scholarly journals Correction of Diabetes Mellitus by Transplanting Minimal Mass of Syngeneic Islets Into Vascularized Small Intestinal Segment

2013 ◽  
Vol 13 (10) ◽  
pp. 2550-2557 ◽  
Author(s):  
Z. Kakabadze ◽  
S. Gupta ◽  
A. Pileggi ◽  
R. D. Molano ◽  
C. Ricordi ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Intestinal Segment ◽  
Small Intestinal ◽  
Minimal Mass

Development of a portocaval shunt using a small intestinal segment in rats

Microsurgery ◽  
2010 ◽  
Vol 30 (4) ◽  
pp. 302-306 ◽  
Author(s):  
Toshiyuki Hata ◽  
Junji Iwasaki ◽  
Shuji Hishikawa ◽  
Yasuhiro Fujimoto ◽  
Shinji Uemoto ◽  
...  
Keyword(s):  
Portocaval Shunt ◽  
Intestinal Segment ◽  
Small Intestinal

scholarly journals DO H. PYLORI STATUS AND SMALL INTESTINAL BACTERIAL OVERGROWTH DETERMINE THE CLINICAL COURSE OF CHRONIC ACTIVE GASTRITIS IN PATIENTS WITH TYPE 2 DIABETES MELLITUS?

2020 ◽  
Vol 73 (6) ◽  
pp. 1223-1228
Author(s):  
Tetiana O. Radionova ◽  
Igor M. Skrypnyk ◽  
Ganna S. Maslova
Keyword(s):  
Diabetes Mellitus ◽  
Type 2 Diabetes ◽  
Epigastric Pain ◽  
Bacterial Overgrowth ◽  
Small Intestinal Bacterial Overgrowth ◽  
Postprandial Fullness ◽  
Chronic Active Gastritis ◽  
Intestinal Bacterial Overgrowth ◽  
Small Intestinal

The aim: To define clinical peculiarities of chronic active gastritis in patients with type 2 diabetes mellitus (T2DM) considering Helicobacter pylori (HP) status and small intestinal bacterial overgrowth (SIBO). Materials and methods: 172 patients with chronic active gastritis were enrolled in the study, 92 out of them had concomitant T2DM. Symptoms were collected with the questionnaire, HP infection was diagnosed with stool antigen test, SIBO was assessed with glucose hydrogen breath test. Results: 87.5% (n=70) patients with chronic gastritis without DM had epigastric pain, however those with T2DM reported pain only in 41.3% (n=38) cases. Other symptoms included: nausea, bloating, early satiety, postprandial fullness, heartburn, belching and vomiting. HP infection in patients with chronic gastritis and concomitant T2DM is significantly associated with symptoms of epigastric pain (OR=2.78, 95%CI 0.92-8.41), bloating (OR=3.92, 95%CI 1.40-10.99), nausea (OR=2.32, 95%CI 0.85-0.6.30), postprandial fullness (OR=1.45, 95%CI 0.54-3.87) and belching (OR=1.01, 95%CI 0.32-3.16), whereas SIBO – with bloating (OR=8.82, 95%CI 2.88-27.01), nausea (OR=5.15, 95%CI 1.88-14.10) and belching (OR=2.53, 95%CI 0.67-9.52). Conclusions: Patients with T2DM and chronic active gastritis report epigastric pain significantly less than non-diabetics. HP infection probably plays a prominent role in development of epigastric pain in patients with T2DM. Additionally, HP is linked to SIBO, which may lead to bloating, belching and nausea onset.

scholarly journals Non-communicating small intestinal duplication in a dog: a case report

2017 ◽  
Vol 62 (No. 9) ◽  
pp. 516-521 ◽  
Author(s):  
TS Hwang ◽  
DI Jung ◽  
JH Kim ◽  
SC Yeon ◽  
HC Lee
Keyword(s):  
Physical Examination ◽  
Complete Blood Count ◽  
Postoperative Recovery ◽  
Histopathological Diagnosis ◽  
Intestinal Segment ◽  
Radiographic Images ◽  
Intestinal Duplication ◽  
National University ◽  
Enteric Duplication ◽  
Small Intestinal

