scholarly journals Zic2 is required for enteric nervous system development and neurite outgrowth: a mouse model of enteric hyperplasia and dysplasia

2013 ◽  
Vol 25 (6) ◽  
pp. 538-541 ◽  
Author(s):  
Y. Zhang ◽  
L. Niswander
Keyword(s):  
Nervous System ◽  
Mouse Model ◽  
Enteric Nervous System ◽  
Neurite Outgrowth ◽  
System Development ◽  
Nervous System Development ◽  
Enteric Nervous System Development

scholarly journals Changes in Nicotinic Neurotransmission during Enteric Nervous System Development

2015 ◽  
Vol 35 (18) ◽  
pp. 7106-7115 ◽  
Author(s):  
J. P. P. Foong ◽  
C. S. Hirst ◽  
M. M. Hao ◽  
S. J. McKeown ◽  
W. Boesmans ◽  
...  
Keyword(s):  
Nervous System ◽  
Enteric Nervous System ◽  
System Development ◽  
Nervous System Development ◽  
Enteric Nervous System Development

scholarly journals Testing the Ret and Sema3d genetic interaction in mouse enteric nervous system development

2017 ◽  
Vol 26 (10) ◽  
pp. 1811-1820 ◽  
Author(s):  
Ashish Kapoor ◽  
Dallas R. Auer ◽  
Dongwon Lee ◽  
Sumantra Chatterjee ◽  
Aravinda Chakravarti
Keyword(s):  
Nervous System ◽  
Enteric Nervous System ◽  
Genetic Interaction ◽  
System Development ◽  
Nervous System Development ◽  
Enteric Nervous System Development

Enteric Nervous System: Development and Developmental Disturbances—Part 2

2002 ◽  
Vol 5 (4) ◽  
pp. 329-349 ◽  
Author(s):  
Donald Newgreen ◽  
Heather M. Young
Keyword(s):  
Nervous System ◽  
Enteric Nervous System ◽  
System Development ◽  
Molecular Genetic ◽  
Nervous System Development ◽  
Developmental Disturbances ◽  
Genetic Abnormalities ◽  
A Cell ◽  
Cells Of Cajal ◽  
Enteric Nervous System Development

This review, which is presented in two parts, summarizes and synthesizes current views on the genetic, molecular, and cell biological underpinnings of the early embryonic phases of enteric nervous system (ENS) formation and its defects. Accurate descriptions of the phenotype of ENS dysplasias, and knowledge of genes which, when mutated, give rise to the disorders (see Part 1 in the previous issue of this journal), are not sufficient to give a real understanding of how these abnormalities arise. The often indirect link between genotype and phenotype must be sought in the early embryonic development of the ENS. Therefore, in this, the second part, we provide a description of the development of the ENS, concentrating mainly on the origin of the ENS precursor cells and on the cell migration by which they become distributed throughout the gastrointestinal tract. This section also includes experimental evidence on the controls of ENS formation derived from classic embryological, cell culture, and molecular genetic approaches. In addition, for reasons of completeness, we also briefly describe the origins of the interstitial cells of Cajal, a cell population closely related anatomically and functionally to the ENS. Finally, a brief sketch is presented of current notions on the developmental processes between the genes and the morphogenesis of the ENS, and of the means by which the known genetic abnormalities might result in the ENS phenotype observed in Hirschsprung's disease.

Inhibition of cell death results in hyperganglionosis: implications for enteric nervous system development

2009 ◽  
Vol 21 (7) ◽  
pp. 768-e49 ◽  
Author(s):  
a. s. wallace ◽  
a. j. barlow ◽  
l. navaratne ◽  
j-m. delalande ◽  
s. tauszig-delamasure ◽  
...  
Keyword(s):  
Cell Death ◽  
Nervous System ◽  
Enteric Nervous System ◽  
System Development ◽  
Nervous System Development ◽  
Enteric Nervous System Development
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