Optimal Subset Selection of Stochastic Model Using Statistical Hypothesis Test

2018 ◽  
Vol 48 (4) ◽  
pp. 557-564 ◽  
Author(s):  
Seon Han Choi ◽  
Tag Gon Kim
2019 ◽  
Vol 19 (2) ◽  
pp. 134-140
Author(s):  
Baek-Ju Sung ◽  
Sung-kyu Lee ◽  
Mu-Seong Chang ◽  
Do-Sik Kim

2021 ◽  
Author(s):  
Abhijit Mahesh Chinchani ◽  
Mahesh Menon ◽  
Meighen Roes ◽  
Heungsun Hwang ◽  
Paul Allen ◽  
...  

Cognitive mechanisms hypothesized to underlie hallucinatory experiences (HEs) include dysfunctional source monitoring, heightened signal detection, or impaired attentional processes. HEs can be very pronounced in psychosis, but similar experiences also occur in nonclinical populations. Using data from an international multisite study on nonclinical subjects (N = 419), we described the overlap between two sets of variables - one measuring cognition and the other HEs - at the level of individual items, allowing extraction of item-specific signal which might considered off-limits when summary scores are analyzed. This involved using a statistical hypothesis test at the multivariate level, and variance constraints, dimension reduction, and split-half reliability checks at the level of individual items. The results showed that (1) modality-general HEs involving sensory distortions (hearing voices/sounds, troubled by voices, everyday things look abnormal, sensations of presence/movement) were associated with more liberal auditory signal detection, and (2) HEs involving experiences of sensory overload and vivid images/imagery (viz., HEs for faces and intense daydreams) were associated with other-ear distraction and reduced laterality in dichotic listening. Based on these results, it is concluded that the overlap between HEs and cognition variables can be conceptualized as modality-general and bi-dimensional: one involving distortions, and the other involving overload or intensity.


Computation ◽  
2020 ◽  
Vol 8 (2) ◽  
pp. 59 ◽  
Author(s):  
Giovanni Delnevo ◽  
Silvia Mirri ◽  
Marco Roccetti

As we prepare to emerge from an extensive and unprecedented lockdown period, due to the COVID-19 virus infection that hit the Northern regions of Italy with the Europe’s highest death toll, it becomes clear that what has gone wrong rests upon a combination of demographic, healthcare, political, business, organizational, and climatic factors that are out of our scientific scope. Nonetheless, looking at this problem from a patient’s perspective, it is indisputable that risk factors, considered as associated with the development of the virus disease, include older age, history of smoking, hypertension and heart disease. While several studies have already shown that many of these diseases can also be favored by a protracted exposure to air pollution, there has been recently an insurgence of negative commentary against authors who have correlated the fatal consequences of COVID-19 (also) to the exposition of specific air pollutants. Well aware that understanding the real connection between the spread of this fatal virus and air pollutants would require many other investigations at a level appropriate to the scale of this phenomenon (e.g., biological, chemical, and physical), we propose the results of a study, where a series of the measures of the daily values of PM2.5, PM10, and NO2 were considered over time, while the Granger causality statistical hypothesis test was used for determining the presence of a possible correlation with the series of the new daily COVID19 infections, in the period February–April 2020, in Emilia-Romagna. Results taken both before and after the governmental lockdown decisions show a clear correlation, although strictly seen from a Granger causality perspective. Moving beyond the relevance of our results towards the real extent of such a correlation, our scientific efforts aim at reinvigorating the debate on a relevant case, that should not remain unsolved or no longer investigated.


2015 ◽  
Vol 117 (2) ◽  
pp. 131-141 ◽  
Author(s):  
Michael Baltaxe ◽  
Peter Meer ◽  
Michael Lindenbaum

2021 ◽  
Vol 11 (3) ◽  
pp. 697-702
Author(s):  
S. Jayanthi ◽  
C. R. Rene Robin

In this study, DNA microarray data is analyzed from a signal processing perspective for cancer classification. An adaptive wavelet transform named Empirical Wavelet Transform (EWT) is analyzed using block-by-block procedure to characterize microarray data. The EWT wavelet basis depends on the input data rather predetermined like in conventional wavelets. Thus, EWT gives more sparse representations than wavelets. The characterization of microarray data is made by block-by-block procedure with predefined block sizes in powers of 2 that starts from 128 to 2048. After characterization, a statistical hypothesis test is employed to select the informative EWT coefficients. Only the selected coefficients are used for Microarray Data Classification (MDC) by the Support Vector Machine (SVM). Computational experiments are employed on five microarray datasets; colon, breast, leukemia, CNS and ovarian to test the developed cancer classification system. The obtained results demonstrate that EWT coefficients with SVM emerged as an effective approach with no misclassification for MDC system.


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