Enteric duplication is rare in dogs. Here, we report the rarest form of duplication in which two segments are parallel and share a wall for most of their lengths. A nine-year-old spayed female Yorkshire terrier was referred to the Veterinary Medical Teaching Hospital at Gyeongsang National University due to anorexia and diarrhoea. Physical examination, haematological examination, radiography, and ultrasonography were performed. On physical examination, dry, pale mucous membrane was identified. Moderate anaemia with decreased packed cell volume was detected in complete blood count. Serum urea nitrogen and creatinine levels were mildly increased. Radiographic images revealed no significant findings. On ultrasonographic examination, a multi-layered appearance of a focal small intestinal segment was identified in the left mid abdomen. Following the lesion, it was divided into two small intestinal segments. Based on imaging findings, intussusceptions or enteric duplication were suspected. To resect the abnormal small intestinal segment, enterectomy was performed. Follow-up was not performed because the patient expired during the postoperative recovery time. The histopathological diagnosis was non-communicating small intestinal duplication. Non-communicating intestinal duplication is related to embryologic abnormalities and is usually concurrent with other anomalies such as vertebral malformations and urogenital duplications. However, this case had no other anomalies associated with the malformation of the intestine.

Small Intestinal Transit, Bacterial Growth, and Bowel Habits in Diabetes Mellitus

Pancreas ◽  
1989 ◽  
Vol 4 (1) ◽  
pp. 65-70 ◽  
Author(s):  
U. Spengler ◽  
F. Stellaard ◽  
G. Ruckdeschel ◽  
C. Scheurlen ◽  
W. Kruis
Keyword(s):  
Diabetes Mellitus ◽  
Bacterial Growth ◽  
Intestinal Transit ◽  
Small Intestinal Transit ◽  
Bowel Habits ◽  
Small Intestinal

Localization of cholesterol synthesis in the small intestine of diabetic rats

1984 ◽  
Vol 247 (5) ◽  
pp. G494-G501
Author(s):  
K. R. Feingold ◽  
A. H. Moser
Keyword(s):  
Diabetes Mellitus ◽  
Small Intestine ◽  
Food Intake ◽  
Cholesterol Synthesis ◽  
Diabetic Rats ◽  
Cholesterol Feeding ◽  
Small Intestinal ◽  
Stimulation Of ◽  
Do So

Previous studies have demonstrated that cholesterol synthesis is increased twofold in the small intestine of diabetic animals. The present study demonstrates that the stimulation of small intestinal cholesterol synthesis by diabetes is a generalized phenomenon occurring in all segments of the small intestine. Quantitatively, in control animals the proximal two segments of the small intestine account for the majority of the total small intestinal cholesterol synthesis, whereas in the diabetic animals, because of the generalized stimulation in cholesterogenesis, the contribution of the terminal segments to total small intestinal cholesterol synthesis is of increased importance. The various manipulations that regulate cholesterol synthesis in the small intestine of diabetic animals also affect cholesterol synthesis in all portions of the small intestine. In diabetic animals cholesterol feeding and the limitation of food intake decrease cholesterol synthesis in the total small intestine and in all segments of the small intestine. Conversely, colestipol feeding increases cholesterol synthesis in all segments of the small intestine. These results demonstrate that, despite the obvious structural, functional, and environmental differences among the various segments of the small intestine, the stimulation of cholesterol synthesis that occurs secondary to diabetes mellitus is a generalized phenomenon. Similarly, the factors that regulate small intestinal cholesterol synthesis do so in a generalized manner.

Changes in small intestinal motility during the long-term course of insulin-dependent diabetes mellitus

2000 ◽  
Vol 118 (4) ◽  
pp. A382 ◽  
Author(s):  
Gabriele Groeger ◽  
Marlene Kenter ◽  
Corinna Schweflinghaus ◽  
Leelamma Cherian ◽  
Harald Goebell ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Intestinal Motility ◽  
Insulin Dependent Diabetes Mellitus ◽  
Insulin Dependent Diabetes ◽  
Dependent Diabetes Mellitus ◽  
Small Intestinal Motility ◽  
Insulin Dependent ◽  
Small Intestinal

Effects of small intestinal nutrients on antropyloroduodenal motility and perceptions in type 1 diabetes mellitus

2001 ◽  
Vol 120 (5) ◽  
pp. A463-A463
Author(s):  
C RAYNER ◽  
M SCHWARTZ ◽  
P VANDAM ◽  
W RENOOIJ ◽  
M DESMET ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Type 1 Diabetes ◽  
Type 1 Diabetes Mellitus ◽  
Small Intestinal
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