scholarly journals APOBEC3C, a nucleolar protein induced by genotoxins, is excluded from DNA damage sites

FEBS Journal ◽  
2021 ◽  
Author(s):  
Daniel Constantin ◽  
Gilles Dubuis ◽  
María del Carmen Conde‐Rubio ◽  
Christian Widmann
Keyword(s):  
Dna Damage ◽  
Nucleolar Protein

scholarly journals CANu1, a novel nucleolar protein, accumulated on centromere in response to DNA damage

2008 ◽  
Vol 13 (8) ◽  
pp. 787-796 ◽  
Author(s):  
Choong-Ryoul Sihn ◽  
Yeon-Su Lee ◽  
Jin-Sook Jeong ◽  
Kyunghee Park ◽  
Sang Hoon Kim
Keyword(s):  
Dna Damage ◽  
Nucleolar Protein

scholarly journals Nucleolar protein nucleolin functions in replication stress–induced DNA damage responses

2019 ◽  
Vol 60 (3) ◽  
pp. 281-288 ◽  
Author(s):  
Kasumi Kawamura ◽  
Fei Qi ◽  
Qingmei Meng ◽  
Ikue Hayashi ◽  
Junya Kobayashi
Keyword(s):  
Dna Damage ◽  
Replication Stress ◽  
Nucleolar Protein ◽  
Dna Damage Responses

1576: Prediction of Spermatozoal DNA Damage by a Novel Semen Quality Score

2004 ◽  
Vol 171 (4S) ◽  
pp. 416-416
Author(s):  
Tamer M. Said ◽  
Shyam Allamaneni ◽  
Kiran P. Nallella ◽  
Rakesh K. Sharma ◽  
Sijo J. Parekattil ◽  
...  
Keyword(s):  
Dna Damage ◽  
Semen Quality ◽  
Quality Score

A safe fix for alcohol-derived DNA damage

Nature ◽  
2020 ◽  
Vol 579 (7800) ◽  
pp. 499-500
Author(s):  
Irene Gallina ◽  
Julien P. Duxin
Keyword(s):  
Dna Damage

Use of the comet assay to measure DNA damage in cells exposed to photosensitizers and gamma radiation

1999 ◽  
Vol 96 (1) ◽  
pp. 143-146 ◽  
Author(s):  
J.-P. Pouget ◽  
J.-L. Ravanat ◽  
T. Douki ◽  
M.-J. Richard ◽  
J. Cadet
Keyword(s):  
Dna Damage ◽  
Comet Assay ◽  
Gamma Radiation ◽  
In Cells

Bestimmung der DNA-Schädigung in vitro

2010 ◽  
Vol 49 (S 01) ◽  
pp. S64-S68
Author(s):  
E. Dikomey
Keyword(s):  
Dna Damage ◽  
Cell Lines ◽  
Chromosome Aberrations ◽  
Genetic Factors ◽  
Double Strand Breaks ◽  
Strand Breaks ◽  
Cell Inactivation ◽  
Repair Mechanisms ◽  
Break Repair

SummaryIonising irradiation acts primarily via induction of DNA damage, among which doublestrand breaks are the most important lesions. These lesions may lead to lethal chromosome aberrations, which are the main reason for cell inactivation. Double-strand breaks can be repaired by several different mechanisms. The regulation of these mechanisms appears be fairly different for normal and tumour cells. Among different cell lines capacity of doublestrand break repair varies by only few percents and is known to be determined mostly by genetic factors. Knowledge about doublestrand break repair mechanisms and their regulation is important for the optimal application of ionising irradiation in medicine.

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