scholarly journals Rare valiant vertical one-and-a-half syndrome without ipsilateral upward gaze palsy in a patient with thalamomesencephalic stroke

2018 ◽  
Vol 6 (5) ◽  
pp. 133-135 ◽  
Author(s):  
Kota Sato ◽  
Yoshiaki Takahashi ◽  
Namiko Matsumoto ◽  
Taijun Yunoki ◽  
Mami Takemoto ◽  
...  
Keyword(s):  
Upward Gaze Palsy ◽  
Gaze Palsy

Acute upward gaze palsy: Not always Parinaud syndrome

2019 ◽  
Vol 30 (6) ◽  
pp. NP5-NP6
Author(s):  
Salvatore Rossi ◽  
Giovanni Frisullo ◽  
Raffaele Iorio
Keyword(s):  
Brain Magnetic Resonance Imaging ◽  
Acute Onset ◽  
Thalamic Lesion ◽  
Ischemic Lesion ◽  
Thalamic Infarction ◽  
The Central Nervous System ◽  
Midbrain Infarction ◽  
Upward Gaze Palsy ◽  
The Right ◽  
Gaze Palsy

Introduction: Parinaud syndrome, caused by midbrain infarction, usually manifests as an ocular conjugate upgaze palsy. However, this sign should not point out straightforwardly to Parinaud syndrome, as other lesions in the central nervous system could cause it. Case description: The case of a 47-year-old woman showing acute onset of diplopia with bilateral upward gaze palsy is described. Parinaud syndrome was suspected on clinical grounds. However, brain magnetic resonance imaging displayed an acute ischemic lesion in the right anteromedial thalamus. Conclusions: Bilateral upward gaze palsy may be caused by unilateral thalamic infarction. The mechanism by which a unilateral thalamic lesion causes bilateral gaze palsy is discussed.

Upward-gaze palsy due to shunt malfunction

Neurosurgery ◽  
1989 ◽  
Vol 24 (1) ◽  
pp. 141
Author(s):  
A C Iplikcio??lu ◽  
F Ozer ◽  
A Erbengi
Keyword(s):  
Shunt Malfunction ◽  
Upward Gaze Palsy ◽  
Gaze Palsy

Sylvian aqueduct syndrome and global rostral midbrain dysfunction associated with shunt malfunction

1999 ◽  
Vol 90 (2) ◽  
pp. 227-236 ◽  
Author(s):  
Giuseppe Cinalli ◽  
Christian Sainte-Rose ◽  
Isabelle Simon ◽  
Guillaume Lot ◽  
Spiros Sgouros
Keyword(s):  
Pressure Gradient ◽  
Obstructive Hydrocephalus ◽  
Shunt Malfunction ◽  
Shunt Revision ◽  
Third Ventriculostomy ◽  
Content Type ◽  
Upward Gaze Palsy ◽  
Ocular Signs ◽  
Gaze Palsy ◽  
Fine Print

Object. This study is a retrospective analysis of clinical data obtained in 28 patients affected by obstructive hydrocephalus who presented with signs of midbrain dysfunction during episodes of shunt malfunction.Methods. All patients presented with an upward gaze palsy, sometimes associated with other signs of oculomotor dysfunction. In seven cases the ocular signs remained isolated and resolved rapidly after shunt revision. In 21 cases the ocular signs were variably associated with other clinical manifestations such as pyramidal and extrapyramidal deficits, memory disturbances, mutism, or alterations in consciousness. Resolution of these symptoms after shunt revision was usually slow. In four cases a transient paradoxical aggravation was observed at the time of shunt revision. In 11 cases ventriculocisternostomy allowed resolution of the symptoms and withdrawal of the shunt.Simultaneous supratentorial and infratentorial intracranial pressure recordings performed in seven of the patients showed a pressure gradient between the supratentorial and infratentorial compartments, with a higher supratentorial pressure before shunt revision. Inversion of this pressure gradient was observed after shunt revision and resolution of the gradient was observed in one case after third ventriculostomy. In six recent cases, a focal midbrain hyperintensity was evidenced on T2-weighted magnetic resonance imaging sequences at the time of shunt malfunction. This rapidly resolved after the patient underwent third ventriculostomy.Conclusions. It is probable that in obstructive hydrocephalus, at the time of shunt malfunction, the development of a transtentorial pressure gradient could initially induce a functional impairment of the upper midbrain, inducing upward gaze palsy. The persistence of the gradient could lead to a global dysfunction of the upper midbrain. Third ventriculostomy contributes to equalization of cerebrospinal fluid pressure across the tentorium by restoring free communication between the infratentorial and supratentorial compartments, resulting in resolution of the patient's clinical symptoms.

