Modified Atkins diet vs classic ketogenic formula in intractable epilepsy

2013 ◽  
Vol 128 (6) ◽  
pp. 402-408 ◽  
Author(s):  
O. F. El-Rashidy ◽  
M. F. Nassar ◽  
I. A. Abdel-Hamid ◽  
R. H. Shatla ◽  
M. H. Abdel-Hamid ◽  
...  
Keyword(s):  
Intractable Epilepsy ◽  
Modified Atkins Diet ◽  
Atkins Diet

scholarly journals Is Modified Atkins Diet Therapy Applicable for Adults with Intractable Epilepsy Patients? - A Short Trial in Adult Volunteers

2011 ◽  
Vol 4 (1) ◽  
pp. 7-14
Author(s):  
Miyako Oguni ◽  
Noriyo Inoue ◽  
Kyoya Takahata ◽  
Masamichi Koseki
Keyword(s):  
Intractable Epilepsy ◽  
Diet Therapy ◽  
Modified Atkins Diet ◽  
Atkins Diet ◽  
Adult Volunteers

scholarly journals Danish study of a Modified Atkins diet for medically intractable epilepsy in children: Can we achieve the same results as with the classical ketogenic diet?

Seizure ◽  
2011 ◽  
Vol 20 (2) ◽  
pp. 151-155 ◽  
Author(s):  
Maria J. Miranda ◽  
Mette Mortensen ◽  
Jane H. Povlsen ◽  
Helle Nielsen ◽  
Sándor Beniczky
Keyword(s):  
Ketogenic Diet ◽  
Intractable Epilepsy ◽  
Modified Atkins Diet ◽  
Atkins Diet ◽  
Danish Study

scholarly journals A Nigerian‐Made Modified Atkins Diet: Hope for Patients with Intractable Epilepsy

2021 ◽  
Vol 35 (S1) ◽  
Author(s):  
Maryam Salaudeen ◽  
Mohammed Magaji ◽  
Abdulkadir Zezi
Keyword(s):  
Intractable Epilepsy ◽  
Modified Atkins Diet ◽  
Atkins Diet

P189 Modified Atkins diet for medically intractable epilepsy in children: a European study

2009 ◽  
Vol 13 ◽  
pp. S79
Author(s):  
M.J. Miranda ◽  
M. Mortensen ◽  
J.H. Povlsen ◽  
H. Nielsen ◽  
S. Beniczky
Keyword(s):  
Intractable Epilepsy ◽  
European Study ◽  
Modified Atkins Diet ◽  
Atkins Diet

scholarly journals Modified Atkins diet to children and adolescents with medical intractable epilepsy

Seizure ◽  
2009 ◽  
Vol 18 (4) ◽  
pp. 237-240 ◽  
Author(s):  
Susanne Weber ◽  
Christian Mølgaard ◽  
KarenTaudorf ◽  
Peter Uldall
Keyword(s):  
Children And Adolescents ◽  
Intractable Epilepsy ◽  
Modified Atkins Diet ◽  
Atkins Diet

scholarly journals Comparison of the Effect of Classical Ketogenic Diet and Modified Atkins Diet on Lipid Profile in Patients with Intractable Epilepsy

2020 ◽  
Vol 4 (Supplement_2) ◽  
pp. 1139-1139
Author(s):  
Shahabeddin Rezaei ◽  
Mona Kavoosi ◽  
Reza Shervin Badv ◽  
Mahmoud Mohammadi ◽  
Gholam Reza Zamani ◽  
...  
Keyword(s):  
Lipid Profile ◽  
Ketogenic Diet ◽  
Repeated Measures ◽  
Intractable Epilepsy ◽  
Density Lipoprotein ◽  
Serum Levels ◽  
Modified Atkins Diet ◽  
Atkins Diet ◽  
Significant Difference ◽  
Before And After

