Nicotine-induced molecular alterations are modulated by GABAB receptor activity

2017 ◽  
Vol 23 (1) ◽  
pp. 230-246 ◽  
Author(s):  
Andres P. Varani ◽  
Valeria T. Pedrón ◽  
Amira J. Aon ◽  
Christian Höcht ◽  
Gabriela B. Acosta ◽  
...  
Keyword(s):  
Gabab Receptor ◽  
Receptor Activity ◽  
Molecular Alterations

scholarly journals Postnatal myelination of the immature rat cingulum is regulated by GABAB receptor activity

2021 ◽  
Author(s):  
Samipa Pudasaini ◽  
Vivien Friedrich ◽  
Christoph Bührer ◽  
Stefanie Endesfelder ◽  
Till Scheuer ◽  
...  
Keyword(s):  
Gabab Receptor ◽  
Receptor Activity ◽  
Immature Rat

EP 138. Impact of recurrent mild traumatic brain injury on GABA concentration and GABAB receptor activity

2016 ◽  
Vol 127 (9) ◽  
pp. e298-e299
Author(s):  
S. Wilke ◽  
J. List ◽  
R. Mekle ◽  
R. Lindenberg ◽  
M. Bukowski ◽  
...  
Keyword(s):  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Mild Traumatic Brain Injury ◽  
Gabab Receptor ◽  
Receptor Activity ◽  
Gaba Concentration

scholarly journals Possible regulation of GABAB receptor activity by dual specific phosphatases

2016 ◽  
Vol 10 ◽  
Author(s):  
Higashi Hiroshi ◽  
Kinjo Toshihiko ◽  
Kuramoto Nobuyuki ◽  
Yoneda Yukio
Keyword(s):  
Gabab Receptor ◽  
Receptor Activity

Modulation of neuronal circuits by GABAB receptor activity in the mouse lateral amygdala

2011 ◽  
Vol 71 ◽  
pp. e212
Author(s):  
Tomomi Fujieda ◽  
Tomoaki Shirao ◽  
Hideki Miwa ◽  
Yuko Sekino
Keyword(s):  
Gabab Receptor ◽  
Receptor Activity ◽  
Neuronal Circuits ◽  
Lateral Amygdala

scholarly journals Characterization of Mice with Targeted Deletion of Glycine Receptor Alpha 2

2006 ◽  
Vol 26 (15) ◽  
pp. 5728-5734 ◽  
Author(s):  
T. L. Young-Pearse ◽  
L. Ivic ◽  
A. R. Kriegstein ◽  
C. L. Cepko
Keyword(s):  
Nervous System ◽  
Chloride Channels ◽  
Neuronal Development ◽  
Glycine Receptor ◽  
Receptor Activity ◽  
Rod Photoreceptor ◽  
Targeted Deletion ◽  
Receptor Alpha ◽  
Molecular Alterations ◽  
Electrophysiological Responses

ABSTRACT Glycine receptors are ligand-gated chloride channels that mediate inhibitory neurotransmission in the adult nervous system. During development, glycine receptor alpha 2 (GlyRα2) is expressed in the retina, in the spinal cord, and throughout the brain. Within the cortex, GlyRα2 is expressed in immature cells and these receptors have been shown to be active and excitatory. In the developing retina, inhibition of glycine receptor activity prevents proper rod photoreceptor development. These data suggest that GlyRα2, the developmentally expressed glycine receptor, may play an important role in neuronal development. We have generated mice with a targeted deletion of glycine receptor alpha 2 (Glra2). Although these mice lack expression of GlyRα2, no gross morphological or molecular alterations were observed in the nervous system. In addition, the cerebral cortex does not appear to require glycine receptor activity for proper development, as Glra2 knockout mice did not show any electrophysiological responses to glycine.

Morphine-induced depression of spinal excitation is not altered following acute disruption of GABAA or GABAB receptor activity

2008 ◽  
Vol 12 (6) ◽  
pp. 677-685 ◽  
Author(s):  
Itsaso Buesa ◽  
Andrés Urrutia ◽  
Juan Bilbao ◽  
Luciano Aguilera ◽  
Manfred Zimmermann ◽  
...  
Keyword(s):  
Gabab Receptor ◽  
Receptor Activity
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