scholarly journals Influence of salinity on survival, growth, hemolymph osmolality, gill sodium potassium ATPase activity, and sodium potassium chloride co‐transporter expression in the redclaw crayfish Cherax quadricarinatus

2020 ◽  
Author(s):  
Reem Rida ◽  
Razan Zein‐Eddine ◽  
Sawsan Kreydiyyeh ◽  
Antonio Garza de Yta ◽  
Imad Patrick Saoud
Keyword(s):  
Atpase Activity ◽  
Potassium Chloride ◽  
Sodium Potassium ◽  
Cherax Quadricarinatus ◽  
Potassium Atpase ◽  
Transporter Expression ◽  
Redclaw Crayfish

Adenosine Receptor Modulation of Hypoxic-ischemic Injury in Striatum of Newborn Piglets

2020 ◽  
Vol 17 (4) ◽  
pp. 510-517
Author(s):  
Santiago Ortega-Gutierrez ◽  
Brandy Jones ◽  
Alan Mendez-Ruiz ◽  
Pankhil Shah ◽  
Michel T. Torbey
Keyword(s):  
Oxidative Stress ◽  
Atpase Activity ◽  
Neuronal Injury ◽  
Receptor Activation ◽  
Adult Mortality ◽  
Vehicle Group ◽  
Sodium Potassium ◽  
Receptor Modulation ◽  
A2a Receptor ◽  
Potassium Atpase

Background: Hypoxic-ischemic encephalopathy (HIE) is a major cause of pediatric and adult mortality and morbidity. Unfortunately, to date, no effective treatment has been identified. In the striatum, neuronal injury is analogous to the cellular mechanism of necrosis observed during NMethyl- D-Aspartate (NMDA) excitotoxicity. Adenosine acts as a neuromodulator in the central nervous system, the role of which relies mostly on controlling excitatory glutamatergic synapses. Objective: To examine the effect of pretreatment of SCH58261, an adenosine 2A (A2A) receptor antagonist and modulator of NMDA receptor function, following hypoxic-ischemia (HI) on sodium- potassium ATPase (Na+, K+-ATPase) activity and oxidative stress. Methods: Piglets (4-7 days old) were subjected to 30 min hypoxia and 7 min of airway occlusion producing asphyxic cardiac arrest. Groups were divided into four categories: HI samples were divided into HI-vehicle group (n = 5) and HI-A2A group (n = 5). Sham controls were divided into Sham vehicle (n = 5) and Sham A2A (n = 5) groups. Vehicle groups were pretreated with 0.9% saline, whereas A2A animals were pretreated with SCH58261 10 min prior to intervention. Striatum samples were collected 3 h post-arrest. Sodium-potassium ATPase (Na+, K+-ATPase) activity, malondialdehyde (MDA) + 4-hydroxyalkenals (4-HDA) and glutathione (GSH) levels were compared. Results: Pretreatment with SCH58261 significantly attenuated the decrease in Na+, K+-ATPase, decreased MDA+4-HDA levels and increased GSH in the HI-A2A group when compared to HIvehicle. Conclusion: A2A receptor activation may contribute to neuronal injury in newborn striatum after HI in association with decreased Na+, K+-ATPase activity and increased oxidative stress.

Sodium Potassium ATPase Activity in Human Rectal Mucosa With and Without Renal Insufficiency

1985 ◽  
Vol 5 (3) ◽  
pp. 177-181 ◽  
Author(s):  
Ronald J. Hené ◽  
Peter Boer ◽  
Hein A. Koomans ◽  
Evert J. Dorhout Mees
Keyword(s):  
Renal Insufficiency ◽  
Atpase Activity ◽  
Rectal Mucosa ◽  
Sodium Potassium ◽  
Potassium Atpase

scholarly journals Impaired rodent vagal nerve sodium-potassium ATPase (Na+-K+-ATPase) activity in streptozotocin diabetes

1990 ◽  
Vol 99 (4) ◽  
pp. 1223 ◽  
Author(s):  
T.V. Nowak ◽  
C. Castelaz ◽  
K. Ramaswamy ◽  
J.P. Weisbruch
Keyword(s):  
Atpase Activity ◽  
Streptozotocin Diabetes ◽  
Vagal Nerve ◽  
Sodium Potassium ◽  
Potassium Atpase
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