Abstract Objectives This study aimed to shed light on the differences between the effect of classical ketogenic diet (KD) and modified Atkins diet (MAD) on lipid profile in children and adolescents with intractable epilepsy. Methods The study was a non-randomized controlled clinical trial, conducted in the period of 2015 to 2017 (ClinicalTrials.gov, NCT03014752). Inclusion criteria included patients aged 1–18 years old, resistance to at least two antiepileptic drugs and no history of metabolic diseases that KD is contraindicated. The exclusion criteria included the occurrence of serious adverse effects and reluctance to adhere to the diet. Patients received the interventions for three months. Classical KD was initiated with 4:1 ketogenic ratio. MAD was initiated with a ketogenic ratio of 1:1 to 2:1 according to the John Hopkins protocol. The blood sample was obtained for measurement of the lipid profile, including triglyceride (TG), total cholesterol (TC), low-density lipoprotein (LDL) and high-density lipoprotein (HDL). A repeated measures ANOVA was performed to compare the differences between groups. Results Twenty-six patients were allocated to classical KD and 26 were allocated to MAD. Fourteen patients remained at the end of the study in each group. In the classical KD group, the serum levels of TC, TG and LDL increased significantly before and after three months. In the MAD group, the serum levels of TC and LDL increased significantly before and after the study. After three months, there was a significant difference in the serum levels of TG and LDL between classical KD and MAD groups (P < 0.05); however, no significant difference was observed in the serum levels of TC and HDL in both groups (P > 0.05). Conclusions Patients who were on the MAD had significantly higher levels of TG and LDL than those who were on classical KD. Funding Sources Tehran University of Medical Sciences.

scholarly journals A Prospective Study of the Modified Atkins Diet for Intractable Epilepsy in Adults

Epilepsia ◽  
2008 ◽  
Vol 49 (2) ◽  
pp. 316-319 ◽  
Author(s):  
Eric H. Kossoff ◽  
Hannah Rowley ◽  
Saurabh R. Sinha ◽  
Eileen P. G. Vining
Keyword(s):  
Prospective Study ◽  
Intractable Epilepsy ◽  
Modified Atkins Diet ◽  
Atkins Diet ◽  
A Prospective Study

scholarly journals Classic Ketogenic Diet and Modified Atkins Diet in SLC2A1 Positive and Negative Patients with Suspected GLUT1 Deficiency Syndrome: A Single Center Analysis of 18 Cases

Nutrients ◽  
2021 ◽  
Vol 13 (3) ◽  
pp. 840
Author(s):  
Jana Ruiz Herrero ◽  
Elvira Cañedo Villarroya ◽  
Luis González Gutiérrez-Solana ◽  
Beatriz García Alcolea ◽  
Begoña Gómez Fernández ◽  
...  
Keyword(s):  
Movement Disorder ◽  
Glucose Transporter ◽  
Deficiency Syndrome ◽  
Laboratory Assessment ◽  
Modified Atkins Diet ◽  
Atkins Diet ◽  
Glut1 Deficiency ◽  
Glut1 Deficiency Syndrome ◽  
The Impact

Background: Glucose transporter type 1 deficiency syndrome (GLUT1DS) is caused by mutations in the SLC2A1 gene and produces seizures, neurodevelopmental impairment, and movement disorders. Ketogenic dietary therapies (KDT) are the gold standard treatment. Similar symptoms may appear in SLC2A1 negative patients. The purpose is to evaluate the effectiveness of KDT in children with GLUT1DS suspected SLC2A1 (+) and (-), side effects (SE), and the impact on patients nutritional status. Methods: An observational descriptive study was conducted to describe 18 children (January 2009–August 2020). SLC2A1 analysis, seizures, movement disorder, anti-epileptic drugs (AEDS), anthropometry, SE, and laboratory assessment were monitored baseline and at 3, 6, 12, and 24 months after the onset of KDT. Results: 6/18 were SLC2A1(+) and 13/18 had seizures. In these groups, the age for debut of symptoms was higher. The mean time from debut to KDT onset was higher in SLC2A1(+). The modified Atkins diet (MAD) was used in 12 (5 SLC2A1(+)). Movement disorder improved (4/5), and a reduction in seizures >50% compared to baseline was achieved in more than half of the epileptic children throughout the follow-up. No differences in effectiveness were found according to the type of KDT. Early SE occurred in 33%. Long-term SE occurred in 10, 5, 7, and 5 children throughout the follow-up. The most frequent SE were constipation, hypercalciuria, and hyperlipidaemia. No differences in growth were found according to the SLC2A1 mutation or type of KDT. Conclusions: CKD and MAD were effective for SLC2A1 positive and negative patients in our cohort. SE were frequent, but mild. Permanent monitoring should be made to identify SE and nutritional deficits.

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