Seizure characteristics of epilepsy in childhood after acute encephalopathy with biphasic seizures and late reduced diffusion

Epilepsia ◽  
2015 ◽  
Vol 56 (8) ◽  
pp. 1286-1293 ◽  
Author(s):  
Yuji Ito ◽  
Jun Natsume ◽  
Hiroyuki Kidokoro ◽  
Naoko Ishihara ◽  
Yoshiteru Azuma ◽  
...  
Keyword(s):  
Acute Encephalopathy

Acute encephalopathy as a primary manifestation of haemophagocytic lymphohistiocytosis

2001 ◽  
Vol 43 (08) ◽  
pp. 555 ◽  
Author(s):  
Matthias Kieslich ◽  
Marilena Vecchi ◽  
Pablo Hernaiz Driever ◽  
Anna Maria Laverda ◽  
Dirk Schwabe ◽  
...  
Keyword(s):  
Acute Encephalopathy ◽  
Haemophagocytic Lymphohistiocytosis

SEVERE ACUTE ENCEPHALOPATHY WITH EXTRAPYRAMIDAL INVOLVEMENT OF UNKNOWN ETIOLOGY

2006 ◽  
Vol 37 (S 1) ◽  
Author(s):  
HA Arroyo ◽  
S Monges ◽  
L Cassar ◽  
L Czornyj ◽  
V Ruggieri ◽  
...  
Keyword(s):  
Unknown Etiology ◽  
Acute Encephalopathy

scholarly journals Reply to Dravet, C. Different Outcomes of Acute Encephalopathy after Status Epilepticus in Patients with Dravet Syndrome. How to Avoid Them? Comment on “De Liso et al. Fatal Status Epilepticus in Dravet Syndrome. Brain Sci. 2020, 10, 889”

2021 ◽  
Vol 11 (6) ◽  
pp. 811
Author(s):  
Paola De Liso ◽  
Virginia Pironi ◽  
Massimo Mastrangelo ◽  
Domenica Battaglia ◽  
Dana Craiu ◽  
...  
Keyword(s):  
Status Epilepticus ◽  
Dravet Syndrome ◽  
Acute Encephalopathy ◽  
To Receive

It has been an honor for us to receive a comment on our article “Fatal Status Epilepticus in Dravet Syndrome” [...]

Prognostic factors in acute encephalopathy with reduced subcortical diffusion

2012 ◽  
Vol 34 (8) ◽  
pp. 632-639 ◽  
Author(s):  
Naoko Hayashi ◽  
Akihisa Okumura ◽  
Tetsuo Kubota ◽  
Takeshi Tsuji ◽  
Hiroyuki Kidokoro ◽  
...  
Keyword(s):  
Prognostic Factors ◽  
Acute Encephalopathy

scholarly journals Involuntary movements and coma as the prognostic marker for acute encephalopathy with biphasic seizures and late reduced diffusion

2016 ◽  
Vol 370 ◽  
pp. 39-43 ◽  
Author(s):  
Sooyoung Lee ◽  
Masafumi Sanefuji ◽  
Michiko Torio ◽  
Noriyuki Kaku ◽  
Yuko Ichimiya ◽  
...  
Keyword(s):  
Prognostic Marker ◽  
Acute Encephalopathy ◽  
Involuntary Movements

Acute encephalopathy in two cases with severe congenital hydrocephalus

2011 ◽  
Vol 33 (7) ◽  
pp. 616-619 ◽  
Author(s):  
Koji Tanoue ◽  
Kiyoshi Matsui ◽  
Akiko Ohshiro ◽  
Atsuko Yamamoto ◽  
Takuya Hayashi ◽  
...  
Keyword(s):  
Congenital Hydrocephalus ◽  
Acute Encephalopathy

Electrographic Seizures and Outcome in Critically Ill Children

Neurology ◽  
2021 ◽  
pp. 10.1212/WNL.0000000000012032
Author(s):  
France W. Fung ◽  
Zi Wang ◽  
Darshana S. Parikh ◽  
Marin Jacobwitz ◽  
Lisa Vala ◽  
...  
Keyword(s):  
Logistic Regression ◽  
Critically Ill ◽  
Odds Ratio ◽  
Cerebral Performance Category ◽  
Unfavorable Outcome ◽  
Critically Ill Children ◽  
Acute Encephalopathy ◽  
Logistic Regression Models ◽  
Electrographic Seizures ◽  
Neurobehavioral Outcomes

