Eating behaviour in contrasting adiposity phenotypes: Monogenic obesity and congenital generalized lipodystrophy

2020 ◽  
Vol 22 (1) ◽  
Author(s):  
José L. Santos ◽  
Víctor A. Cortés
Keyword(s):  
Eating Behaviour ◽  
Monogenic Obesity ◽  
Congenital Generalized Lipodystrophy

To intend or to be willing? The prediction of unhealthy eating behaviour amongst adolescents

2008 ◽  
Author(s):  
J. H. Gerrits ◽  
J. B. F. De Wit ◽  
R. G. Kuijer ◽  
D. T. D. De Ridder
Keyword(s):  
Eating Behaviour ◽  
Unhealthy Eating

Why A Doc With a Dip Doc?

2011 ◽  
Vol 1 (1) ◽  
pp. 30-34
Author(s):  
Catherine Morley
Keyword(s):  
Nutrition Education ◽  
Visual Arts ◽  
Documentary Film ◽  
Eating Behaviour ◽  
Knowledge And Skills ◽  
Time Out ◽  
Power Differentials ◽  
Research Findings

In 2007, when I began studies toward two diplomas, one in textile arts, and one in documentary film this seeming ‘change of focus’ prompted questions from dietetics and research colleagues: Was I changing careers? What did visual arts and film have to do with dietetics and research? In addition to personal reasons for these studies, I wanted ‘time out’ from consulting and research to develop my knowledge and skills in these artforms, and to explore them as means to broaden the reach of research findings. In this article, I discuss the potential for film and visual arts in dietetics practice and education. Arts-based inquiry and practice offer ways to disrupt power differentials, to question what counts as knowledge and whose/what voices ought to count, to invite reflections on and conversations about meanings imbedded in food and in eating behaviour, and to integrate this knowledge into collaborative, client-centred approaches to nutrition education.

STUDIES IN CONGENITAL GENERALIZED LIPODYSTROPHY

1973 ◽  
Vol 72 (3) ◽  
pp. 495-505 ◽  
Author(s):  
Oddmund Søvik ◽  
Svein Oseid
Keyword(s):  
Biological Activity ◽  
Insulin Response ◽  
Epididymal Adipose Tissue ◽  
Large Individual ◽  
Immunoreactive Insulin ◽  
List Type ◽  
Glucose Load ◽  
Congenital Generalized Lipodystrophy ◽  
The One

ABSTRACT The biological activity of plasma insulin from 4 cases of congenital generalized lipodystrophy has been studied, using rat diaphragm and epididymal adipose tissue in vivo. The results are compared with previous data on plasma immunoreactive insulin obtained in these patients. 2 of the 4 cases exhibited unusually high biological insulin activities during the fasting state as well as after an intravenous (iv) glucose load. In the fat pad assay activities as high as 10 000 μU insulin per ml were observed. During childhood the biological insulin activities were generally high, although there were large individual variations. However, in the one case studied after the age of puberty, the insulin response to a glucose load was negligible. Taken together, the biological and immunological activities observed strongly suggest the presence of pancreatic insulin in these patients. It appears that the circulating insulin has a fully biological activity. The decreasing insulin activities after cessation of growth are in agreement with the appearance of frank diabetes at this time.

STUDIES IN CONGENITAL GENERALIZED LIPODYSTROPHY (SEIP-BERARDINELLI SYNDROME)

1973 ◽  
Vol 72 (3) ◽  
pp. 475-494 ◽  
Author(s):  
Svein Oseid
Keyword(s):  
Insulin Resistance ◽  
Glucose Tolerance ◽  
Normal Glucose Tolerance ◽  
Juvenile Form ◽  
Congenital Generalized Lipodystrophy ◽  
Glucose Levels ◽  
Normal Glucose ◽  
Glucose Tolerance Tests ◽  
The One ◽  
Adipose Organ

ABSTRACT Six cases of congenital generalized lipodystrophy have been studied at different ages from infancy to adolescence with regard to glucose tolerance, insulin secretion, and insulin sensitivity. During the first few years of life there is normal glucose tolerance. The fasting immuno-reactive insulin (IRI) levels are either slightly elevated or normal. The IRI response to glucose is exaggerated and prolonged, at least from the third year of life. Some degree of insulin resistance is already present in infancy. From the age of 8–10 years glucose tolerance decreases rapidly. The fasting IRI levels are usually grossly elevated, while fasting plasma glucose levels are only moderately elevated or normal. The IRI responses to oral and iv administered glucose, and to tolbutamide are exaggerated; the insulinogenic indices are high. Cortisone primed glucose tolerance tests become abnormal. Insulin resistance is marked, and increases with age. After cessation of growth at approximately 12 years of age, frank diabetes with fasting hyperglycaemia and diabetic glucose tolerance curves developed in the one patient followed beyond this age. Her fasting IRI was increased, but there was a poor IRI response to glucose stimulation, suggesting a partial exhaustion of the β-cells. Her initial IRI response to tolbutamide was still good, but not as brisk as in the younger patients. This type of diabetes is quite different from the juvenile form, and also from the diabetes of older age. It may be causally related to the lack of an adequate adipose organ necessary for the disposal of excesses of glucose, or possibly related to another anti-insulin mechanism.

Eating behaviour disorders in infants

2016 ◽  
Vol 77 (5) ◽  
pp. 139-141
Author(s):  
S.N. Zinchenko ◽  
◽  
V.G. Kozachuk ◽  
O.A. Maystruk ◽  
◽  
...  
Keyword(s):  
Eating Behaviour

Ophthalmologic Findings in Congenital Generalized Lipodystrophy—A Possible Marker of Metabolic Disorders

Diabetes ◽  
2018 ◽  
Vol 67 (Supplement 1) ◽  
pp. 628-P
Author(s):  
VIRGINIA O. FERNANDES ◽  
LORENA M.A. GOMES ◽  
ANA PAULA D.R. MONTENEGRO ◽  
CLARISSE M.M. PONTE ◽  
LIVIA A.A. BATISTA ◽  
...  
Keyword(s):  
Metabolic Disorders ◽  
Congenital Generalized Lipodystrophy

Sleep Disorders in Patients with Congenital Generalized Lipodystrophy

Diabetes ◽  
2018 ◽  
Vol 67 (Supplement 1) ◽  
pp. 2192-PUB
Author(s):  
VIRGINIA O. FERNANDES ◽  
CARLA P. SILVA ◽  
ANA PAULA D.R. MONTENEGRO ◽  
CLARISSE M.M. PONTE ◽  
LIVIA A.A. BATISTA ◽  
...  
Keyword(s):  
Sleep Disorders ◽  
Congenital Generalized Lipodystrophy
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