Motor nerve excitability after childhood Guillain-Barré syndrome

2017 ◽  
Vol 22 (2) ◽  
pp. 100-105 ◽  
Author(s):  
Judith Drenthen ◽  
Joyce Roodbol ◽  
Ellen M. Maathuis ◽  
Coriene E. Catsman-Berrevoets ◽  
Joleen H. Blok ◽  
...  
Keyword(s):  
Motor Nerve ◽  
Guillain Barre Syndrome ◽  
Nerve Excitability ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

scholarly journals Changes in motor nerve excitability in acute phase Guillain‐Barré syndrome

2021 ◽  
Author(s):  
Judith Drenthen ◽  
Badrul Islam ◽  
Zhahirul Islam ◽  
Quazi D. Mohammad ◽  
Ellen M. Maathuis ◽  
...  
Keyword(s):  
Acute Phase ◽  
Motor Nerve ◽  
Guillain Barre Syndrome ◽  
Nerve Excitability ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

Muscle MRI in severe Guillain–Barré syndrome with motor nerve inexcitability

2013 ◽  
Vol 260 (6) ◽  
pp. 1624-1630 ◽  
Author(s):  
María J. Sedano ◽  
Ana Canga ◽  
Carmen Pablos ◽  
José M. Polo ◽  
José Berciano
Keyword(s):  
Motor Nerve ◽  
Guillain Barre Syndrome ◽  
Muscle Mri ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

scholarly journals Guillain –Barrè Syndrome following Hepatitis E

2014 ◽  
Vol 2 (1) ◽  
pp. 48-49
Author(s):  
Poly Sengupta ◽  
Rama Biswas ◽  
Hasan Shahrear Ahmed ◽  
Kaniz Fatema
Keyword(s):  
Motor Nerve ◽  
Electrophysiological Study ◽  
Hepatitis E ◽  
Immunoglobulin M ◽  
Guillain Barre Syndrome ◽  
Limb Weakness ◽  
Laboratory Findings ◽  
Guillain Barré Syndrome ◽  
Guillain Barré ◽  
Upper Abdominal

Guillain- Barrè Syndrome is characterized by acute progressive symmetric limb weakness and areflexia. A 32 year old female presented with progressive ascending areflexic muscular weakness and bilateral lower motor neuron type of facial palsy. She had anorexia, nausea and upper abdominal pain for 2 weeks. The findings of motor nerve conduction study are consistent with acute inflammatory demyelinating polyradiculoneuropathy. She had elevated liver enzyme and positive immunoglobulin M antibody against hepatitis E in blood. Based on clinical features, laboratory findings and electrophysiological study, she was diagnosed as Guillain- Barrè Syndrome following hepatitis E. She was treated with intravenous immunoglobulin and recovered fully. DOI: http://dx.doi.org/10.3329/bccj.v2i1.19973 Bangladesh Crit Care J March 2014; 2 (1): 48-49

scholarly journals Neuropathophysiological potential of Guillain-Barré syndrome anti-ganglioside-complex antibodies at mouse motor nerve terminals

2011 ◽  
Vol 2 (3) ◽  
pp. 59-67 ◽  
Author(s):  
Femke M. P. Zitman ◽  
Kay N. Greenshields ◽  
Mark L. Kuijf ◽  
Masami Ueda ◽  
Ken-ichi Kaida ◽  
...  
Keyword(s):  
Motor Nerve ◽  
Guillain Barre Syndrome ◽  
Nerve Terminals ◽  
Motor Nerve Terminals ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

Motor nerve biopsy in severe Guillain-Barré syndrome

1992 ◽  
Vol 31 (4) ◽  
pp. 441-444 ◽  
Author(s):  
Susan M. Hall ◽  
Richard A. C. Hughes ◽  
Penny F. Atkinson ◽  
Ian McColl ◽  
Andrew Gale
Keyword(s):  
Motor Nerve ◽  
Nerve Biopsy ◽  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

scholarly journals Severe Guillain-Barré syndrome after surgery for multiple fractures: a rare case report with a 5-year follow-up and a brief review of the literature

2021 ◽  
Vol 22 (1) ◽  
Author(s):  
Jian Chen ◽  
Jian-xiong Ma ◽  
Cai-hong Zuo ◽  
Qing Zhang ◽  
Heng-ting Chen ◽  
...  
Keyword(s):  
Cerebrospinal Fluid ◽  
Orthopaedic Surgery ◽  
Motor Nerve ◽  
Rare Complication ◽  
Lower Limbs ◽  
Guillain Barre Syndrome ◽  
Multiple Fractures ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

Abstract Background Guillain-Barré syndrome (GBS) is the most common and serious acute paralytic neuropathy and is usually caused by infection. It is thought to be the result of an aberrant response of the immune system. To our knowledge, GBS, especially severe GBS, after orthopaedic surgery has rarely been reported. Case presentation We herein report the case of a 58-year-old man who developed quadriplegia and respiratory failure on the 6th day after surgery for multiple fractures. The patient had no symptoms of respiratory or gastrointestinal tract infection within 4 weeks before the onset. The white blood cell count was normal, and there was no redness, swelling, heat or pain in the surgical incision. Brain, cervical and thoracic magnetic resonance imaging were normal, albuminocytological dissociation was found on cerebrospinal fluid examination, and electrophysiological examination showed that sensory and motor nerve evoked potentials could not be elicited. A diagnosis of post-traumatic GBS was made, and the patient was treated with intravenous immunoglobulin and plasma exchange, as well as supportive care and rehabilitation exercise. The length of stay was 18 months, and the in-hospital-related costs amounted to $127,171. At the last follow-up, the patient had recovered only grade 3 power in the upper limbs and grade 2 power in the lower limbs. Conclusions Severe GBS is a rare complication after orthopaedic surgery. When progressive weakness occurs in trauma patients, the possibility of GBS should be considered, and cerebrospinal fluid and electrophysiological examinations should be performed in a timely manner. For patients with severe GBS after trauma, the treatment costs may be high, and the prognosis may be poor.

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