Upward Gaze Palsy: a Valuable Sign to Distinguish Spinocerebellar Ataxias

2020 ◽  
Vol 19 (5) ◽  
pp. 685-690
Author(s):  
Gustavo L. Franklin ◽  
Alex T. Meira ◽  
Carlos H. F. Camargo ◽  
Fábio A. Nascimento ◽  
Hélio A. G. Teive
Keyword(s):  
Spinocerebellar Ataxias ◽  
Upward Gaze Palsy ◽  
Gaze Palsy

scholarly journals Sylvian aqueduct syndrome and global rostral midbrain dysfunction associated with shunt malfunction

1998 ◽  
Vol 5 (6) ◽  
pp. E2
Author(s):  
Giuseppe Cinalli ◽  
Christian Sainte-Rose ◽  
Isabelle Simon ◽  
Guillaume Lot ◽  
Spiros Sgouros
Keyword(s):  
Pressure Gradient ◽  
Obstructive Hydrocephalus ◽  
Fluid Pressure ◽  
Clinical Manifestations ◽  
Shunt Malfunction ◽  
Shunt Revision ◽  
Third Ventriculostomy ◽  
Upward Gaze Palsy ◽  
Ocular Signs ◽  
Gaze Palsy

Object This study is a retrospective analysis of clinical data obtained in 28 patients affected by obstructive hydrocephalus who presented with signs of midbrain dysfunction during episodes of shunt malfunction. Methods All patients presented with an upward gaze palsy, sometimes associated with other signs of oculomotor dysfunction. In seven cases the ocular signs remained isolated and resolved rapidly after shunt revision. In 21 cases the ocular signs were variably associated with other clinical manifestations such as pyramidal and extrapyramidal deficits, memory disturbances, mutism, or alterations in consciousness. Resolution of these symptoms after shunt revision was usually slow. In four cases a transient paradoxical aggravation was observed at the time of shunt revision. In 11 cases ventriculocisternostomy allowed resolution of the symptoms and withdrawal of the shunt. Simultaneous supratentorial and infratentorial intracranial pressure recordings performed in seven of the patients showed a pressure gradient between the supratentorial and infratentorial compartments with a higher supratentorial pressure before shunt revision. Inversion of this pressure gradient was observed after shunt revision and resolution of the gradient was observed in one case after third ventriculostomy. In six recent cases, a focal midbrain hyperintensity was evidenced on T2-weighted magnetic resonance imaging sequences at the time of shunt malfunction. This rapidly resolved after the patient underwent third ventriculostomy. It is probable that in obstructive hydrocephalus at the time of shunt malfunction, the development of a transtentorial pressure gradient could initially induce a functional impairment of the upper midbrain, inducing upward gaze palsy. The persistence of the gradient could lead to a global dysfunction of the upper midbrain. Conclusions Third ventriculostomy contributes to equalization of cerebrospinal fluid pressure across the tentorium by restoring free communication between the infratentorial and supratentorial compartments, resulting in resolution of the patient's clinical symptoms.

scholarly journals A case of primary intraventricular hemorrhage associated with upward gaze palsy.

Nosotchu ◽  
1992 ◽  
Vol 14 (6) ◽  
pp. 661-665
Author(s):  
Yuji Yano ◽  
Kentaro Takano ◽  
Setsuro Ibayashi ◽  
Seizo Sadoshima ◽  
Masatoshi Fujishima
Keyword(s):  
Intraventricular Hemorrhage ◽  
Upward Gaze Palsy ◽  
Gaze Palsy

scholarly journals Teaching Video NeuroImages: Upward gaze palsy is a sign of spinocerebellar ataxia type 3

Neurology ◽  
2018 ◽  
Vol 91 (5) ◽  
pp. e494-e494 ◽  
Author(s):  
Fábio A. Nascimento ◽  
Bruno Carniatto Marques Garcia ◽  
Helio A.G. Teive
Keyword(s):  
Spinocerebellar Ataxia ◽  
Spinocerebellar Ataxia Type ◽  
Spinocerebellar Ataxia Type 3 ◽  
Upward Gaze Palsy ◽  
Type 3 ◽  
Teaching Video ◽  
Gaze Palsy
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