Objective:To determine the association between electroencephalographic seizure (ES) and electroencephalographic (ESE) exposure and unfavorable neurobehavioral outcomes in critically ill children with acute encephalopathy.Methods:This was a prospective cohort study of acutely encephalopathic critically ill children undergoing CEEG. ES exposure was assessed as: (1) no ES/ESE, (2) ES, or (3) ESE. Outcomes assessed at discharge included the Glasgow Outcome Scale - Extended Pediatric Version (GOS-E-Peds), Pediatric Cerebral Performance Category (PCPC), and mortality. Unfavorable outcome was defined as a reduction in GOS-E-Peds or PCPC score from pre-admission to discharge. Stepwise selection was used to generate multivariate logistic regression models that assessed associations between ES exposure and outcomes while adjusting for multiple other variables.Results:Among 719 consecutive critically ill subjects, there was no evidence of ES in 535 subjects (74.4%), ES in 140 subjects (19.5%), and ESE in 44 subjects (6.1%). The final multivariable logistic regression analyses included ES exposure, age dichotomized at 1-year, acute encephalopathy category, initial EEG background category, comatose at CEEG initiation, and the Pediatric Index of Mortality 2 score. There was an association between ESE and unfavorable GOS-E-Peds (Odds Ratio 2.21, 95%CI 1.07-4.54) and PCPC (Odds Ratio 2.17, 95%CI 1.05-4.51) but not mortality. There was no association between ES and unfavorable outcome or mortality.Conclusions:Among acutely encephalopathic critically ill children, there was an association between ESE and unfavorable neurobehavioral outcomes, but no association between ESE and mortality. ES exposure was not associated with unfavorable neurobehavioral outcomes or mortality.

Abstract 1122‐000187: Acute Stroke, Subarachnoid Hemorrhage, VZV vasculopathy, and Carotid Dissection: An Interesting Case

2021 ◽  
Vol 1 (S1) ◽  
Author(s):  
Hashaam Arshad ◽  
Zhenhua Gui ◽  
Dakota Owens ◽  
Binod Wagle ◽  
Charles Donohoe
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Lumbar Puncture ◽  
Internal Carotid ◽  
Acute Encephalopathy ◽  
Igg Antibody ◽  
Cerebral Angiogram ◽  
Ct Angiogram ◽  
The Right

Introduction : A 51‐year‐old lady with a past medical history of Essential Hypertension, Hypothyroidism, prior Herpes Zoster infection 8 weeks ago was admitted with complaints of abdominal pain, bilateral flank pain, and restlessness. Her initial workup was significant for hyponatremia and hypokalemia. On the 3rd day of admission, she developed acute hypoxemic respiratory failure which led to intubation. At that time, CTA Chest was not done but CT Chest revealed prominent mucous plugging with left side glass ground opacities, Ultrasound of lower extremities revealed right common femoral vein DVT which led to concerns that she may have suffered from Pulmonary Embolism and led to starting Heparin drip. On the 6th day of admission, she developed Acute Encephalopathy, MRI Brain revealed acute infarcts in bilateral cerebral cortices and cerebella, CT Angiogram Head showed acute subarachnoid hemorrhage in the high posterior right parietal lobe, stenosis of the right high cervical internal carotid artery, and irregular, the appearance of the arterial vasculature throughout and CT Angiogram Neck abrupt change in caliber of the right ICA, 1.5 cm distal to the bifurcation with markedly severe narrowing of the majority of the extracranial right ICA throughout its course. A cerebral Angiogram was done which showed diffuse tandem segments of tandem cervical and intracranial portions of the right internal carotid artery and she was given nitroglycerin was administered as a therapeutic intervention. Lumbar Puncture showed WBC 2, RBC 7, Protein 162, Glucose 64, VZV PCR was negative, CSF VZV IgG Antibody positive at 303 IV (>165 IV indicative of current or past infection). Serum VZV IgG Antibody was positive at >4000 IV. Infectious Diseases were consulted after Lumbar Puncture, they initially started Acyclovir but once the Serum VZV IgG Antibody came back much higher than Serum VZV IgG Antibody levels, their assessment was that VZV vasculitis is unlikely and Acyclovir was discontinued. Eventually, the case was discussed at Neuroradiology which led to us getting a repeat MRA Neck without contrast which showed a concentric T1 and T2 hyperintensity along with a small and irregular caliber right cervical ICA consistent with dissection. She eventually completed a 21‐day course of Nimodipine due to underlying Subarachnoid Hemorrhage. Methods : NA Results : NA Conclusions : Our case demonstrates how it can become difficult to ascertain the etiology of stroke in certain patients. Our patient presented with multiple non‐specific symptoms initially and it was later on due to her Acute Encephalopathy that her Strokes and Subarachnoid Hemorrhage were discovered. It is still difficult to pinpoint whether the cause of strokes was dissection or VZV infection. Lumbar Puncture remains an essential tool to complete work up on uncommon etiologies of stroke.

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