Malignant progression of an extraventricular neurocytoma arising from the VIIIth cranial nerve: A case report and literature review

2018 ◽  
Vol 39 (2) ◽  
pp. 120-126 ◽  
Author(s):  
Yasuo Sugita ◽  
Takuya Furuta ◽  
Satoru Komaki ◽  
Koichi Ohshima ◽  
Kiyohiko Sakata ◽  
...  
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Cranial Nerve ◽  
Malignant Progression ◽  
Extraventricular Neurocytoma ◽  
Viiith Cranial Nerve

Meckel Cave Epidermoid Cyst Presenting as Multiple Cranial Nerve Deficits Due to Indirect Tumoral Compression of the Cavernous Sinus: A Case Report and Literature Review

2019 ◽  
Vol 121 ◽  
pp. 88-94 ◽  
Author(s):  
Christopher M. Busch ◽  
Joshua T. Prickett ◽  
Rachel Stein ◽  
Joshua A. Cuoco ◽  
Eric A. Marvin ◽  
...  
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Cavernous Sinus ◽  
Epidermoid Cyst ◽  
Cranial Nerve ◽  
Multiple Cranial Nerve

scholarly journals A Case Report of Classical eight-and-a-Half Syndrome and Etiology Analysis

2021 ◽  
Vol 8 (7) ◽  
pp. 01-03
Author(s):  
Duo Fan
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Facial Nerve ◽  
Cranial Nerve ◽  
Nerve Palsy ◽  
Vascular Pathology ◽  
Medial Longitudinal Fasciculus ◽  
Pontine Reticular Formation ◽  
And Control ◽  
Perforator Arteries

Eight-and-a-half syndrome is one-and-a-half syndrome plus ipsilateral seventh (facial) cranial nerve palsy, which was first reported by Eggenberger in 1998. This syndrome is caused by a lesion (most often vascular or demyelinating) in the dorsal tegmentum of the caudal pons. It involves the para pontine reticular formation and the medial longitudinal fasciculus, as well as the nucleus and the fasciculus of the facial nerve. We describe a case of a 56-year-old man presented with eight-and-a-half syndrome. We also discuss the vascular pathology and etiology of this syndrome through the literature review. Most eight-and-a-half syndrome cases are caused by occlusions of the perforating branches of the pons. The pathology can be transparent lipid-like changes at the perforator arteries. It can also be thrombosis at the orifice of the main or perforator arteries. BAH could be a very important factor in perforator artery infarction at the pontine. The etiology is closely related to hyperglycemia. An effective intervention is to avoid bad habits and control the hyperglycemia.

scholarly journals P03.16 Intracranial extraventricular neurocytoma: case report and literature review

2017 ◽  
Vol 19 (suppl_3) ◽  
pp. iii37-iii37
Author(s):  
J. Monteiro ◽  
C. Sousa ◽  
R. Manilha ◽  
D. Gonçalves ◽  
C. Viegas ◽  
...  
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Extraventricular Neurocytoma

Neurocytoma of the VIIIth Cranial Nerve: Case Report

Neurosurgery ◽  
2005 ◽  
Vol 57 (5) ◽  
pp. E1065-E1065 ◽  
Author(s):  
Onder Onguru ◽  
Özlem Kurtkaya-Yapıcıer ◽  
Bernd W. Scheithauer ◽  
Patrick Luetmer ◽  
Peter C. Burger ◽  
...  
Keyword(s):  
Case Report ◽  
Cranial Nerve ◽  
Viiith Cranial Nerve

Extraventricular Neurocytoma in the Left Frontal Lobe: A Case Report and Literature Review

2018 ◽  
Vol 112 ◽  
pp. 178-181 ◽  
Author(s):  
Fan Chen ◽  
Rihua Jin ◽  
Xinmin Wu ◽  
Zengping Dong ◽  
Dawei Chen
Keyword(s):  
Case Report ◽  
Literature Review ◽  
Frontal Lobe ◽  
Left Frontal Lobe ◽  
Extraventricular Neurocytoma
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