scholarly journals Childhood social environmental and behavioural predictors of early adolescent onset cannabis use

2020 ◽  
Vol 39 (4) ◽  
pp. 384-393
Author(s):  
Kirsty E. Scholes‐Balog ◽  
Sheryl A. Hemphill ◽  
Jessica A. Heerde ◽  
John W. Toumbourou ◽  
George C. Patton
Keyword(s):  
Cannabis Use ◽  
Early Adolescent ◽  
Social Environmental

Early adolescent drinking and cannabis use predicts later sleep-quality problems.

2019 ◽  
Vol 33 (3) ◽  
pp. 266-273 ◽  
Author(s):  
Rowan P. Ogeil ◽  
Ali Cheetham ◽  
Anna Mooney ◽  
Nicholas B. Allen ◽  
Orli Schwartz ◽  
...  
Keyword(s):  
Sleep Quality ◽  
Cannabis Use ◽  
Early Adolescent ◽  
Adolescent Drinking

scholarly journals Multimodal neuroimaging of frontal white matter microstructure in early phase schizophrenia: the impact of early adolescent cannabis use

2013 ◽  
Vol 13 (1) ◽  
Author(s):  
Denise Bernier ◽  
Jacob Cookey ◽  
David McAllindon ◽  
Robert Bartha ◽  
Christopher C Hanstock ◽  
...  
Keyword(s):  
White Matter ◽  
Early Phase ◽  
Cannabis Use ◽  
Early Adolescent ◽  
Multimodal Neuroimaging ◽  
White Matter Microstructure ◽  
Frontal White Matter ◽  
The Impact

Cannabis Use and Dependence

2002 ◽  
Author(s):  
Wayne Hall ◽  
Rosalie Liccardo Pacula
Keyword(s):  
Cannabis Use

Cannabis use in Switzerland: The role of attribution of drug use to friends, urbanization and repression 1The study was supported by the Swiss Federal Office of Public Health (Bundesamt für Gesundheit BAG) Grant 316.96.5980.

2001 ◽  
Vol 60 (2) ◽  
pp. 99-107 ◽  
Author(s):  
Holger Schmid
Keyword(s):  
Drug Use ◽  
Urban Areas ◽  
Hierarchical Linear Model ◽  
Cannabis Use ◽  
School Aged Children ◽  
Structural Variables ◽  
Hbsc Study ◽  
Nationally Representative ◽  
Highly Correlated ◽  
Individual Use

Cannabis use does not show homogeneous patterns in a country. In particular, urbanization appears to influence prevalence rates, with higher rates in urban areas. A hierarchical linear model (HLM) was employed to analyze these structural influences on individuals in Switzerland. Data for this analysis were taken from the Switzerland survey of Health Behavior in School-Aged Children (HBSC) Study, the most recent survey to assess drug use in a nationally representative sample of 3473 15-year-olds. A total of 1487 male and 1620 female students indicated their cannabis use and their attributions of drug use to friends. As second level variables we included address density in the 26 Swiss Cantons as an indicator of urbanization and officially recorded offences of cannabis use in the Cantons as an indicator of repressive policy. Attribution of drug use to friends is highly correlated with cannabis use. The correlation is even more pronounced in urban Cantons. However, no association between recorded offences and cannabis use was found. The results suggest that structural variables influence individuals. Living in an urban area effects the attribution of drug use to friends. On the other hand repressive policy does not affect individual use.

Cannabis-associated arteritis

VASA ◽  
2010 ◽  
Vol 39 (1) ◽  
pp. 43-53 ◽  
Author(s):  
Grotenhermen
Keyword(s):  
Systematic Review ◽  
Case Reports ◽  
Case Series ◽  
Cardiovascular Effects ◽  
Causative Factor ◽  
Cannabis Use ◽  
Thromboangiitis Obliterans ◽  
Pathological Features ◽  
Scientific Support ◽  
Clinical And Pathological Features

Background: To investigate the hypothesis that cases of arteritis similar to thromboangiitis obliterans (TAO) and associated with the use of cannabis were caused by cannabis or THC (dronabinol), or that cannabis use is a co-factor of TAO. Patients and methods: A systematic review on case reports and the literature on so-called cannabis arteritis, TAO, and cardiovascular effects of cannabinoids was conducted. Results: Fifteen reports with 57 cases of an arteritis associated with the use of cannabis and two additional case series of TAO, in which some patients also used cannabis, were identified. Clinical and pathological features of cannabis-associated arteritis do not differ from TAO and the major risk factor of TAO, tobacco use, was present in most, if not in all of these cases. The proposed pathophysiological mechanisms for the development of an arteritis by cannabis use are not substantiated. Conclusions: The hypothesis of cannabis being a causative factor or co-factor of TAO or an arteritis similar to TAO is not supported by the available evidence. The use of the term “cannabis arteritis” should be avoided until or unless more convincing scientific support is forthcoming.

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