zhouwei Wu
Keyword(s):  
Annular Lesions

Pathogenesis of annular lesions: Why a ring presentation?

2022 ◽  
Author(s):  
Melissa Hoffman ◽  
Michael Renzi ◽  
Warren R. Heymann
Keyword(s):  
Annular Lesions

scholarly journals Cutaneous annular lesions as the first sign of transformation of follicular lymphoma into diffuse large B-cell lymphoma

2015 ◽  
Vol 81 (5) ◽  
pp. 495 ◽  
Author(s):  
Irene Palacios-Álvarez ◽  
Concepción Román-Curto ◽  
AlejandroMartín García-Sancho ◽  
Ángel Santos-Briz ◽  
JuanCarlos Santos-Durán ◽  
...  
Keyword(s):  
Follicular Lymphoma ◽  
B Cell ◽  
Cell Lymphoma ◽  
B Cell Lymphoma ◽  
Large B Cell Lymphoma ◽  
Annular Lesions ◽  
Large B Cell

Visual Diagnosis: Annular Lesions in a 3-month-old Girl

2019 ◽  
Vol 40 (4) ◽  
pp. e14-e17
Author(s):  
Joseph S. Tadros ◽  
Yang Yu ◽  
Anne W. Lucky
Keyword(s):  
Annular Lesions ◽  
Visual Diagnosis

An Unusual Dermatitis with Annular Lesions

2008 ◽  
Vol 25 (2) ◽  
pp. 253-254
Author(s):  
Christina Cernik ◽  
Julian Trevino ◽  
Matthew Janik
Keyword(s):  
Annular Lesions

scholarly journals Hybrid Clinical and Histopathological Pattern in Annular Lesions: An Overlap between Annular Elastolytic Giant Cell Granuloma and Granuloma Annulare?

2012 ◽  
Vol 2012 ◽  
pp. 1-4
Author(s):  
Felipe Ladeira de Oliveira ◽  
Luisa Kelmer Côrtes de Barros Silveira ◽  
Alice de Miranda Machado ◽  
José Augusto da Costa Nery
Keyword(s):  
Diabetes Mellitus ◽  
Case Report ◽  
Giant Cell ◽  
Clinical Features ◽  
Skin Disease ◽  
Granuloma Annulare ◽  
Elastic Fibers ◽  
Giant Cell Granuloma ◽  
Endocrine Disease ◽  
Annular Lesions

Annular elastolytic giant cell granuloma (AEGCG) is a rare granulomatous skin disease of unclear pathogenesis which belongs to the group of disorders in the skin and elastic fibers with similar clinical features of granuloma annulare (GA). This case report is intended to describe a rare hybrid pattern in histopathology demonstrating coexistence of AEGCG and GA. An endocrine disease, such as diabetes mellitus (DM), could contribute to the coexistence of both lesions, and this possibility must be included in the medical investigation.

scholarly journals Atypical presentation of secondary syphilis: annular lesions in an elderly patient

2021 ◽  
Vol 63 ◽  
Author(s):  
Danielle Machado Pagani ◽  
Fernanda Brandão Pacheco ◽  
Natália Andressa Buss Venier ◽  
Isadora da Luz Silva ◽  
Giselda Kipper Richter ◽  
...  
Keyword(s):  
Elderly Patient ◽  
Secondary Syphilis ◽  
Atypical Presentation ◽  
Annular Lesions

scholarly journals Annular lesions located on the right forearm

2014 ◽  
Vol 59 (6) ◽  
pp. 636
Author(s):  
Vera Tengattini ◽  
Cosimo Misciali ◽  
Annalisa Patrizi ◽  
Federico Bardazzi ◽  
Alessandro Pileri
Keyword(s):  
Annular Lesions ◽  
The Right

scholarly journals Spatiotemporal Expression of 3-B-3(−) and 7-D-4 Chondroitin Sulfation, Tissue Remodeling, and Attempted Repair in an Ovine Model of Intervertebral Disc Degeneration

Cartilage ◽  
2019 ◽  
Vol 11 (2) ◽  
pp. 234-250 ◽  
Author(s):  
Brooke Farrugia ◽  
Susan M. Smith ◽  
Cindy C. Shu ◽  
James Melrose
Keyword(s):  
Intervertebral Disc ◽  
Disc Degeneration ◽  
Annulus Fibrosus ◽  
Intervertebral Disc Degeneration ◽  
Tissue Sections ◽  
Sulfated Glycosaminoglycan ◽  
Spatiotemporal Expression ◽  
Chondroid Metaplasia ◽  
Annular Lesions ◽  
Spontaneous Repair

Objective Examination of intervertebral disc (IVD) regeneration in an ovine annular lesion model. Hypothesis Sulfation motifs are important functional determinants in glycosaminoglycans (GAGs). Previous studies have correlated 3-B-3(−) and 7-D-4 chondroitin sulfate (CS) motifs in tissues undergoing morphogenetic transition in development. We hypothesize that these motifs may also be expressed in degenerate IVDs and may represent a reparative response. Design Induction of disc degeneration by 5 mm or 6 × 20 mm lesions in the annulus fibrosus (AF) over 6 or 3 to 6 months postoperation (PO). Tissue sections were stained with toluidine blue–fast green, 3-B-3(−) and 7-D-4 CS-sulfation motifs were immunolocalized in 3-month PO 6 × 20 mm lesion IVDs. Sulfated glycosaminoglycan (GAG), 3-B-3(−), and 7-D-4 epitopes were quantitated by ELISIA (enzyme-linked immunosorbent inhibition assay) in extracts of AF (lesion site and contralateral half) and nucleus pulposus (NP) 0, 3, and 6 months PO. Results Collagenous overgrowth of lesions occurred in the outer AF. Chondroid metaplasia in ~20% of the 6 × 20 mm affected discs resulted in integration of an outgrowth of NP tissue with the inner AF lamellae preventing propagation of the lesion. 3-B-3(−) and 7-D-4 CS sulfation motifs were immunolocalized in this chondroid tissue. ELISIA quantified CS sulfation motifs demonstrating an increase 3 to 6 months PO in the AF lesion and a reduction in sulfated GAG not evident in the contralateral AF. Conclusions (1) Outer annular lesions underwent spontaneous repair. (2) Chondroid metaplasia of the inner 6 × 20 mm defect prevented its propagation suggesting an apparent reparative response.

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